Tal Iram

5.5k total citations · 2 hit papers
14 papers, 1.7k citations indexed

About

Tal Iram is a scholar working on Molecular Biology, Immunology and Cellular and Molecular Neuroscience. According to data from OpenAlex, Tal Iram has authored 14 papers receiving a total of 1.7k indexed citations (citations by other indexed papers that have themselves been cited), including 7 papers in Molecular Biology, 5 papers in Immunology and 4 papers in Cellular and Molecular Neuroscience. Recurrent topics in Tal Iram's work include Alzheimer's disease research and treatments (4 papers), Neuroinflammation and Neurodegeneration Mechanisms (3 papers) and Neurogenesis and neuroplasticity mechanisms (3 papers). Tal Iram is often cited by papers focused on Alzheimer's disease research and treatments (4 papers), Neuroinflammation and Neurodegeneration Mechanisms (3 papers) and Neurogenesis and neuroplasticity mechanisms (3 papers). Tal Iram collaborates with scholars based in United States, Israel and Australia. Tal Iram's co-authors include Tony Wyss‐Coray, Michael S. Haney, Michael C. Bassik, John V. Pluvinage, David W. Morgens, Carolyn R. Bertozzi, Ludwig Aigner, Heimo Wolinski, Vidhu Mathur and Macy E. Zardeneta and has published in prestigious journals such as Nature, Proceedings of the National Academy of Sciences and Neuron.

In The Last Decade

Tal Iram

14 papers receiving 1.7k citations

Hit Papers

Lipid-droplet-accumulating microglia represent a dysfunct... 2019 2026 2021 2023 2020 2019 250 500 750
What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

  1. Lipid-droplet-accumulating microglia represent a dysfunctional and proinflammatory state in the aging brain
    2020 793 citations Julia Marschallinger, Tal Iram et al. Nature Neuroscience profile →
  2. CD22 blockade restores homeostatic microglial phagocytosis in ageing brains
    2019 325 citations John V. Pluvinage, Michael S. Haney et al. Nature profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Tal Iram United States 9 846 594 550 550 204 14 1.7k
John V. Pluvinage United States 11 927 1.1× 617 1.0× 759 1.4× 483 0.9× 120 0.6× 14 2.0k
Christian Jung Germany 20 427 0.5× 219 0.4× 644 1.2× 552 1.0× 231 1.1× 31 1.5k
Ludovico Cantuti‐Castelvetri Germany 21 1.1k 1.3× 636 1.1× 1.1k 2.0× 792 1.4× 382 1.9× 27 2.8k
Lieneke Kooijman Netherlands 18 924 1.1× 323 0.5× 838 1.5× 858 1.6× 464 2.3× 26 2.1k
Laia Acarín Spain 26 711 0.8× 394 0.7× 569 1.0× 255 0.5× 346 1.7× 42 1.7k
Martina Moeton Netherlands 16 402 0.5× 260 0.4× 462 0.8× 292 0.5× 183 0.9× 19 1.2k
Annie Gervais France 11 686 0.8× 416 0.7× 304 0.6× 208 0.4× 245 1.2× 21 1.2k
Brian P. Hafler United States 13 798 0.9× 384 0.6× 1.2k 2.2× 572 1.0× 259 1.3× 22 2.2k
Cécile Delarasse France 22 573 0.7× 528 0.9× 508 0.9× 249 0.5× 242 1.2× 43 2.0k
Yuka Atagi United States 12 894 1.1× 514 0.9× 522 0.9× 849 1.5× 310 1.5× 13 1.8k

Countries citing papers authored by Tal Iram

Since Specialization
Citations

This map shows the geographic impact of Tal Iram's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Tal Iram with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Tal Iram more than expected).

Fields of papers citing papers by Tal Iram

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Tal Iram. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Tal Iram. The network helps show where Tal Iram may publish in the future.

Co-authorship network of co-authors of Tal Iram

This figure shows the co-authorship network connecting the top 25 collaborators of Tal Iram. A scholar is included among the top collaborators of Tal Iram based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Tal Iram. Tal Iram is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

14 of 14 papers shown
1.
Iram, Tal, Miguel Ángel García, Achint Kaur, et al.. (2024). SRF transcriptionally regulates the oligodendrocyte cytoskeleton during CNS myelination. Proceedings of the National Academy of Sciences. 121(12). e2307250121–e2307250121. 3 indexed citations
2.
Kaur, Achint, et al.. (2023). A protocol for collection and infusion of cerebrospinal fluid in mice. STAR Protocols. 4(1). 102015–102015. 5 indexed citations
3.
Kapitansky, Oxana, Gidon Karmon, Shlomo Sragovich, et al.. (2020). Single Cell ADNP Predictive of Human Muscle Disorders: Mouse Knockdown Results in Muscle Wasting. Cells. 9(10). 2320–2320. 13 indexed citations
4.
Marschallinger, Julia, Tal Iram, Macy E. Zardeneta, et al.. (2020). Lipid-droplet-accumulating microglia represent a dysfunctional and proinflammatory state in the aging brain. Nature Neuroscience. 23(2). 194–208. 793 indexed citations breakdown →
5.
Gozes, Illana, Yanina Ivashko‐Pachima, Oxana Kapitansky, Carmen Laura Sayas, & Tal Iram. (2019). Single-cell analysis of cytoskeleton dynamics: From isoelectric focusing to live cell imaging and RNA-seq. Journal of Neuroscience Methods. 323. 119–124. 4 indexed citations
6.
Pluvinage, John V., Michael S. Haney, Benjamin Smith, et al.. (2019). CD22 blockade restores homeostatic microglial phagocytosis in ageing brains. Nature. 568(7751). 187–192. 325 indexed citations breakdown →
7.
Iram, Tal, Zaida G. Ramirez‐Ortiz, Michael H. Byrne, et al.. (2016). Megf10 Is a Receptor for C1Q That Mediates Clearance of Apoptotic Cells by Astrocytes. Journal of Neuroscience. 36(19). 5185–5192. 131 indexed citations
8.
Iram, Tal, Dorit Trudler, David Kain, et al.. (2016). Astrocytes from old Alzheimer's disease mice are impaired in Aβ uptake and in neuroprotection. Neurobiology of Disease. 96. 84–94. 91 indexed citations
9.
Schmukler, Eran, Tal Iram, Dan Frenkel, et al.. (2016). Impaired Autophagy in APOE4 Astrocytes. Journal of Alzheimer s Disease. 51(3). 915–927. 105 indexed citations
10.
11.
Gozes, Illana, et al.. (2014). Novel Tubulin and Tau Neuroprotective Fragments Sharing Structural Similarities with the Drug Candidate NAP (Davuentide). Journal of Alzheimer s Disease. 40(s1). S23–S36. 24 indexed citations
12.
Ramirez‐Ortiz, Zaida G., William F. Pendergraft, Amit Prasad, et al.. (2013). The scavenger receptor SCARF1 mediates the clearance of apoptotic cells and prevents autoimmunity. Nature Immunology. 14(9). 917–926. 169 indexed citations
13.
Yu, Xiaomeng, Timothy J. Mosca, Tal Iram, et al.. (2013). Plum, an Immunoglobulin Superfamily Protein, Regulates Axon Pruning by Facilitating TGF-β Signaling. Neuron. 78(3). 456–468. 47 indexed citations
14.
Iram, Tal & Dan Frenkel. (2012). Targeting the Role of Astrocytes in the Progression of Alzheimers Disease. Current Signal Transduction Therapy. 7(1). 20–27. 6 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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