Silvia Lommel

1.1k total citations
11 papers, 915 citations indexed

About

Silvia Lommel is a scholar working on Molecular Biology, Cell Biology and Genetics. According to data from OpenAlex, Silvia Lommel has authored 11 papers receiving a total of 915 indexed citations (citations by other indexed papers that have themselves been cited), including 5 papers in Molecular Biology, 5 papers in Cell Biology and 4 papers in Genetics. Recurrent topics in Silvia Lommel's work include Bacterial Genetics and Biotechnology (3 papers), Pancreatic function and diabetes (3 papers) and Pluripotent Stem Cells Research (2 papers). Silvia Lommel is often cited by papers focused on Bacterial Genetics and Biotechnology (3 papers), Pancreatic function and diabetes (3 papers) and Pluripotent Stem Cells Research (2 papers). Silvia Lommel collaborates with scholars based in Germany, Denmark and United Kingdom. Silvia Lommel's co-authors include Klemens Rottner, Stefanie Benesch, Jürgen Wehland, Ralf Kühn, Thomas Franz, Theresia E. B. Stradal, Marianne Wiechers, Marion Jung, Claudia A. O. Stuermer and Ute Laessing and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Biological Chemistry and Development.

In The Last Decade

Silvia Lommel

11 papers receiving 905 citations

Peers

Silvia Lommel
Stanislav Samarin United States
Miriam V. Bujny United Kingdom
Marije Marsman Netherlands
Guissou A. Dabiri United States
Vincent Idone United States
Andreas Gerondopoulos United Kingdom
Fang Liao United States
Silvia Lommel
Citations per year, relative to Silvia Lommel Silvia Lommel (= 1×) peers Stefanie Benesch

Countries citing papers authored by Silvia Lommel

Since Specialization
Citations

This map shows the geographic impact of Silvia Lommel's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Silvia Lommel with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Silvia Lommel more than expected).

Fields of papers citing papers by Silvia Lommel

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Silvia Lommel. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Silvia Lommel. The network helps show where Silvia Lommel may publish in the future.

Co-authorship network of co-authors of Silvia Lommel

This figure shows the co-authorship network connecting the top 25 collaborators of Silvia Lommel. A scholar is included among the top collaborators of Silvia Lommel based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Silvia Lommel. Silvia Lommel is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

11 of 11 papers shown
1.
Kesavan, Gokul, Anant Mamidi, Jenny K. Johansson, et al.. (2014). Cdc42/N-WASP signaling links actin dynamics to pancreatic β cell delamination and differentiation. Journal of Cell Science. 127(3). e1–e1. 1 indexed citations
2.
Kesavan, Gokul, Anant Mamidi, Jenny K. Johansson, et al.. (2014). Cdc42/N-WASP signaling links actin dynamics to pancreatic β cell delamination and differentiation. Development. 141(3). 685–696. 42 indexed citations
3.
Jin, Fuzi, Baoxia Dong, John Georgiou, et al.. (2011). N-WASp is required for Schwann cell cytoskeletal dynamics, normal myelin gene expression and peripheral nerve myelination. Development. 138(7). 1329–1337. 51 indexed citations
4.
Ladwein, Markus, Silvia Lommel, Ulrike Beutling, et al.. (2009). IRSp53 Links the Enterohemorrhagic E. coli Effectors Tir and EspFU for Actin Pedestal Formation. Cell Host & Microbe. 5(3). 244–258. 83 indexed citations
5.
Pedersen, Esben, A. Basse, Ralf Paus, et al.. (2009). N-WASP is a novel regulator of hair-follicle cycling that controls antiproliferative TGFβ pathways. Journal of Cell Science. 123(1). 128–140. 30 indexed citations
6.
Stamm, Luisa M., J. Hiroshi Morisaki, Scott B. Snapper, et al.. (2005). Role of the WASP family proteins for Mycobacterium marinum actin tail formation. Proceedings of the National Academy of Sciences. 102(41). 14837–14842. 50 indexed citations
7.
Rottner, Klemens, Silvia Lommel, Jürgen Wehland, & Theresia E. B. Stradal. (2004). Pathogen‐induced actin filament rearrangement in infectious diseases. The Journal of Pathology. 204(4). 396–406. 39 indexed citations
8.
Lommel, Silvia, Stefanie Benesch, Manfred Rohde, Jürgen Wehland, & Klemens Rottner. (2004). Enterohaemorrhagic and enteropathogenic Escherichia coli use different mechanisms for actin pedestal formation that converge on N-WASP. Cellular Microbiology. 6(3). 243–254. 61 indexed citations
9.
Benesch, Stefanie, Silvia Lommel, Anika Steffen, et al.. (2002). Phosphatidylinositol 4,5-Biphosphate (PIP2)-induced Vesicle Movement Depends on N-WASP and Involves Nck, WIP, and Grb2. Journal of Biological Chemistry. 277(40). 37771–37776. 128 indexed citations
10.
Lommel, Silvia, Stefanie Benesch, Klemens Rottner, et al.. (2001). Actin pedestal formation by enteropathogenic Escherichia coli and intracellular motility of Shigella flexneri are abolished in N‐WASP‐defective cells. EMBO Reports. 2(9). 850–857. 216 indexed citations
11.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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