Shujun Ge

1.1k total citations
18 papers, 895 citations indexed

About

Shujun Ge is a scholar working on Neurology, Molecular Biology and Cell Biology. According to data from OpenAlex, Shujun Ge has authored 18 papers receiving a total of 895 indexed citations (citations by other indexed papers that have themselves been cited), including 13 papers in Neurology, 7 papers in Molecular Biology and 4 papers in Cell Biology. Recurrent topics in Shujun Ge's work include Barrier Structure and Function Studies (10 papers), Neuroinflammation and Neurodegeneration Mechanisms (8 papers) and Neurological Disease Mechanisms and Treatments (4 papers). Shujun Ge is often cited by papers focused on Barrier Structure and Function Studies (10 papers), Neuroinflammation and Neurodegeneration Mechanisms (8 papers) and Neurological Disease Mechanisms and Treatments (4 papers). Shujun Ge collaborates with scholars based in United States, United Kingdom and Italy. Shujun Ge's co-authors include Joel S. Pachter, Song Li, Debayon Paul, Bandana Shrestha, David R. Serwanski, Evan R. Jellison, Nivetha Murugesan, Ann E. Cowan, William A. Kuziel and Xi Jiang and has published in prestigious journals such as Journal of Biological Chemistry, Blood and Biochemical Journal.

In The Last Decade

Shujun Ge

18 papers receiving 891 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Shujun Ge United States 14 411 329 147 146 144 18 895
Patrick Ventura United States 11 323 0.8× 494 1.5× 97 0.7× 101 0.7× 233 1.6× 11 1.1k
Belinda Cairns United States 12 259 0.6× 494 1.5× 93 0.6× 75 0.5× 189 1.3× 15 1.1k
Alexander S. Easton Canada 18 268 0.7× 326 1.0× 118 0.8× 64 0.4× 141 1.0× 31 750
Heinrich F. Bürgers Germany 12 201 0.5× 425 1.3× 96 0.7× 106 0.7× 78 0.5× 14 1.0k
Thorsten Bangsow Germany 7 371 0.9× 680 2.1× 146 1.0× 141 1.0× 78 0.5× 13 1.1k
Simone Terouz Canada 6 571 1.4× 473 1.4× 156 1.1× 77 0.5× 234 1.6× 11 1.2k
Marta Segarra Germany 19 178 0.4× 495 1.5× 134 0.9× 108 0.7× 165 1.1× 24 1.1k
Kim Lakeman Netherlands 9 233 0.6× 431 1.3× 110 0.7× 78 0.5× 102 0.7× 9 747
Marco Reis Germany 7 413 1.0× 611 1.9× 149 1.0× 136 0.9× 52 0.4× 7 1.0k
Eric Koncina Luxembourg 12 183 0.4× 608 1.8× 161 1.1× 87 0.6× 130 0.9× 17 1.1k

Countries citing papers authored by Shujun Ge

Since Specialization
Citations

This map shows the geographic impact of Shujun Ge's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Shujun Ge with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Shujun Ge more than expected).

Fields of papers citing papers by Shujun Ge

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Shujun Ge. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Shujun Ge. The network helps show where Shujun Ge may publish in the future.

Co-authorship network of co-authors of Shujun Ge

This figure shows the co-authorship network connecting the top 25 collaborators of Shujun Ge. A scholar is included among the top collaborators of Shujun Ge based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Shujun Ge. Shujun Ge is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

18 of 18 papers shown
1.
Balsbaugh, Jeremy L., et al.. (2023). Proteomic interrogation of the meninges reveals the molecular identities of structural components and regional distinctions along the CNS axis. Fluids and Barriers of the CNS. 20(1). 74–74. 3 indexed citations
2.
Paul, Debayon, et al.. (2021). Appearance of claudin-5+ leukocyte subtypes in the blood and CNS during progression of EAE. Journal of Neuroinflammation. 18(1). 296–296. 6 indexed citations
3.
Ge, Shujun, Xi Jiang, Debayon Paul, et al.. (2019). Human ES-derived MSCs correct TNF-α-mediated alterations in a blood–brain barrier model. Fluids and Barriers of the CNS. 16(1). 18–18. 13 indexed citations
4.
Shrestha, Bandana, Xi Jiang, Shujun Ge, et al.. (2017). Spatiotemporal resolution of spinal meningeal and parenchymal inflammation during experimental autoimmune encephalomyelitis. Neurobiology of Disease. 108. 159–172. 15 indexed citations
5.
Paul, Debayon, Valentina Baena, Shujun Ge, et al.. (2016). Appearance of claudin-5+ leukocytes in the central nervous system during neuroinflammation: a novel role for endothelial-derived extracellular vesicles. Journal of Neuroinflammation. 13(1). 292–292. 64 indexed citations
6.
Shrestha, Bandana, Shujun Ge, & Joel S. Pachter. (2014). Resolution of central nervous system astrocytic and endothelial sources of CCL2 gene expression during evolving neuroinflammation. Fluids and Barriers of the CNS. 11(1). 6–6. 16 indexed citations
7.
Paul, Debayon, Shujun Ge, Evan R. Jellison, et al.. (2014). Cell-selective knockout and 3D confocal image analysis reveals separate roles for astrocyte-and endothelial-derived CCL2 in neuroinflammation. Journal of Neuroinflammation. 11(1). 10–10. 59 indexed citations
8.
Ge, Shujun, Bandana Shrestha, Debayon Paul, et al.. (2012). The CCL2 synthesis inhibitor bindarit targets cells of the neurovascular unit, and suppresses experimental autoimmune encephalomyelitis. Journal of Neuroinflammation. 9(1). 171–171. 56 indexed citations
9.
Paul, Debayon, Ann E. Cowan, Shujun Ge, & Joel S. Pachter. (2012). Novel 3D analysis of Claudin-5 reveals significant endothelial heterogeneity among CNS microvessels. Microvascular Research. 86. 1–10. 55 indexed citations
10.
11.
Murugesan, Nivetha, Jennifer A. Macdonald, Shujun Ge, & Joel S. Pachter. (2011). Probing the CNS Microvascular Endothelium by Immune-Guided Laser-Capture Microdissection Coupled to Quantitative RT-PCR. Methods in molecular biology. 755. 385–394. 4 indexed citations
12.
Ge, Shujun, Nivetha Murugesan, & Joel S. Pachter. (2009). Astrocyte- and Endothelial-Targeted CCL2 Conditional Knockout Mice: Critical Tools for Studying the Pathogenesis of Neuroinflammation. Journal of Molecular Neuroscience. 39(1-2). 269–283. 19 indexed citations
13.
Ge, Shujun, Song Li, David R. Serwanski, William A. Kuziel, & Joel S. Pachter. (2007). Transcellular transport of CCL2 across brain microvascular endothelial cells. Journal of Neurochemistry. 104(5). 1219–1232. 68 indexed citations
14.
Ge, Shujun & Joel S. Pachter. (2006). Isolation and culture of microvascular endothelial cells from murine spinal cord. Journal of Neuroimmunology. 177(1-2). 209–214. 80 indexed citations
15.
Li, Song, Shujun Ge, & Joel S. Pachter. (2006). Caveolin-1 regulates expression of junction-associated proteins in brain microvascular endothelial cells. Blood. 109(4). 1515–1523. 143 indexed citations
16.
Ge, Shujun, Song Li, & Joel S. Pachter. (2005). Where is the blood–brain barrier … really?. Journal of Neuroscience Research. 79(4). 421–427. 101 indexed citations
17.
Ge, Shujun & Joel S. Pachter. (2004). Caveolin-1 Knockdown by Small Interfering RNA Suppresses Responses to the Chemokine Monocyte Chemoattractant Protein-1 by Human Astrocytes. Journal of Biological Chemistry. 279(8). 6688–6695. 55 indexed citations
18.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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