Shubiao Ye

901 total citations
26 papers, 402 citations indexed

About

Shubiao Ye is a scholar working on Oncology, Molecular Biology and Immunology. According to data from OpenAlex, Shubiao Ye has authored 26 papers receiving a total of 402 indexed citations (citations by other indexed papers that have themselves been cited), including 14 papers in Oncology, 9 papers in Molecular Biology and 7 papers in Immunology. Recurrent topics in Shubiao Ye's work include Genetic factors in colorectal cancer (6 papers), Cancer Immunotherapy and Biomarkers (6 papers) and Colorectal Cancer Treatments and Studies (4 papers). Shubiao Ye is often cited by papers focused on Genetic factors in colorectal cancer (6 papers), Cancer Immunotherapy and Biomarkers (6 papers) and Colorectal Cancer Treatments and Studies (4 papers). Shubiao Ye collaborates with scholars based in China, United States and Germany. Shubiao Ye's co-authors include Yi‐Kan Cheng, Ping Lan, Peisi Li, Yanhong Deng, Xianrui Wu, Huashan Liu, Ru Deng, Runqing Huang, Jianwei Zhang and Ping Lan and has published in prestigious journals such as SHILAP Revista de lepidopterología, ACS Nano and Oncogene.

In The Last Decade

Shubiao Ye

24 papers receiving 399 citations

Peers

Shubiao Ye
Yuanfei Shi China
Dejun Wu China
Tianliang Xia China
Bowen Yuan China
Biao Deng China
Huafu Li China
Shuier Zheng China
Qingnuo Zeng China
Ririno Honma Japan
Yuanfei Shi China
Shubiao Ye
Citations per year, relative to Shubiao Ye Shubiao Ye (= 1×) peers Yuanfei Shi

Countries citing papers authored by Shubiao Ye

Since Specialization
Citations

This map shows the geographic impact of Shubiao Ye's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Shubiao Ye with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Shubiao Ye more than expected).

Fields of papers citing papers by Shubiao Ye

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Shubiao Ye. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Shubiao Ye. The network helps show where Shubiao Ye may publish in the future.

Co-authorship network of co-authors of Shubiao Ye

This figure shows the co-authorship network connecting the top 25 collaborators of Shubiao Ye. A scholar is included among the top collaborators of Shubiao Ye based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Shubiao Ye. Shubiao Ye is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Cheng, Yi‐Kan, et al.. (2025). Tumor-intrinsic TTLL12 drives resistance to cancer immunotherapy via modulating myeloid-derived suppressor cells. Journal for ImmunoTherapy of Cancer. 13(6). e010873–e010873. 1 indexed citations
2.
Ye, Shubiao, et al.. (2025). Targeting SIRT2 induces MLH1 deficiency and boosts antitumor immunity in preclinical colorectal cancer models. Science Translational Medicine. 17(807). eadv0766–eadv0766.
3.
Yuan, Ming, Shaopeng Chen, Kang Chao, et al.. (2025). ACSS2‐Mediated Histone H4 Lysine 12 Crotonylation (H4K12cr) Alleviates Colitis via Enhancing Transcription of CLDN7. Advanced Science. 12(30). e00461–e00461.
4.
Zhang, Chi, Ming Yuan, Shubiao Ye, et al.. (2025). Exercise-induced Metabolite N-lactoyl-phenylalanine Ameliorates Colitis by Inhibiting M1 Macrophage Polarization via the Suppression of the NF-κB Signaling Pathway. Cellular and Molecular Gastroenterology and Hepatology. 19(10). 101558–101558. 4 indexed citations
5.
Zhang, Chi, Ming Yuan, Tuo Hu, et al.. (2025). KRAS mutation increases histone H3 lysine 9 lactylation (H3K9la) to promote colorectal cancer progression by facilitating cholesterol transporter GRAMD1A expression. Cell Death and Differentiation. 32(12). 2225–2238. 1 indexed citations
6.
Yuan, Ming, Chi Zhang, Shaopeng Chen, et al.. (2024). PDP1 promotes KRAS mutant colorectal cancer progression by serving as a scaffold for BRAF and MEK1. Cancer Letters. 597. 217007–217007. 5 indexed citations
7.
Cai, Jiawei, Shubiao Ye, Junfeng Huang, et al.. (2024). MTHFD1 Regulates Autophagy to Promote Growth and Metastasis in Colorectal Cancer via the PI3K‐AKT–mTOR Signaling Pathway. Cancer Medicine. 13(22). e70267–e70267. 2 indexed citations
8.
Zhang, Qiang, Shubiao Ye, Junli Gong, et al.. (2023). Reprogramming of palmitic acid induced by dephosphorylation of ACOX1 promotes β-catenin palmitoylation to drive colorectal cancer progression. Cell Discovery. 9(1). 26–26. 47 indexed citations
9.
Li, Peisi, Yi‐Kan Cheng, Yuan Chen, et al.. (2023). Tumor-secreted IFI35 promotes proliferation and cytotoxic activity of CD8+ T cells through PI3K/AKT/mTOR signaling pathway in colorectal cancer. Journal of Biomedical Science. 30(1). 47–47. 25 indexed citations
10.
Chen, Guanxing, et al.. (2023). Discovery of Kinetin in inhibiting colorectal cancer progression via enhancing PSMB1-mediated RAB34 degradation. Cancer Letters. 584. 216600–216600. 4 indexed citations
11.
Xing, Zhe, et al.. (2022). OLFM4 deficiency delays the progression of colitis to colorectal cancer by abrogating PMN-MDSCs recruitment. Oncogene. 41(22). 3131–3150. 17 indexed citations
12.
Ye, Shubiao, et al.. (2022). The Role of RAB GTPases and Its Potential in Predicting Immunotherapy Response and Prognosis in Colorectal Cancer. Frontiers in Genetics. 13. 828373–828373. 17 indexed citations
13.
Liu, Binbin, Tengfei Liu, Yiting Liu, et al.. (2022). TSG-6 promotes Cancer Cell aggressiveness in a CD44-Dependent Manner and Reprograms Normal Fibroblasts to create a Pro-metastatic Microenvironment in Colorectal Cancer. International Journal of Biological Sciences. 18(4). 1677–1694. 22 indexed citations
14.
Cheng, Yi‐Kan, Ping Chen, Xiaosheng He, et al.. (2022). Association of Peripheral Blood Biomarkers With Response to Anti-PD-1 Immunotherapy for Patients With Deficient Mismatch Repair Metastatic Colorectal Cancer: A Multicenter Cohort Study. Frontiers in Immunology. 13. 809971–809971. 15 indexed citations
15.
Li, Zhihao, Peisi Li, Shubiao Ye, et al.. (2022). Dynamic heterogeneity of colorectal cancer during progression revealed clinical risk-associated cell types and regulations in single-cell resolution and spatial context. Gastroenterology report. 11. goad034–goad034. 4 indexed citations
16.
Ye, Shubiao, Yi‐Kan Cheng, Jiancong Hu, Feng Gao, & Ping Lan. (2020). Development and validation of an individualized gene expression-based signature to predict overall survival in metastatic colorectal cancer. Annals of Translational Medicine. 8(4). 96–96. 6 indexed citations
17.
18.
Ye, Shubiao, Yi‐Kan Cheng, Lin Zhang, et al.. (2019). Prognostic value of estrogen receptor-α and progesterone receptor in curatively resected colorectal cancer: a retrospective analysis with independent validations. BMC Cancer. 19(1). 933–933. 19 indexed citations
19.
Du, Minghui, Shubiao Ye, Shichao Lv, et al.. (2018). Scalable In-Fiber Manufacture of Functional Composite Particles. ACS Nano. 12(11). 11130–11138. 14 indexed citations
20.
Deng, Weiye, Yanfeng Chen, Zhuming Guo, et al.. (2015). Expression of Notch-1 and recurrence-related factors in papillary thyroid carcinoma. 27(1). 11–14. 1 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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