Takeshi Kunimatsu

684 total citations
23 papers, 524 citations indexed

About

Takeshi Kunimatsu is a scholar working on Molecular Biology, Health, Toxicology and Mutagenesis and Endocrinology, Diabetes and Metabolism. According to data from OpenAlex, Takeshi Kunimatsu has authored 23 papers receiving a total of 524 indexed citations (citations by other indexed papers that have themselves been cited), including 10 papers in Molecular Biology, 8 papers in Health, Toxicology and Mutagenesis and 6 papers in Endocrinology, Diabetes and Metabolism. Recurrent topics in Takeshi Kunimatsu's work include Effects and risks of endocrine disrupting chemicals (8 papers), Cardiac electrophysiology and arrhythmias (5 papers) and Neuroscience and Neural Engineering (4 papers). Takeshi Kunimatsu is often cited by papers focused on Effects and risks of endocrine disrupting chemicals (8 papers), Cardiac electrophysiology and arrhythmias (5 papers) and Neuroscience and Neural Engineering (4 papers). Takeshi Kunimatsu collaborates with scholars based in Japan and United States. Takeshi Kunimatsu's co-authors include Takaki Seki, Hitoshi Funabashi, Juki Kimura, Kiyoko Bando, Tomoya Yamada, Takeshi Bamba, Jun Sakai, Eiichiro Fukusaki, Yasuyoshi Okuno and Iwao Nakatsuka and has published in prestigious journals such as European Journal of Pharmacology, Toxicology and Applied Pharmacology and Toxicological Sciences.

In The Last Decade

Takeshi Kunimatsu

22 papers receiving 514 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Takeshi Kunimatsu Japan 12 218 166 87 72 63 23 524
Hitoshi Funabashi Japan 12 279 1.3× 84 0.5× 53 0.6× 29 0.4× 35 0.6× 28 588
Michael Gill United States 13 139 0.6× 86 0.5× 48 0.6× 139 1.9× 12 0.2× 42 595
John B. Coleman United States 11 163 0.7× 34 0.2× 17 0.2× 70 1.0× 28 0.4× 17 590
Anita Annas Sweden 13 152 0.7× 90 0.5× 27 0.3× 9 0.1× 11 0.2× 25 514
Sainath R. Kotha United States 16 214 1.0× 137 0.8× 10 0.1× 56 0.8× 47 0.7× 26 722
Neil G. Carmichael United Kingdom 10 78 0.4× 144 0.9× 25 0.3× 44 0.6× 15 0.2× 16 356
Han Yu China 11 142 0.7× 117 0.7× 19 0.2× 14 0.2× 10 0.2× 26 455
Moslem Abolhassani Iran 15 177 0.8× 106 0.6× 36 0.4× 11 0.2× 24 0.4× 38 552
David Achaintre France 9 427 2.0× 51 0.3× 10 0.1× 21 0.3× 21 0.3× 18 816
Takaki Seki Japan 16 255 1.2× 172 1.0× 29 0.3× 3 0.0× 96 1.5× 36 713

Countries citing papers authored by Takeshi Kunimatsu

Since Specialization
Citations

This map shows the geographic impact of Takeshi Kunimatsu's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Takeshi Kunimatsu with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Takeshi Kunimatsu more than expected).

Fields of papers citing papers by Takeshi Kunimatsu

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Takeshi Kunimatsu. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Takeshi Kunimatsu. The network helps show where Takeshi Kunimatsu may publish in the future.

Co-authorship network of co-authors of Takeshi Kunimatsu

This figure shows the co-authorship network connecting the top 25 collaborators of Takeshi Kunimatsu. A scholar is included among the top collaborators of Takeshi Kunimatsu based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Takeshi Kunimatsu. Takeshi Kunimatsu is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Kunimatsu, Takeshi, et al.. (2018). The in vitro pharmacology and non-clinical cardiovascular safety studies of a novel 5-HT 4 receptor agonist, DSP-6952. European Journal of Pharmacology. 826. 96–105. 10 indexed citations
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Takasuna, Kiyoshi, Keiichi Asakura, Seiichi Araki, et al.. (2016). Comprehensive in vitro cardiac safety assessment using human stem cell technology: Overview of CSAHi HEART initiative. Journal of Pharmacological and Toxicological Methods. 83. 42–54. 45 indexed citations
4.
Honda, Yayoi, et al.. (2014). Availability of human induced pluripotent stem cell-derived cardiomyocytes in assessment of drug potential for QT prolongation. Toxicology and Applied Pharmacology. 278(1). 72–77. 44 indexed citations
5.
Nishizato, Yohei, et al.. (2014). Translational research into species differences of endocrine toxicity via steroidogenesis inhibition by SMP-028 — For human safety in clinical study. Toxicology and Applied Pharmacology. 276(3). 213–219. 2 indexed citations
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Nishizato, Yohei, et al.. (2013). Effect of SMP-028 on steroidogenesis in rats; mechanism of toxicological events on endocrine organs of rats. Toxicology in Vitro. 28(3). 397–402. 3 indexed citations
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Inada, Hiroshi, Izuru Miyawaki, Takeshi Kunimatsu, et al.. (2012). Evaluation of ovarian toxicity of mono-(2-ethylhexyl) phthalate (MEHP) using cultured rat ovarian follicles. The Journal of Toxicological Sciences. 37(3). 483–490. 55 indexed citations
11.
Bando, Kiyoko, Takeshi Kunimatsu, Jun Sakai, et al.. (2010). GC‐MS‐based metabolomics reveals mechanism of action for hydrazine induced hepatotoxicity in rats. Journal of Applied Toxicology. 31(6). 524–535. 86 indexed citations
12.
Bando, Kiyoko, et al.. (2010). Comparison of potential risks of lactic acidosis induction by biguanides in rats. Regulatory Toxicology and Pharmacology. 58(1). 155–160. 24 indexed citations
13.
Bando, Kiyoko, Takeshi Kunimatsu, Jun Sakai, et al.. (2010). Influences of biofluid sample collection and handling procedures on GC–MS based metabolomic studies. Journal of Bioscience and Bioengineering. 110(4). 491–499. 47 indexed citations
14.
Kunimatsu, Takeshi, Juki Kimura, Hitoshi Funabashi, Tadashi Inoue, & Takaki Seki. (2010). The antipsychotics haloperidol and chlorpromazine increase bone metabolism and induce osteopenia in female rats. Regulatory Toxicology and Pharmacology. 58(3). 360–368. 24 indexed citations
15.
Kunimatsu, Takeshi, Tomoya Yamada, Kaori Miyata, et al.. (2004). Evaluation for reliability and feasibility of the draft protocol for the enhanced rat 28-day subacute study (OECD Guideline 407) using androgen antagonist flutamide. Toxicology. 200(1). 77–89. 34 indexed citations
16.
Kunimatsu, Takeshi, et al.. (2002). Lack of (Anti-) Androgenic or Estrogenic Effects of Three Pyrethroids (Esfenvalerate, Fenvalerate, and Permethrin) in the Hershberger and Uterotrophic Assays. Regulatory Toxicology and Pharmacology. 35(2). 227–237. 47 indexed citations
17.
Yamada, Tomoya, Takeshi Kunimatsu, Yasuyoshi Okuno, et al.. (2001). Dissection and weighing of accessory sex glands after formalin fixation, and a 5-day assay using young mature rats are reliable and feasible in the Hershberger assay. Toxicology. 162(2). 103–119. 21 indexed citations
18.
Kunimatsu, Takeshi, Tomoya Yamada, Tokuo Sukata, et al.. (2000). Evaluation of a 5-day Hershberger assay using young mature male rats. Methyltestosterone and p,p'-DDE, but not fenitrothion, exhibited androgenic or antiandrogenic activity in vivo.. The Journal of Toxicological Sciences. 25(5). 403–415. 34 indexed citations
19.
Kunimatsu, Takeshi, et al.. (1996). Immunotoxicological Insignificance of Fenitrothion in Mice and Rats. Toxicological Sciences. 33(2). 246–253. 1 indexed citations
20.
Okuno, Yasuyoshi, Takeshi Kunimatsu, Tomoyuki Watanabe, et al.. (1996). Effect of simultaneous treatment of large amounts of vitamin A and thiourea on thyroidal iodine uptake and organification in rats.. Journal of Toxicologic Pathology. 9(4). 385–390. 5 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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