Shufang Cai

725 total citations
30 papers, 509 citations indexed

About

Shufang Cai is a scholar working on Molecular Biology, Surgery and Genetics. According to data from OpenAlex, Shufang Cai has authored 30 papers receiving a total of 509 indexed citations (citations by other indexed papers that have themselves been cited), including 21 papers in Molecular Biology, 5 papers in Surgery and 4 papers in Genetics. Recurrent topics in Shufang Cai's work include Muscle Physiology and Disorders (8 papers), Ubiquitin and proteasome pathways (4 papers) and RNA Research and Splicing (4 papers). Shufang Cai is often cited by papers focused on Muscle Physiology and Disorders (8 papers), Ubiquitin and proteasome pathways (4 papers) and RNA Research and Splicing (4 papers). Shufang Cai collaborates with scholars based in China, Canada and United Kingdom. Shufang Cai's co-authors include Delin Mo, Yaosheng Chen, Qi Zhu, Yanqing Wu, Renqiang Yuan, Jian Xiao, Jingyu Xu, Xumeng Zhang, Jiajia Ye and Yuan Yuan and has published in prestigious journals such as Journal of Agricultural and Food Chemistry, The FASEB Journal and Biochemical and Biophysical Research Communications.

In The Last Decade

Shufang Cai

28 papers receiving 503 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Shufang Cai China 14 268 58 53 52 52 30 509
Hak Hyun Jung South Korea 14 251 0.9× 52 0.9× 53 1.0× 56 1.1× 36 0.7× 26 549
Yuanyuan Gong China 14 297 1.1× 46 0.8× 56 1.1× 37 0.7× 23 0.4× 37 820
Mohammad Ghasem Golmohammadi Iran 14 170 0.6× 30 0.5× 71 1.3× 34 0.7× 38 0.7× 35 535
Nataliya Chorna Puerto Rico 16 261 1.0× 40 0.7× 76 1.4× 34 0.7× 76 1.5× 32 644
Chris Young United Kingdom 15 353 1.3× 63 1.1× 39 0.7× 45 0.9× 78 1.5× 24 727
Tzung‐Chieh Tsai Taiwan 13 187 0.7× 45 0.8× 51 1.0× 34 0.7× 26 0.5× 19 449
Chuan Cai China 16 189 0.7× 71 1.2× 22 0.4× 42 0.8× 67 1.3× 33 539
Jingxian Xu China 11 262 1.0× 98 1.7× 81 1.5× 49 0.9× 92 1.8× 24 532

Countries citing papers authored by Shufang Cai

Since Specialization
Citations

This map shows the geographic impact of Shufang Cai's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Shufang Cai with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Shufang Cai more than expected).

Fields of papers citing papers by Shufang Cai

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Shufang Cai. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Shufang Cai. The network helps show where Shufang Cai may publish in the future.

Co-authorship network of co-authors of Shufang Cai

This figure shows the co-authorship network connecting the top 25 collaborators of Shufang Cai. A scholar is included among the top collaborators of Shufang Cai based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Shufang Cai. Shufang Cai is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Zhou, Mei, Shufang Cai, Tao Wei, et al.. (2025). Chitosan oligosaccharides ameliorates maternal diabetes-induced embryonic neural tube defects via inhibitting excessive pyroptosis of neuroepithelial cells. International Immunopharmacology. 148. 114074–114074.
3.
Yuan, Renqiang, Shufang Cai, Yunxiang Zhao, et al.. (2024). UBE2C promotes myoblast differentiation and skeletal muscle regeneration through the Akt signaling pathway. Acta Biochimica et Biophysica Sinica. 56(7). 1065–1071. 3 indexed citations
4.
Cai, Shufang, Xiaoyu Wang, Rong Xu, et al.. (2023). Integrative single-cell RNA-seq and ATAC-seq analysis of myogenic differentiation in pig. BMC Biology. 21(1). 19–19. 28 indexed citations
5.
Zhang, Susu, Jiaxin Zhao, Man Wu, et al.. (2023). Over-activation of TRPM2 ion channel accelerates blood-spinal cord barrier destruction in diabetes combined with spinal cord injury rat. International Journal of Biological Sciences. 19(8). 2475–2494. 7 indexed citations
6.
Cai, Shufang, Xiaoyu Wang, Rong Xu, et al.. (2023). KLF4 regulates skeletal muscle development and regeneration by directly targeting P57 and Myomixer. Cell Death and Disease. 14(9). 612–612. 12 indexed citations
7.
Cai, Shufang, Qi Zhu, Xiaoyu Wang, et al.. (2022). Notch signaling leads to a slower progression of embryonic myogenic differentiation in Landrace than in Langtang pigs. Acta Biochimica et Biophysica Sinica. 54(8). 1122–1132. 2 indexed citations
8.
Cai, Shufang, et al.. (2022). A Comparative Analysis of Metabolic Profiles of Embryonic Skeletal Muscle from Lantang and Landrace Pigs. Animals. 12(4). 420–420. 3 indexed citations
10.
Wu, Yanqing, Jun Xiong, Zili He, et al.. (2021). Metformin promotes microglial cells to facilitate myelin debris clearance and accelerate nerve repairment after spinal cord injury. Acta Pharmacologica Sinica. 43(6). 1360–1371. 77 indexed citations
11.
Xu, Jingyu, Shufang Cai, Jiaxin Zhao, et al.. (2021). Advances in the Relationship Between Pyroptosis and Diabetic Neuropathy. Frontiers in Cell and Developmental Biology. 9. 753660–753660. 20 indexed citations
12.
Zhu, Qi, Liang Feng, Shufang Cai, et al.. (2021). KDM4A regulates myogenesis by demethylating H3K9me3 of myogenic regulatory factors. Cell Death and Disease. 12(6). 514–514. 27 indexed citations
13.
Xu, Jingyu, Zonghao Tang, Shufang Cai, et al.. (2021). Dl-3-n-Butylphthalide Ameliorates Diabetic Nephropathy by Ameliorating Excessive Fibrosis and Podocyte Apoptosis. Frontiers in Pharmacology. 12. 628950–628950. 8 indexed citations
14.
Zheng, Peipei, Zonghao Tang, Jun Xiong, et al.. (2021). RAGE: A potential therapeutic target during FGF1 treatment of diabetes‐mediated liver injury. Journal of Cellular and Molecular Medicine. 25(10). 4776–4785. 14 indexed citations
15.
Chen, Keren, Liang Feng, Qi Zhu, et al.. (2021). HMGB2 orchestrates mitotic clonal expansion by binding to the promoter of C/EBPβ to facilitate adipogenesis. Cell Death and Disease. 12(7). 666–666. 24 indexed citations
16.
Lu, Qi, Jun Xiong, Yuan Yuan, et al.. (2021). Minocycline improves the functional recovery after traumatic brain injury via inhibition of aquaporin-4. International Journal of Biological Sciences. 18(1). 441–458. 34 indexed citations
17.
Fang, Ying, Liang Feng, Renqiang Yuan, et al.. (2020). High mobility group box 2 regulates skeletal muscle development through ribosomal protein S6 kinase 1. The FASEB Journal. 34(9). 12367–12378. 7 indexed citations
18.
Zhang, Xumeng, Xiaohong Liu, Chen Wang, et al.. (2019). The SNPs in myoD gene from normal muscle developing individuals have no effect on muscle mass. BMC Genetics. 20(1). 72–72. 5 indexed citations
19.
Cai, Shufang, Renqiang Yuan, Xumeng Zhang, et al.. (2019). Zfp422 promotes skeletal muscle differentiation by regulating EphA7 to induce appropriate myoblast apoptosis. Cell Death and Differentiation. 27(5). 1644–1659. 19 indexed citations
20.
Yuan, Renqiang, Xumeng Zhang, Ying Fang, et al.. (2018). mir-127-3p inhibits the proliferation of myocytes by targeting KMT5a. Biochemical and Biophysical Research Communications. 503(2). 970–976. 18 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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