Shinnosuke Morikawa

820 total citations · 1 hit paper
10 papers, 639 citations indexed

About

Shinnosuke Morikawa is a scholar working on Molecular Biology, Immunology and Nephrology. According to data from OpenAlex, Shinnosuke Morikawa has authored 10 papers receiving a total of 639 indexed citations (citations by other indexed papers that have themselves been cited), including 9 papers in Molecular Biology, 5 papers in Immunology and 3 papers in Nephrology. Recurrent topics in Shinnosuke Morikawa's work include Inflammasome and immune disorders (9 papers), Gout, Hyperuricemia, Uric Acid (3 papers) and interferon and immune responses (2 papers). Shinnosuke Morikawa is often cited by papers focused on Inflammasome and immune disorders (9 papers), Gout, Hyperuricemia, Uric Acid (3 papers) and interferon and immune responses (2 papers). Shinnosuke Morikawa collaborates with scholars based in Japan. Shinnosuke Morikawa's co-authors include Junya Masumoto, Naoe Kaneko, Mie Kurata, Toshihiro Yamamoto, Tamotsu Zako, Tatsuya Sawasaki, Hiroyuki Takeda, Wei Zhou, Sheikh Mohammad Fazle Akbar and Yuki Ito and has published in prestigious journals such as Journal of Immunological Methods, The Scientific World JOURNAL and Molecular Biology Reports.

In The Last Decade

Shinnosuke Morikawa

10 papers receiving 634 citations

Hit Papers

The role of interleukin-1 in general pathology 2019 2026 2021 2023 2019 100 200 300 400

Peers

Shinnosuke Morikawa
Shinnosuke Morikawa
Citations per year, relative to Shinnosuke Morikawa Shinnosuke Morikawa (= 1×) peers Naoe Kaneko

Countries citing papers authored by Shinnosuke Morikawa

Since Specialization
Citations

This map shows the geographic impact of Shinnosuke Morikawa's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Shinnosuke Morikawa with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Shinnosuke Morikawa more than expected).

Fields of papers citing papers by Shinnosuke Morikawa

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Shinnosuke Morikawa. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Shinnosuke Morikawa. The network helps show where Shinnosuke Morikawa may publish in the future.

Co-authorship network of co-authors of Shinnosuke Morikawa

This figure shows the co-authorship network connecting the top 25 collaborators of Shinnosuke Morikawa. A scholar is included among the top collaborators of Shinnosuke Morikawa based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Shinnosuke Morikawa. Shinnosuke Morikawa is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

10 of 10 papers shown
1.
Masumoto, Junya, Wei Zhou, Shinnosuke Morikawa, et al.. (2021). Molecular biology of autoinflammatory diseases. Inflammation and Regeneration. 41(1). 33–33. 14 indexed citations
2.
Kaneko, Naoe, Mie Kurata, Toshihiro Yamamoto, Shinnosuke Morikawa, & Junya Masumoto. (2019). The role of interleukin-1 in general pathology. Inflammation and Regeneration. 39(1). 12–12. 458 indexed citations breakdown →
3.
Kaneko, Naoe, Hiroyuki Takeda, Tatsuya Sawasaki, et al.. (2018). Amyloid β directly interacts with NLRP3 to initiate inflammasome activation: identification of an intrinsic NLRP3 ligand in a cell-free system. Inflammation and Regeneration. 38(1). 27–27. 85 indexed citations
4.
Morikawa, Shinnosuke, Naoe Kaneko, Mie Kurata, et al.. (2018). IAPP/amylin deposition, which is correlated with expressions of ASC and IL-1β in β-cells of Langerhans’ islets, directly initiates NLRP3 inflammasome activation. International Journal of Immunopathology and Pharmacology. 32. 31 indexed citations
5.
Kaneko, Naoe, Yuki Ito, Hiroyuki Takeda, et al.. (2017). Applications of reconstituted inflammasomes in a cell-free system to drug discovery and elucidation of the pathogenesis of autoinflammatory diseases. Inflammation and Regeneration. 37(1). 9–9. 6 indexed citations
6.
Kaneko, Naoe, Yuki Ito, Hiroyuki Takeda, et al.. (2017). Poly (I:C) and hyaluronic acid directly interact with NLRP3, resulting in the assembly of NLRP3 and ASC in a cell-free system. European Journal of Inflammation. 15(2). 85–97. 6 indexed citations
7.
Kaneko, Naoe, Hiroyuki Takeda, Tatsuya Sawasaki, et al.. (2016). Nod2-Nodosome in a Cell-Free System: Implications in Pathogenesis and Drug Discovery for Blau Syndrome and Early-Onset Sarcoidosis. The Scientific World JOURNAL. 2016. 1–7. 10 indexed citations
8.
Kaneko, Naoe, Yuki Ito, Hiroyuki Takeda, et al.. (2015). Reconstituted AIM2 inflammasome in cell-free system. Journal of Immunological Methods. 426. 76–81. 9 indexed citations
9.
Ito, Yuki, et al.. (2015). IL-1 as a target in inflammation. Endocrine Metabolic & Immune Disorders - Drug Targets. 15(3). 206–211. 10 indexed citations
10.
Agematsu, Kazunaga, Kiyoshi Migita, Jun Nakayama, et al.. (2013). Defect of suppression of inflammasome-independent interleukin-8 secretion from SW982 synovial sarcoma cells by familial Mediterranean fever-derived pyrin mutations. Molecular Biology Reports. 41(1). 545–553. 10 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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