Tai Akera

5.5k total citations
192 papers, 4.5k citations indexed

About

Tai Akera is a scholar working on Molecular Biology, Cardiology and Cardiovascular Medicine and Cellular and Molecular Neuroscience. According to data from OpenAlex, Tai Akera has authored 192 papers receiving a total of 4.5k indexed citations (citations by other indexed papers that have themselves been cited), including 121 papers in Molecular Biology, 82 papers in Cardiology and Cardiovascular Medicine and 33 papers in Cellular and Molecular Neuroscience. Recurrent topics in Tai Akera's work include Cardiac electrophysiology and arrhythmias (67 papers), Ion channel regulation and function (56 papers) and Ion Transport and Channel Regulation (40 papers). Tai Akera is often cited by papers focused on Cardiac electrophysiology and arrhythmias (67 papers), Ion channel regulation and function (56 papers) and Ion Transport and Channel Regulation (40 papers). Tai Akera collaborates with scholars based in United States, Japan and Canada. Tai Akera's co-authors include Theodore M. Brody, Thomas Brody, Ming Huey Lin, Thomas Tobin, Kyosuke Temma, Min Soo Kim, David D. Ku, Yuk-Chow Ng, Brody Tm and Paul M. Stemmer and has published in prestigious journals such as Science, Proceedings of the National Academy of Sciences and Journal of Biological Chemistry.

In The Last Decade

Tai Akera

191 papers receiving 4.1k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Tai Akera United States 34 2.7k 1.3k 913 572 352 192 4.5k
Taku Nagao Japan 36 2.6k 1.0× 968 0.7× 892 1.0× 474 0.8× 307 0.9× 188 4.4k
George I. Drummond Canada 38 2.9k 1.1× 524 0.4× 642 0.7× 841 1.5× 339 1.0× 101 4.5k
Théophile Godfraind Belgium 34 2.2k 0.8× 1.4k 1.0× 789 0.9× 1.3k 2.2× 212 0.6× 176 3.8k
A. Fleckenstein Germany 38 2.9k 1.1× 2.6k 1.9× 1.5k 1.6× 926 1.6× 458 1.3× 192 6.2k
Mohammed A. Matlib United States 30 2.1k 0.8× 1.2k 0.9× 708 0.8× 539 0.9× 335 1.0× 68 3.1k
Amir Askari United States 37 4.0k 1.5× 614 0.5× 428 0.5× 549 1.0× 263 0.7× 131 5.3k
Chiu‐Yin Kwan Canada 35 2.2k 0.8× 533 0.4× 765 0.8× 791 1.4× 141 0.4× 187 3.9k
D. Palm Germany 35 2.0k 0.7× 451 0.3× 678 0.7× 624 1.1× 99 0.3× 171 4.1k
Nobuyuki Yanagihara Japan 34 1.9k 0.7× 716 0.5× 1.0k 1.1× 1.2k 2.2× 205 0.6× 183 4.2k
Ming‐Jai Su Taiwan 35 1.8k 0.7× 1.2k 0.9× 393 0.4× 555 1.0× 394 1.1× 182 4.3k

Countries citing papers authored by Tai Akera

Since Specialization
Citations

This map shows the geographic impact of Tai Akera's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Tai Akera with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Tai Akera more than expected).

Fields of papers citing papers by Tai Akera

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Tai Akera. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Tai Akera. The network helps show where Tai Akera may publish in the future.

Co-authorship network of co-authors of Tai Akera

This figure shows the co-authorship network connecting the top 25 collaborators of Tai Akera. A scholar is included among the top collaborators of Tai Akera based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Tai Akera. Tai Akera is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Taniguchi, Kazumi, Takashi Murayama, Tsuyoshi Uchide, et al.. (2003). Subcellular distribution of ryanodine receptors in the cardiac muscle of carp (Cyprinus carpio). American Journal of Physiology-Regulatory, Integrative and Comparative Physiology. 285(3). R601–R609. 17 indexed citations
2.
Uchide, Tsuyoshi, et al.. (2000). Anti-muscarinic actions of mitoxantrone in isolated heart muscles of guinea pigs. European Journal of Pharmacology. 407(1-2). 183–189. 9 indexed citations
3.
OYAMADA, Toshifumi, et al.. (1998). Doxorubicin-induced late cardiotoxicity : relation to impairment of SR Ca^ release.. The Journal of Toxicological Sciences. 23(4). 305. 1 indexed citations
4.
Temma, Kyosuke, et al.. (1997). Ca2+ overloading causes the negative inotropic effect of doxorubicin in myocytes isolated from guinea-pig hearts. European Journal of Pharmacology. 322(2-3). 235–242. 16 indexed citations
5.
Temma, Kyosuke, et al.. (1996). Doxorubicin alters Ca2+ transients but fails to change Ca2+ sensitivity of contractile proteins. Environmental Toxicology and Pharmacology. 1(2). 131–139. 13 indexed citations
6.
Temma, Kyosuke, et al.. (1992). Doxorubicin: an antagonist of muscarinic receptors in guinea pig heart. European Journal of Pharmacology. 220(1). 63–69. 16 indexed citations
7.
Akera, Tai & Yuk-Chow Ng. (1991). Digitalis sensitivity of Na+,K+-ATPase, myocytes and the heart. Life Sciences. 48(2). 97–106. 41 indexed citations
8.
Ishizuka, N, Yoshihiro Fukushima, Osamu Urayama, & Tai Akera. (1991). Na+,K+-ATPase inhibition by an endogenous peptide, SPAI-1, isolated from porcine duodenum. Biochimica et Biophysica Acta (BBA) - Biomembranes. 1069(2). 259–266. 7 indexed citations
9.
Temma, Kyosuke, Hiroshi Kondo, & Tai Akera. (1991). Developmental changes in inotropic actions of neurotoxins observed in ventricular muscle of rat heart. Canadian Journal of Physiology and Pharmacology. 69(4). 494–500. 2 indexed citations
10.
11.
Berlin, Joshua R., Tai Akera, & Thomas Brody. (1986). Lack of pharmacodynamic interactions between quinidine and digoxin in isolated atrial muscle of guinea pig heart.. Journal of Pharmacology and Experimental Therapeutics. 238(2). 632–641. 3 indexed citations
12.
Akera, Tai, et al.. (1983). Decreases in active sodium pumping sites and their interaction with ouabain caused by low Na+ incubation of isolated guinea-pig atrial muscle.. Journal of Pharmacology and Experimental Therapeutics. 225(3). 660–666. 7 indexed citations
13.
Akera, Tai, et al.. (1980). Inotropic actions of doxorubicin in isolated guinea-pig atria: evidence for lack of involvement of Na+,K+-adenosine triphosphatase.. Journal of Pharmacology and Experimental Therapeutics. 214(2). 368–374. 16 indexed citations
14.
Akera, Tai, et al.. (1979). Differential effect of potassium on the action of digoxin and digoxigenin in guinea-pig heart. European Journal of Pharmacology. 57(4). 343–351. 12 indexed citations
15.
Chang, Chia‐Cheng, James E. Trosko, & Tai Akera. (1978). Characterization of ultraviolet light-induced ouabain-resistant mutations in chinese hamster cells. Mutation Research/Fundamental and Molecular Mechanisms of Mutagenesis. 51(1). 85–98. 42 indexed citations
16.
17.
Akera, Tai, et al.. (1976). Effects of grayanotoxin I on cardiac Na + K + -adenosine triphosphatase activity, transmembrane potential and myocardial contractile force.. Journal of Pharmacology and Experimental Therapeutics. 199(1). 247–254. 17 indexed citations
18.
Akera, Tai, et al.. (1975). The effects of age on the development of tolerance to and physical dependence on morphine in rats.. Journal of Pharmacology and Experimental Therapeutics. 192(3). 506–512. 33 indexed citations
19.
Akera, Tai, et al.. (1973). LACK OF RELATIONSHIP BETWEEN BRAIN (Na+ + K+)-ACTIVATED ADENOSINE TRIPHOSPHATASE AND THE DEVELOPMENT OF TOLERANCE TO ETHANOL IN RATS. Journal of Pharmacology and Experimental Therapeutics. 185(3). 594–601. 24 indexed citations
20.
Akera, Tai & Theodore M. Brody. (1968). Inhibition of Brain Sodium- and Potassium-Stimulated Adenosine Triphosphatase Activity by Chlorpromazine Free Radical. Molecular Pharmacology. 4(6). 600–612. 24 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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