Susan J. Faas

2.4k total citations
37 papers, 1.9k citations indexed

About

Susan J. Faas is a scholar working on Immunology, Molecular Biology and Genetics. According to data from OpenAlex, Susan J. Faas has authored 37 papers receiving a total of 1.9k indexed citations (citations by other indexed papers that have themselves been cited), including 24 papers in Immunology, 13 papers in Molecular Biology and 7 papers in Genetics. Recurrent topics in Susan J. Faas's work include T-cell and B-cell Immunology (11 papers), Immunotherapy and Immune Responses (7 papers) and Complement system in diseases (7 papers). Susan J. Faas is often cited by papers focused on T-cell and B-cell Immunology (11 papers), Immunotherapy and Immune Responses (7 papers) and Complement system in diseases (7 papers). Susan J. Faas collaborates with scholars based in United States, Switzerland and Canada. Susan J. Faas's co-authors include Anjana Rao, Harvey Cantor, Russell P. Rother, Barbara B. Knowles, Dayang Wu, Ruurd Torensma, Massimo Trucco, Roxanne Cofiell, Anke Kretz-Rommel and Camille L. Bedrosian and has published in prestigious journals such as Nature, Cell and The Journal of Experimental Medicine.

In The Last Decade

Susan J. Faas

36 papers receiving 1.9k citations

Peers

Susan J. Faas

IMMUN

HEMAT

ONCOL

GENET

Dorothee Wernet Germany

Michael F. Concino United States

Fabian Flores‐Borja United Kingdom

Joachim R. Grün Germany

Guglielmo M. Venturi United States

Jonathan S. Maltzman United States

B Bellon France

Stéphanie Graff‐Dubois France

Marina Botto United Kingdom

Kenneth J. Katschke United States

Dorothee Wernet Germany

Susan J. Faas

1.1k ×0.9

IMMUN

717 ×1.1

252 ×0.7

HEMAT

546 ×2.2

ONCOL

177 ×0.9

GENET

Citations per year, relative to Susan J. Faas Susan J. Faas (= 1×) peers Dorothee Wernet

Countries citing papers authored by Susan J. Faas

Since Specialization

Citations

This map shows the geographic impact of Susan J. Faas's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Susan J. Faas with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Susan J. Faas more than expected).

Fields of papers citing papers by Susan J. Faas

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Susan J. Faas. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Susan J. Faas. The network helps show where Susan J. Faas may publish in the future.

Co-authorship network of co-authors of Susan J. Faas

This figure shows the co-authorship network connecting the top 25 collaborators of Susan J. Faas. A scholar is included among the top collaborators of Susan J. Faas based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Susan J. Faas. Susan J. Faas is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Cammett, Tobin J., et al.. (2022). Exploratory Prognostic Biomarkers of Complement-Mediated Thrombotic Microangiopathy (CM-TMA) in Adults with Atypical Hemolytic Uremic Syndrome (aHUS): Analysis of a Phase III Study of Ravulizumab. Molecular Diagnosis & Therapy. 27(1). 61–74. 4 indexed citations

Mahadevan, Daruka, Mark C. Lanasa, Charles M. Farber, et al.. (2019). Phase I study of samalizumab in chronic lymphocytic leukemia and multiple myeloma: blockade of the immune checkpoint CD200. Journal for ImmunoTherapy of Cancer. 7(1). 227–227. 63 indexed citations

Cofiell, Roxanne, Anjli Kukreja, Yan Yan, et al.. (2015). Eculizumab reduces complement activation, inflammation, endothelial damage, thrombosis, and renal injury markers in aHUS. Blood. 125(21). 3253–3262. 143 indexed citations

Kwan, W. H., Min Yang, Krista Johnson, et al.. (2011). Anti-Complement Component C5 mAb Synergizes with CTLA4Ig to Inhibit Alloreactive T cells and Prolong Cardiac Allograft Survival in Mice. American Journal of Transplantation. 11(7). 1397–1406. 43 indexed citations

Rother, Russell P., J. Arp, J. Jiang, et al.. (2008). C5 Blockade with Conventional Immunosuppression Induces Long-Term Graft Survival in Presensitized Recipients. American Journal of Transplantation. 8(6). 1129–1142. 49 indexed citations

Wang, Hao, Jacqueline Arp, Weihua Liu, et al.. (2007). Inhibition of Terminal Complement Components in Presensitized Transplant Recipients Prevents Antibody-Mediated Rejection Leading to Long-Term Graft Survival and Accommodation. The Journal of Immunology. 179(7). 4451–4463. 78 indexed citations

Kretz-Rommel, Anke, Fenghua Qin, Naveen Dakappagari, et al.. (2007). In Vivo Targeting of Antigens to Human Dendritic Cells Through DC-SIGN Elicits Stimulatory Immune Responses and Inhibits Tumor Growth in Grafted Mouse Models. Journal of Immunotherapy. 30(7). 715–726. 71 indexed citations

Pereira, Cândida F., Ruurd Torensma, Konnie M. Hebeda, et al.. (2007). In Vivo Targeting of DC-SIGN-positive Antigen-presenting Cells in a Nonhuman Primate Model. Journal of Immunotherapy. 30(7). 705–714. 29 indexed citations

Küerten, Stefanie, Felix S. Lichtenegger, Susan J. Faas, et al.. (2006). MBP-PLP fusion protein-induced EAE in C57BL/6 mice. Journal of Neuroimmunology. 177(1-2). 99–111. 44 indexed citations

10.

Tacken, Paul J., I. Jolanda M. de Vries, Karlijn Gijzen, et al.. (2005). Effective induction of naive and recall T-cell responses by targeting antigen to human dendritic cells via a humanized anti–DC-SIGN antibody. Blood. 106(4). 1278–1285. 219 indexed citations

11.

Subudhi, Sumit K., Russell P. Rother, Susan J. Faas, et al.. (2001). Inducible Costimulator Regulates Th2-Mediated Inflammation, but Not Th2 Differentiation, in a Model of Allergic Airway Disease. The Journal of Immunology. 167(4). 1996–2003. 107 indexed citations

12.

Faas, Susan J., Deborah J. Reed, Dayang Wu, et al.. (2000). Primary Structure and Functional Characterization of a Soluble, Alternatively Spliced Form of B7-1. The Journal of Immunology. 164(12). 6340–6348. 43 indexed citations

13.

Faas, Susan J., et al.. (1996). Sequence‐specific priming and exonuclease‐released fluorescence detection of HLA‐DQB1 alleles. Tissue Antigens. 48(2). 97–112. 34 indexed citations

14.

Luppi, Patrizia, et al.. (1995). Genetic background and environment contribute synergistically to the onset of autoimmune diseases. Journal of Molecular Medicine. 73(8). 381–93. 36 indexed citations

15.

Faas, Susan J. & Massimo Trucco. (1994). The genes influencing the susceptibility to IDDM in humans. Journal of Endocrinological Investigation. 17(7). 477–495. 30 indexed citations

16.

Nakada, M T, et al.. (1990). Neonatal exposure to thymotropic gross murine leukemia virus induces virus-specific immunologic nonresponsiveness.. The Journal of Experimental Medicine. 172(6). 1765–1775. 26 indexed citations

17.

Wettstein, Peter J., et al.. (1988). SV40 T-antigen is a histocompatibility antigen of SV40-transgenic mice. Immunogenetics. 27(6). 436–441. 15 indexed citations

18.

Friedman, Steven, et al.. (1985). A subset of Ly-1 inducer T cell clones activates B cell proliferation but directly inhibits subsequent IgG secretion.. The Journal of Experimental Medicine. 161(4). 785–804. 15 indexed citations

19.

Rao, Anjana, Susan J. Faas, & Harvey Cantor. (1984). Activation specificity of arsonate-reactive T cell clones. Structural requirements for hapten recognition and comparison with monoclonal antibodies.. The Journal of Experimental Medicine. 159(2). 479–494. 53 indexed citations

20.

Rao, Anjana, et al.. (1983). Lysis of inducer T cell clones by activated macrophages and macrophage-like cell lines.. The Journal of Experimental Medicine. 158(4). 1243–1258. 16 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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