Subhankar Dolai

1.1k total citations
31 papers, 913 citations indexed

About

Subhankar Dolai is a scholar working on Cell Biology, Surgery and Molecular Biology. According to data from OpenAlex, Subhankar Dolai has authored 31 papers receiving a total of 913 indexed citations (citations by other indexed papers that have themselves been cited), including 22 papers in Cell Biology, 17 papers in Surgery and 9 papers in Molecular Biology. Recurrent topics in Subhankar Dolai's work include Pancreatic function and diabetes (17 papers), Cellular transport and secretion (16 papers) and Calcium signaling and nucleotide metabolism (5 papers). Subhankar Dolai is often cited by papers focused on Pancreatic function and diabetes (17 papers), Cellular transport and secretion (16 papers) and Calcium signaling and nucleotide metabolism (5 papers). Subhankar Dolai collaborates with scholars based in Canada, India and United States. Subhankar Dolai's co-authors include Subrata Adak, Rajesh Kumar Yadav, Swati Pal, Herbert Y. Gaisano, Li Xie, Tao Liang, Youhou Kang, Tairan Qin, Dan Zhu and Peter Thorn and has published in prestigious journals such as Journal of Biological Chemistry, Gastroenterology and PLoS ONE.

In The Last Decade

Subhankar Dolai

31 papers receiving 908 citations

Peers

Subhankar Dolai
Fannie W. Chen United States
Martina Wirth United Kingdom
Judy K. Cundiff United States
Subhankar Dolai
Citations per year, relative to Subhankar Dolai Subhankar Dolai (= 1×) peers Ryoko Akai

Countries citing papers authored by Subhankar Dolai

Since Specialization
Citations

This map shows the geographic impact of Subhankar Dolai's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Subhankar Dolai with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Subhankar Dolai more than expected).

Fields of papers citing papers by Subhankar Dolai

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Subhankar Dolai. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Subhankar Dolai. The network helps show where Subhankar Dolai may publish in the future.

Co-authorship network of co-authors of Subhankar Dolai

This figure shows the co-authorship network connecting the top 25 collaborators of Subhankar Dolai. A scholar is included among the top collaborators of Subhankar Dolai based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Subhankar Dolai. Subhankar Dolai is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Dolai, Subhankar, et al.. (2024). Ru-Catalyzed Redox-Neutral Coupling of N-Chlorobenzamides with Unsymmetrical Alkynes in Water. The Journal of Organic Chemistry. 89(22). 16850–16864. 1 indexed citations
2.
Takahashi, Toshimasa, Yifan Miao, Fei Kang, Subhankar Dolai, & Herbert Y. Gaisano. (2020). Susceptibility Factors and Cellular Mechanisms Underlying Alcoholic Pancreatitis. Alcoholism Clinical and Experimental Research. 44(4). 777–789. 11 indexed citations
3.
Dolai, Subhankar, Toshimasa Takahashi, Tairan Qin, et al.. (2020). Pancreas-specific SNAP23 depletion prevents pancreatitis by attenuating pathological basolateral exocytosis and formation of trypsin-activating autolysosomes. Autophagy. 17(10). 3068–3081. 26 indexed citations
4.
Liang, Tao, Tairan Qin, Fei Kang, et al.. (2020). SNAP23 depletion enables more SNAP25/calcium channel excitosome formation to increase insulin exocytosis in type 2 diabetes. JCI Insight. 5(3). 12 indexed citations
5.
Dolai, Subhankar, Tao Liang, Abrahim I. Orabi, et al.. (2018). Pancreatitis-Induced Depletion of Syntaxin 2 Promotes Autophagy and Increases Basolateral Exocytosis. Gastroenterology. 154(6). 1805–1821.e5. 49 indexed citations
6.
Dolai, Subhankar, Tao Liang, Abrahim I. Orabi, et al.. (2017). Depletion of the membrane-fusion regulator Munc18c attenuates caerulein hyperstimulation–induced pancreatitis. Journal of Biological Chemistry. 293(7). 2510–2522. 13 indexed citations
7.
Zhu, Dan, Li Xie, Youhou Kang, et al.. (2017). Syntaxin 2 Acts as Inhibitory SNARE for Insulin Granule Exocytosis. Diabetes. 66(4). 948–959. 18 indexed citations
8.
Liang, Tao, Tairan Qin, Li Xie, et al.. (2016). New Roles of Syntaxin-1A in Insulin Granule Exocytosis and Replenishment. Journal of Biological Chemistry. 292(6). 2203–2216. 29 indexed citations
9.
Xie, Li, Subhankar Dolai, Youhou Kang, et al.. (2016). Syntaxin-3 Binds and Regulates Both R- and L-Type Calcium Channels in Insulin-Secreting INS-1 832/13 Cells. PLoS ONE. 11(2). e0147862–e0147862. 11 indexed citations
10.
Xie, Li, Dan Zhu, Subhankar Dolai, et al.. (2015). Syntaxin-4 mediates exocytosis of pre-docked and newcomer insulin granules underlying biphasic glucose-stimulated insulin secretion in human pancreatic beta cells. Diabetologia. 58(6). 1250–1259. 34 indexed citations
11.
Dolai, Subhankar, et al.. (2012). Effects of Ethanol Metabolites on Exocytosis of Pancreatic Acinar Cells in Rats. Gastroenterology. 143(3). 832–843.e7. 27 indexed citations
12.
Zhu, Dan, Yi Zhang, Patrick Lam, et al.. (2012). Dual Role of VAMP8 in Regulating Insulin Exocytosis and Islet β Cell Growth. Cell Metabolism. 16(2). 238–249. 66 indexed citations
14.
Dolai, Subhankar, Swati Pal, Rajesh Kumar Yadav, & Subrata Adak. (2011). Endoplasmic Reticulum Stress-induced Apoptosis in Leishmania through Ca2+-dependent and Caspase-independent Mechanism. Journal of Biological Chemistry. 286(15). 13638–13646. 93 indexed citations
15.
Yadav, Rajesh Kumar, Swati Pal, Subhankar Dolai, & Subrata Adak. (2011). Role of proximal methionine residues in Leishmania major peroxidase. Archives of Biochemistry and Biophysics. 515(1-2). 21–27. 5 indexed citations
16.
Pal, Swati, Subhankar Dolai, Rajesh Kumar Yadav, & Subrata Adak. (2010). Ascorbate Peroxidase from Leishmania major Controls the Virulence of Infective Stage of Promastigotes by Regulating Oxidative Stress. PLoS ONE. 5(6). e11271–e11271. 62 indexed citations
17.
Dolai, Subhankar, Rajesh Kumar Yadav, Swati Pal, & Subrata Adak. (2008). Leishmania major ascorbate peroxidase overexpression protects cells against reactive oxygen species-mediated cardiolipin oxidation. Free Radical Biology and Medicine. 45(11). 1520–1529. 55 indexed citations
18.
Yadav, Rajesh Kumar, Subhankar Dolai, Swati Pal, & Subrata Adak. (2008). Role of tryptophan-208 residue in cytochrome c oxidation by ascorbate peroxidase from Leishmania major-kinetic studies on Trp208Phe mutant and wild type enzyme. Biochimica et Biophysica Acta (BBA) - Proteins and Proteomics. 1784(5). 863–871. 29 indexed citations
19.
Chatterjee, Ratna, Subhankar Dolai, Subrata Adak, et al.. (2008). Chenopodium album seed extract-induced sperm cell death: exploration of a plausible pathway. Contraception. 77(6). 456–462. 14 indexed citations
20.
Dolai, Subhankar, Rajesh Kumar Yadav, Alok K. Datta, & Subrata Adak. (2006). Effect of thiocyanate on the peroxidase and pseudocatalase activities of Leishmania major ascorbate peroxidase. Biochimica et Biophysica Acta (BBA) - General Subjects. 1770(2). 247–256. 15 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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