Signe Schultz Pedersen

401 total citations
9 papers, 285 citations indexed

About

Signe Schultz Pedersen is a scholar working on Surgery, Molecular Biology and Genetics. According to data from OpenAlex, Signe Schultz Pedersen has authored 9 papers receiving a total of 285 indexed citations (citations by other indexed papers that have themselves been cited), including 5 papers in Surgery, 3 papers in Molecular Biology and 3 papers in Genetics. Recurrent topics in Signe Schultz Pedersen's work include Pancreatic function and diabetes (5 papers), Diabetes and associated disorders (3 papers) and Diet, Metabolism, and Disease (2 papers). Signe Schultz Pedersen is often cited by papers focused on Pancreatic function and diabetes (5 papers), Diabetes and associated disorders (3 papers) and Diet, Metabolism, and Disease (2 papers). Signe Schultz Pedersen collaborates with scholars based in Denmark, Australia and Canada. Signe Schultz Pedersen's co-authors include Nils Billestrup, Michala Prause, Martin Iain Bahl, Tine Rask Licht, Maria Louise Leth, Christopher T. Workman, Maher Abou Hachem, Bjørge Westereng, Claus Sternberg and Morten Ejby and has published in prestigious journals such as Journal of Biological Chemistry, The FASEB Journal and Biochemical and Biophysical Research Communications.

In The Last Decade

Signe Schultz Pedersen

8 papers receiving 281 citations

Peers

Signe Schultz Pedersen
Amanda Photenhauer United States
Negar Foolad United States
Xue Huang China
Sarah L. Eady New Zealand
Signe Schultz Pedersen
Citations per year, relative to Signe Schultz Pedersen Signe Schultz Pedersen (= 1×) peers Lanmei Yin

Countries citing papers authored by Signe Schultz Pedersen

Since Specialization
Citations

This map shows the geographic impact of Signe Schultz Pedersen's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Signe Schultz Pedersen with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Signe Schultz Pedersen more than expected).

Fields of papers citing papers by Signe Schultz Pedersen

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Signe Schultz Pedersen. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Signe Schultz Pedersen. The network helps show where Signe Schultz Pedersen may publish in the future.

Co-authorship network of co-authors of Signe Schultz Pedersen

This figure shows the co-authorship network connecting the top 25 collaborators of Signe Schultz Pedersen. A scholar is included among the top collaborators of Signe Schultz Pedersen based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Signe Schultz Pedersen. Signe Schultz Pedersen is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

9 of 9 papers shown
1.
Pedersen, Signe Schultz, et al.. (2025). The Gut Microbial Metabolite Butyrate Alleviates IL‐1β‐Induced Mitochondrial Dysfunction and Oxidative Stress in Pancreatic Islets. The FASEB Journal. 39(11). e70623–e70623. 1 indexed citations
2.
Jørgensen, K., Signe Schultz Pedersen, Siv A. Hjorth, Nils Billestrup, & Michala Prause. (2024). Protection of beta cells against cytokine‐induced apoptosis by the gut microbial metabolite butyrate. FEBS Journal. 292(1). 226–240.
3.
Pedersen, Signe Schultz, Michala Prause, Christina Sørensen, et al.. (2023). Targeted Delivery of Butyrate Improves Glucose Homeostasis, Reduces Hepatic Lipid Accumulation and Inflammation in db/db Mice. International Journal of Molecular Sciences. 24(5). 4533–4533. 13 indexed citations
4.
Pedersen, Signe Schultz, et al.. (2023). Butyrate functions as a histone deacetylase inhibitor to protect pancreatic beta cells from IL ‐1β‐induced dysfunction. FEBS Journal. 291(3). 566–583. 19 indexed citations
5.
Pedersen, Signe Schultz, Michala Prause, Kristine Williams, Romain Barrès, & Nils Billestrup. (2022). Butyrate inhibits IL-1β-induced inflammatory gene expression by suppression of NF-κB activity in pancreatic beta cells. Journal of Biological Chemistry. 298(9). 102312–102312. 47 indexed citations
6.
Graversen, Katrine, Jeppe Madura Larsen, Signe Schultz Pedersen, et al.. (2021). Partially Hydrolysed Whey Has Superior Allergy Preventive Capacity Compared to Intact Whey Regardless of Amoxicillin Administration in Brown Norway Rats. Frontiers in Immunology. 12. 12 indexed citations
7.
Prause, Michala, et al.. (2021). Butyrate Protects Pancreatic Beta Cells from Cytokine-Induced Dysfunction. International Journal of Molecular Sciences. 22(19). 10427–10427. 24 indexed citations
8.
Leth, Maria Louise, Morten Ejby, Christopher T. Workman, et al.. (2018). Differential bacterial capture and transport preferences facilitate co-growth on dietary xylan in the human gut. Nature Microbiology. 3(5). 570–580. 136 indexed citations
9.
Gesser, Borbala, Bent Deleuran, Marianne Lund, et al.. (1995). Interleukin-8 Induces Its Own Production in CD4+ T Lymphocytes: A Process Regulated by Interleukin 10. Biochemical and Biophysical Research Communications. 210(3). 660–669. 33 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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