Stuart Austin

5.4k total citations
85 papers, 4.5k citations indexed

About

Stuart Austin is a scholar working on Genetics, Molecular Biology and Ecology. According to data from OpenAlex, Stuart Austin has authored 85 papers receiving a total of 4.5k indexed citations (citations by other indexed papers that have themselves been cited), including 72 papers in Genetics, 55 papers in Molecular Biology and 33 papers in Ecology. Recurrent topics in Stuart Austin's work include Bacterial Genetics and Biotechnology (69 papers), Bacteriophages and microbial interactions (33 papers) and DNA Repair Mechanisms (28 papers). Stuart Austin is often cited by papers focused on Bacterial Genetics and Biotechnology (69 papers), Bacteriophages and microbial interactions (33 papers) and DNA Repair Mechanisms (28 papers). Stuart Austin collaborates with scholars based in United States, United Kingdom and Sweden. Stuart Austin's co-authors include Ann L. Abeles, Kurt Nordström, Brenda Youngren, Nat Sternberg, Yong-Fang Li, Stanley Friedman, Hans Jørgen Nielsen, Michael A. Davis, James A. Sawitzke and K. Martin and has published in prestigious journals such as Nature, Cell and Proceedings of the National Academy of Sciences.

In The Last Decade

Stuart Austin

85 papers receiving 4.3k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Stuart Austin United States 40 3.3k 3.0k 1.6k 762 681 85 4.5k
Anthony P. Pugsley France 41 2.8k 0.8× 2.5k 0.8× 999 0.6× 562 0.7× 366 0.5× 78 4.3k
Kathleen Postle United States 38 2.8k 0.8× 3.0k 1.0× 773 0.5× 780 1.0× 814 1.2× 62 4.8k
E. Gerhart H. Wagner Sweden 40 3.1k 0.9× 4.8k 1.6× 2.1k 1.4× 453 0.6× 928 1.4× 78 6.2k
Dhruba K. Chattoraj United States 33 2.0k 0.6× 2.2k 0.7× 979 0.6× 425 0.6× 294 0.4× 99 3.2k
Thomas A. Bickle Switzerland 44 2.3k 0.7× 4.6k 1.6× 1.7k 1.0× 332 0.4× 586 0.9× 111 5.6k
Eiichi Ohtsubo Japan 46 3.3k 1.0× 4.5k 1.5× 2.0k 1.2× 828 1.1× 2.0k 2.9× 149 7.0k
Robert W. Simons United States 30 2.3k 0.7× 3.5k 1.2× 1.3k 0.8× 196 0.3× 343 0.5× 49 4.4k
Udo Bläsi Austria 46 3.0k 0.9× 4.8k 1.6× 2.7k 1.7× 395 0.5× 381 0.6× 131 6.2k
Nadim Majdalani United States 24 2.6k 0.8× 3.0k 1.0× 1.3k 0.8× 408 0.5× 258 0.4× 40 4.2k
Simon L. Dove United States 36 2.0k 0.6× 2.9k 1.0× 1.1k 0.7× 613 0.8× 211 0.3× 68 3.7k

Countries citing papers authored by Stuart Austin

Since Specialization
Citations

This map shows the geographic impact of Stuart Austin's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Stuart Austin with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Stuart Austin more than expected).

Fields of papers citing papers by Stuart Austin

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Stuart Austin. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Stuart Austin. The network helps show where Stuart Austin may publish in the future.

Co-authorship network of co-authors of Stuart Austin

This figure shows the co-authorship network connecting the top 25 collaborators of Stuart Austin. A scholar is included among the top collaborators of Stuart Austin based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Stuart Austin. Stuart Austin is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Youngren, Brenda, Hans Jørgen Nielsen, Suckjoon Jun, & Stuart Austin. (2014). The multifork Escherichia coli chromosome is a self-duplicating and self-segregating thermodynamic ring polymer. Genes & Development. 28(1). 71–84. 98 indexed citations
2.
Sawitzke, James A., et al.. (2012). The segregation of Escherichia coli minichromosomes constructed in vivo by recombineering. Plasmid. 67(2). 148–154. 2 indexed citations
3.
Chung, Young Sup, T. Brendler, Stuart Austin, & Alba Guarné. (2009). Structural insights into the cooperative binding of SeqA to a tandem GATC repeat. Nucleic Acids Research. 37(10). 3143–3152. 14 indexed citations
4.
Nielsen, Hans Jørgen, et al.. (2006). The Escherichia coli chromosome is organized with the left and right chromosome arms in separate cell halves. Molecular Microbiology. 62(2). 331–338. 154 indexed citations
5.
Guarné, Alba, T. Brendler, Qinghai Zhao, et al.. (2005). Crystal structure of a SeqA–N filament: implications for DNA replication and chromosome organization. The EMBO Journal. 24(8). 1502–1511. 46 indexed citations
6.
Camara, Johanna E, Adam M. Breier, T. Brendler, et al.. (2005). Hda inactivation of DnaA is the predominant mechanism preventing hyperinitiation of Escherichia coli DNA replication. EMBO Reports. 6(8). 736–741. 68 indexed citations
7.
Li, Yong-Fang, Brenda Youngren, Kirill V. Sergueev, & Stuart Austin. (2003). Segregation of the Escherichia coli chromosome terminus. Molecular Microbiology. 50(3). 825–834. 77 indexed citations
8.
Li, Yong-Fang & Stuart Austin. (2002). The P1 plasmid in action: time-lapse photomicroscopy reveals some unexpected aspects of plasmid partition. Plasmid. 48(3). 174–178. 25 indexed citations
9.
Li, Yong-Fang & Stuart Austin. (2002). The P1 plasmid is segregated to daughter cells by a ‘capture and ejection’ mechanism coordinated with Escherichia coli cell division. Molecular Microbiology. 46(1). 63–74. 61 indexed citations
10.
Li, Yong-Fang, Kirill V. Sergueev, & Stuart Austin. (2002). The segregation of the Escherichia coli origin and terminus of replication. Molecular Microbiology. 46(4). 985–996. 108 indexed citations
11.
Sergueev, Kirill V., et al.. (2002). E.coli Cell-cycle Regulation by Bacteriophage Lambda. Journal of Molecular Biology. 324(2). 297–307. 37 indexed citations
12.
Lim, Seah H., et al.. (1999). Expression of testicular genes in haematological malignancies. British Journal of Cancer. 81(7). 1162–1164. 44 indexed citations
13.
14.
Davis, Michael A., Lyndsay Radnedge, K. Martin, et al.. (1996). The P1 ParA protein and its ATPase activity play a direct role in the segregation of plasmid copies to daughter cells. Molecular Microbiology. 21(5). 1029–1036. 73 indexed citations
15.
Hayes, Finbarr, Lyndsay Radnedge, Michael A. Davis, & Stuart Austin. (1994). The homologous operons for P1 and P7 plasmid partition are autoregulated from dissimilar operator sites. Molecular Microbiology. 11(2). 249–260. 55 indexed citations
16.
Brendler, T., et al.. (1991). Unique sequence requirements for the P1 plasmid replication origin. Research in Microbiology. 142(2-3). 209–216. 3 indexed citations
17.
Austin, Stuart & Kurt Nordström. (1990). Partition-mediated incompatibility of bacterial plasmids. Cell. 60(3). 351–354. 116 indexed citations
18.
Austin, Stuart, et al.. (1989). Plasmid-partition functions of the P7 prophage. Journal of Molecular Biology. 209(3). 393–406. 59 indexed citations
19.
Friedman, Stanley & Stuart Austin. (1988). The P1 plasmid-partition system synthesizes two essential proteins from an autoregulated operon. Plasmid. 19(2). 103–112. 100 indexed citations
20.
Austin, Stuart, et al.. (1987). The plasmid-maintenance functions of the P7 prophage. Plasmid. 18(1). 93–98. 9 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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