Stephen E. Kurtz

6.6k total citations
79 papers, 3.3k citations indexed

About

Stephen E. Kurtz is a scholar working on Molecular Biology, Hematology and Immunology. According to data from OpenAlex, Stephen E. Kurtz has authored 79 papers receiving a total of 3.3k indexed citations (citations by other indexed papers that have themselves been cited), including 38 papers in Molecular Biology, 28 papers in Hematology and 19 papers in Immunology. Recurrent topics in Stephen E. Kurtz's work include Acute Myeloid Leukemia Research (21 papers), Chronic Myeloid Leukemia Treatments (12 papers) and Protein Degradation and Inhibitors (11 papers). Stephen E. Kurtz is often cited by papers focused on Acute Myeloid Leukemia Research (21 papers), Chronic Myeloid Leukemia Treatments (12 papers) and Protein Degradation and Inhibitors (11 papers). Stephen E. Kurtz collaborates with scholars based in United States, India and Germany. Stephen E. Kurtz's co-authors include Andrew Blauvelt, David Shore, Arthur J. Lustig, Susan Lindquist, Shinji Kagami, J M Rossi, S Lindquist, Changsheng Guo, Mihail S. Iordanov and David E. Purdy and has published in prestigious journals such as Science, Cell and Proceedings of the National Academy of Sciences.

In The Last Decade

Stephen E. Kurtz

72 papers receiving 3.2k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Stephen E. Kurtz United States	25	1.5k	1.3k	560	558	400	79	3.3k
Jane Goodall United Kingdom	32	2.1k 1.4×	1.8k 1.4×	187 0.3×	306 0.5×	422 1.1×	82	4.8k
Junichiro Mizuguchi Japan	39	1.8k 1.2×	3.6k 2.8×	253 0.5×	274 0.5×	252 0.6×	129	5.5k
Avery August United States	38	1.8k 1.2×	2.6k 2.0×	626 1.1×	93 0.2×	139 0.3×	162	5.3k
Gavin F. Painter New Zealand	33	2.9k 1.9×	1.8k 1.4×	253 0.5×	85 0.2×	91 0.2×	95	5.2k
Laurent Genestier France	31	2.0k 1.3×	1.9k 1.4×	184 0.3×	75 0.1×	431 1.1×	64	4.6k
Ottmar Janßen Germany	42	2.3k 1.5×	3.0k 2.3×	224 0.4×	83 0.1×	165 0.4×	135	5.6k
Susan Wong United States	27	1.4k 0.9×	532 0.4×	114 0.2×	502 0.9×	187 0.5×	50	4.0k
M K Robinson United States	24	1.2k 0.8×	990 0.8×	190 0.3×	140 0.3×	212 0.5×	33	2.8k
Céline Eidenschenk United States	27	906 0.6×	2.1k 1.6×	242 0.4×	96 0.2×	131 0.3×	34	3.3k
Borbala Gesser Denmark	29	1.1k 0.7×	857 0.7×	259 0.5×	418 0.7×	31 0.1×	46	2.6k

Countries citing papers authored by Stephen E. Kurtz

Since Specialization

Citations

This map shows the geographic impact of Stephen E. Kurtz's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Stephen E. Kurtz with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Stephen E. Kurtz more than expected).

Fields of papers citing papers by Stephen E. Kurtz

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Stephen E. Kurtz. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Stephen E. Kurtz. The network helps show where Stephen E. Kurtz may publish in the future.

Co-authorship network of co-authors of Stephen E. Kurtz

This figure shows the co-authorship network connecting the top 25 collaborators of Stephen E. Kurtz. A scholar is included among the top collaborators of Stephen E. Kurtz based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Stephen E. Kurtz. Stephen E. Kurtz is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Eide, Christopher A., Stephen E. Kurtz, Nicola Long, et al.. (2026). DDX41-Mutated AML: A Case Report and Perspectives. JCO Precision Oncology. 10(1). e2500992–e2500992.

Eide, Christopher A., Stephen E. Kurtz, Andy Kaempf, et al.. (2025). A rapid gene expression profiler predicts tumor responsiveness and patient outcome for standard-of-care therapies in acute myeloid leukemia. Blood. 146(Supplement 1). 657–657.

Lachowiez, Curtis A., Joshua F. Zeidner, D. Peters, et al.. (2024). Influence of AML Differentiation State in Risk Stratification of Frontline Therapy with Hypomethylating Agents + Venetoclax in AML. Blood. 144(Supplement 1). 62–62.

Eide, Christopher A., Stephen E. Kurtz, Andy Kaempf, et al.. (2023). Clinical Correlates of Venetoclax-Based Combination Sensitivities to Augment Acute Myeloid Leukemia Therapy. Blood Cancer Discovery. 4(6). 452–467. 12 indexed citations

Rice, William G., Stephen B. Howell, Nasrin Rastgoo, et al.. (2022). Luxeptinib (CG-806) Targets FLT3 and Clusters of Kinases Operative in Acute Myeloid Leukemia. Molecular Cancer Therapeutics. 21(7). 1125–1135. 6 indexed citations

Kurtz, Stephen E., Christopher A. Eide, Andy Kaempf, et al.. (2022). Associating drug sensitivity with differentiation status identifies effective combinations for acute myeloid leukemia. Blood Advances. 6(10). 3062–3067. 6 indexed citations

Liu, Tingting, Xiaoguang Wang, Tamilla Nechiporuk, et al.. (2022). Dual BTK/SYK inhibition with CG-806 (luxeptinib) disrupts B-cell receptor and Bcl-2 signaling networks in mantle cell lymphoma. Cell Death and Disease. 13(3). 246–246. 15 indexed citations

Liu, Tingting, Duanchen Sun, Xiaoguang Wang, et al.. (2021). Pharmacologic Targeting of Mcl-1 Induces Mitochondrial Dysfunction and Apoptosis in B-Cell Lymphoma Cells in a TP53- and BAX- Dependent Manner. Clinical Cancer Research. 27(17). 4910–4922. 34 indexed citations

Eide, Christopher A., Stephen E. Kurtz, Andy Kaempf, et al.. (2020). Simultaneous kinase inhibition with ibrutinib and BCL2 inhibition with venetoclax offers a therapeutic strategy for acute myeloid leukemia. Leukemia. 34(9). 2342–2353. 16 indexed citations

10.

Nechiporuk, Tamilla, Stephen E. Kurtz, Olga Nikolova, et al.. (2019). The TP53 Apoptotic Network Is a Primary Mediator of Resistance to BCL2 Inhibition in AML Cells. Cancer Discovery. 9(7). 910–925. 222 indexed citations

11.

Zhang, Haijiao, Beth Wilmot, Daniel Bottomly, et al.. (2018). Biomarkers Predicting Venetoclax Sensitivity and Strategies for Venetoclax Combination Treatment. Blood. 132(Supplement 1). 175–175. 23 indexed citations

12.

Hodgson, Graeme, Liv Johannessen, Nisha Rajagopal, et al.. (2017). Sy-1365, a Potent and Selective CDK7 Inhibitor, Exhibits Anti-Tumor Activity in Preclinical Models of Hematologic Malignancies, and Demonstrates Interactions with the BCL-XL/BCL2 Mitochondrial Apoptosis Signaling Pathway in Leukemia. Blood. 130. 2651–2651. 5 indexed citations

13.

Zhāng, Qí, Lina Han, Ce Shi, et al.. (2016). Upregulation of MAPK/MCL-1 Maintaining Mitochondrial Oxidative Phosphorylation Confers Acquired Resistance to BCL-2 Inhibitor Venetoclax in AML. Blood. 128(22). 101–101. 5 indexed citations

14.

Guo, Changsheng, Joseph V. Lillis, Stephen E. Kurtz, et al.. (2009). Th17 Cytokines Stimulate CCL20 Expression in Keratinocytes In Vitro and In Vivo: Implications for Psoriasis Pathogenesis. Journal of Investigative Dermatology. 129(9). 2175–2183. 416 indexed citations

15.

Lillis, Joseph V., Vernon Ansdell, Eric L. Simpson, et al.. (2009). Sequelae of World War II: An Outbreak of Chronic Cutaneous Nontuberculous Mycobacterial Infection among Satowanese Islanders. Clinical Infectious Diseases. 48(11). 1541–1546. 9 indexed citations

16.

Kurtz, Stephen E., et al.. (2009). Inflammatory Skin Disease in K5.hTGF-β1 Transgenic Mice Is Not Dependent on the IL-23/Th17 Inflammatory Pathway. Journal of Investigative Dermatology. 129(10). 2443–2450. 26 indexed citations

17.

McCoy, Sharon L., Stephen E. Kurtz, Carol J. MacArthur, Dennis R. Trune, & Steven H. Hefeneider. (2005). Identification of a Peptide Derived from Vaccinia Virus A52R Protein That Inhibits Cytokine Secretion in Response to TLR-Dependent Signaling and Reduces In Vivo Bacterial-Induced Inflammation. The Journal of Immunology. 174(5). 3006–3014. 48 indexed citations

18.

Kawamura, Tatsuyoshi, Stephen E. Kurtz, Andrew Blauvelt, & Shinji Shimada. (2005). The role of Langerhans cells in the sexual transmission of HIV. Journal of Dermatological Science. 40(3). 147–155. 51 indexed citations

19.

Kurtz, Stephen E., et al.. (2003). Inhibition of an activated Ras protein with genetically selected peptide aptamers. Biotechnology and Bioengineering. 82(1). 38–46. 8 indexed citations

20.

Tang, Weimin, et al.. (1995). Functional expression of a vertebrate inwardly rectifying K+ channel in yeast.. Molecular Biology of the Cell. 6(9). 1231–1240. 58 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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