Shirley Rogers
- Clinical Biochemistry top 2%
- Molecular Biology
- Biochemistry top 5%
- Pediatrics, Perinatology and Child Health top 10%
- Physiology
- Co-authors
- Stanton SegalPhilip G. HoltzappleJames J. AndersonRandall A. HeidenreichJohn MalleeGerard T. BerryWilliam J. MellmanMichael Palmieri
- Topics
- Metabolism and Genetic Disorders (16 papers)Aldose Reductase and Taurine (12 papers)Amino Acid Enzymes and Metabolism (9 papers)
- Journals
- Journal of Clinical InvestigationAnalytical BiochemistryBiochemical and Biophysical Research Communications
- Partner nations
- United States
In The Last Decade
Shirley Rogers
25 papers receiving 328 citations
Peers
Comparison fields: 5 of 61
- Clinical Biochemistry 211
- Molecular Biology 122
- Biochemistry 97
- Pediatrics, Perinatology and Child Health 95
- Physiology 75
Countries citing papers authored by Shirley Rogers
This map shows the geographic impact of Shirley Rogers's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Shirley Rogers with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Shirley Rogers more than expected).
Fields of papers citing papers by Shirley Rogers
This network shows the impact of papers produced by Shirley Rogers. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Shirley Rogers. The network helps show where Shirley Rogers may publish in the future.
Co-authorship network of co-authors of Shirley Rogers
This figure shows the co-authorship network connecting the top 25 collaborators of Shirley Rogers. A scholar is included among the top collaborators of Shirley Rogers based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Shirley Rogers. Shirley Rogers is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 13 | |
| 2 | 28 | |
| 3 | 8 | |
| 4 | 5 | |
| 5 | 36 | |
| 6 | 10 | |
| 7 | 5 | |
| 8 | 16 | |
| 9 | 18 | |
| 10 | 5 | |
| 11 | 5 | |
| 12 | 9 | |
| 13 | 3 | |
| 14 | 27 | |
| 15 | 8 | |
| 16 | 11 | |
| 17 | 26 | |
| 18 | 7 | |
| 19 | 14 | |
| 20 | 1 |
About Shirley Rogers
Shirley Rogers is a scholar working on Clinical Biochemistry, Biochemistry and Cell Biology, having authored 25 papers that have together received 345 indexed citations. Recurring topics across this work include Metabolism and Genetic Disorders (16 papers), Aldose Reductase and Taurine (12 papers) and Amino Acid Enzymes and Metabolism (9 papers). The work is most often cited by research in Clinical Biochemistry (211 citations), Biochemistry (97 citations) and Pediatrics, Perinatology and Child Health (95 citations). Shirley Rogers has collaborated with scholars based in United States. Frequent co-authors include Stanton Segal, Philip G. Holtzapple, James J. Anderson, Randall A. Heidenreich, John Mallee, Stanton Segal, Gerard T. Berry, William J. Mellman, Michael Palmieri and Robert Reynolds. Their work appears in journals such as Journal of Clinical Investigation, Analytical Biochemistry and Biochemical and Biophysical Research Communications.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.