Takehiko Maeda

4.4k total citations
138 papers, 3.6k citations indexed

About

Takehiko Maeda is a scholar working on Cellular and Molecular Neuroscience, Molecular Biology and Electrical and Electronic Engineering. According to data from OpenAlex, Takehiko Maeda has authored 138 papers receiving a total of 3.6k indexed citations (citations by other indexed papers that have themselves been cited), including 54 papers in Cellular and Molecular Neuroscience, 43 papers in Molecular Biology and 42 papers in Electrical and Electronic Engineering. Recurrent topics in Takehiko Maeda's work include Neuroscience and Neuropharmacology Research (30 papers), Pain Mechanisms and Treatments (28 papers) and Radio Frequency Integrated Circuit Design (15 papers). Takehiko Maeda is often cited by papers focused on Neuroscience and Neuropharmacology Research (30 papers), Pain Mechanisms and Treatments (28 papers) and Radio Frequency Integrated Circuit Design (15 papers). Takehiko Maeda collaborates with scholars based in Japan, United States and Indonesia. Takehiko Maeda's co-authors include Shiroh Kishioka, Norikazu Kiguchi, Yuka Kobayashi, Yohji Fukazawa, Toshiaki Kume, Shun Shimohama, Jun Kimura, Shuji Kaneko, Akinori Akaike and Masanori Ozaki and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Biological Chemistry and Neuron.

In The Last Decade

Takehiko Maeda

130 papers receiving 3.5k citations

Peers

Takehiko Maeda

CMN

PHYSI

PHARM

EEE

Satoshi Imai Japan

Qing Tian China

Michael J. M. Fischer Germany

Hiroaki Fukumoto Japan

Hong Qing China

Kenjiro Mori Japan

Shuhua Chen China

Xiaowei Chen China

Choong Hyun Lee South Korea

Satoshi Imai Japan

Takehiko Maeda

1.7k ×1.2

789 ×0.6

CMN

991 ×0.9

PHYSI

433 ×0.8

PHARM

104 ×0.3

EEE

Citations per year, relative to Takehiko Maeda Takehiko Maeda (= 1×) peers Satoshi Imai

Countries citing papers authored by Takehiko Maeda

Since Specialization

Citations

This map shows the geographic impact of Takehiko Maeda's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Takehiko Maeda with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Takehiko Maeda more than expected).

Fields of papers citing papers by Takehiko Maeda

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Takehiko Maeda. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Takehiko Maeda. The network helps show where Takehiko Maeda may publish in the future.

Co-authorship network of co-authors of Takehiko Maeda

This figure shows the co-authorship network connecting the top 25 collaborators of Takehiko Maeda. A scholar is included among the top collaborators of Takehiko Maeda based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Takehiko Maeda. Takehiko Maeda is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Kawahara, Kohichi, Takuya Hasegawa, Koji Sato, et al.. (2024). Truncated GPNMB , a microglial transmembrane protein, serves as a scavenger receptor for oligomeric β‐amyloid peptide _1‐42 in primary type 1 microglia. Journal of Neurochemistry. 168(7). 1317–1339. 1 indexed citations

Hasegawa, Takuya, Kohichi Kawahara, Koji Sato, Yoshihide Asano, & Takehiko Maeda. (2024). Characterization of a Cancer-Induced Bone Pain Model for Use as a Model of Cancer Cachexia. Current Issues in Molecular Biology. 46(12). 13364–13382. 2 indexed citations

Yamada, Daisuke, Kohichi Kawahara, & Takehiko Maeda. (2016). mTORC1 is a critical mediator of oncogenic Semaphorin3A signaling. Biochemical and Biophysical Research Communications. 476(4). 475–480. 10 indexed citations

Yamada, Daisuke, et al.. (2016). Autocrine Semaphorin3A signaling is essential for the maintenance of stem-like cells in lung cancer. Biochemical and Biophysical Research Communications. 480(3). 375–379. 12 indexed citations

Kiguchi, Norikazu, et al.. (2015). Peripheral interleukin-4 ameliorates inflammatory macrophage-dependent neuropathic pain. Pain. 156(4). 684–693. 85 indexed citations

Kiguchi, Norikazu, Takehiko Maeda, Yuka Kobayashi, Fumihiro Saika, & Shiroh Kishioka. (2009). Chapter 14 Involvement of Inflammatory Mediators in Neuropathic Pain Caused by Vincristine. International review of neurobiology. 85. 179–190. 61 indexed citations

Kiguchi, Norikazu, Takehiko Maeda, Mie Tsuruga, et al.. (2007). Involvement of spinal Met–enkephalin in nicotine-induced antinociception in mice. Brain Research. 1189. 70–77. 20 indexed citations

Maeda, Takehiko, Wakako Hamabe, Yuan Gao, et al.. (2005). Morphine has an antinociceptive effect through activation of the okadaic-acid-sensitive Ser/Thr protein phosphatases PP2A and PP5 estimated by tail-pinch test in mice. Brain Research. 1056(2). 191–199. 17 indexed citations

Fukazawa, Yohji, Takehiko Maeda, Wakako Hamabe, et al.. (2005). Activation of Spinal Anti-analgesic System Following Electroacupuncture Stimulation in Rats. Journal of Pharmacological Sciences. 99(4). 408–414. 21 indexed citations

10.

Maeda, Takehiko, et al.. (2002). Naloxone-Precipitated Morphine Withdrawal Elicits Increases in c-fos mRNA Expression in Restricted Regions of the Infant Rat Brain. The Japanese Journal of Pharmacology. 90(3). 270–275. 9 indexed citations

11.

Kaneko, H, Yuichiro Otsuka, Takehiko Maeda, et al.. (2001). Reassessment of monoethylglycinexylidide as preoperative liver function test in a rat model of liver cirrhosis and man. Clinical and Experimental Medicine. 1(1). 19–26. 5 indexed citations

12.

Nishikawa, Hiroyuki, Hiroshi Katsuki, Akinori Akaike, et al.. (2000). p75-mediated neuroprotection by NGF against glutamate cytotoxicity in cortical cultures. Brain Research. 852(2). 279–289. 74 indexed citations

13.

Akaike, A, Hiroshi Katsuki, Toshiaki Kume, & Takehiko Maeda. (1999). Reactive oxygen species in NMDA receptor-mediated glutamate neurotoxicity. Parkinsonism & Related Disorders. 5(4). 203–207. 17 indexed citations

14.

Akaike, Akinori, et al.. (1998). Bifemelane Protects Cultured Cortical Neurons Against N-Methyl-D-aspartate Receptor-Mediated Glutamate Cytotoxicity. The Japanese Journal of Pharmacology. 76(3). 313–316. 3 indexed citations

15.

Maeda, Takehiko, et al.. (1997). Involvement of M2 receptor in an enhancement of long-term potentiation by carbachol in Schaffer collateral-CA1 synapses of hippocampal slices. Neuroscience Research. 27(2). 175–180. 38 indexed citations

16.

Kaneko, Satoshi, Takehiko Maeda, Toshiaki Kume, et al.. (1997). Nicotine protects cultured cortical neurons against glutamate-induced cytotoxicity via α7-neuronal receptors and neuronal CNS receptors. Brain Research. 765(1). 135–140. 170 indexed citations

17.

Kihara, T, Shun Shimohama, Hideyuki Sawada, et al.. (1997). Nicotinic receptor stimulation protects neurons against β‐amyloid toxicity. Annals of Neurology. 42(2). 159–163. 272 indexed citations

18.

Maeda, Takehiko, et al.. (1996). Galanin inhibits long-term potentiation at Schaffer collateral-CA1 synapses in guinea-pig hippocampal slices. Neuroscience Letters. 212(1). 21–24. 45 indexed citations

19.

Maeda, Takehiko, Shuji Kaneko, & Masamichi Satoh. (1993). Bidirectional modulation of long-term potentiation by carbachol via M1 and M2 muscarinic receptors in guinea pig hippocampal mossy fiber-CA3 synapses. Brain Research. 619(1-2). 324–330. 24 indexed citations

20.

Maeda, Takehiko, et al.. (1989). AERODYNAMIC DRAG OF SHINKANSEN ELECTRIC CARS (SERIES 0, SERIES 200, SERIES 100). Quarterly Report of Rtri. 30(1). 9 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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