Shimeng Zhou

466 total citations
22 papers, 367 citations indexed

About

Shimeng Zhou is a scholar working on Molecular Biology, Pulmonary and Respiratory Medicine and Cancer Research. According to data from OpenAlex, Shimeng Zhou has authored 22 papers receiving a total of 367 indexed citations (citations by other indexed papers that have themselves been cited), including 16 papers in Molecular Biology, 5 papers in Pulmonary and Respiratory Medicine and 5 papers in Cancer Research. Recurrent topics in Shimeng Zhou's work include RNA modifications and cancer (9 papers), Epigenetics and DNA Methylation (5 papers) and Ferroptosis and cancer prognosis (5 papers). Shimeng Zhou is often cited by papers focused on RNA modifications and cancer (9 papers), Epigenetics and DNA Methylation (5 papers) and Ferroptosis and cancer prognosis (5 papers). Shimeng Zhou collaborates with scholars based in China, Hong Kong and United States. Shimeng Zhou's co-authors include Liu-kun Yang, Qi Yang, Ming Zhao, Qixin Shi, Le Yang, Huinan Zhang, Jingru Meng, Xubo Li, Xue Ma and Di Qu and has published in prestigious journals such as PLoS ONE, Free Radical Biology and Medicine and Environmental Pollution.

In The Last Decade

Shimeng Zhou

21 papers receiving 363 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Shimeng Zhou China 12 161 79 59 50 41 22 367
Chao Luo China 12 174 1.1× 40 0.5× 59 1.0× 25 0.5× 43 1.0× 29 432
Ha Jin Jeong South Korea 13 165 1.0× 39 0.5× 16 0.3× 43 0.9× 41 1.0× 24 425
Renyuan Zhou China 10 194 1.2× 38 0.5× 59 1.0× 22 0.4× 39 1.0× 17 453
Ahmed S. Kamel Egypt 10 149 0.9× 111 1.4× 61 1.0× 56 1.1× 17 0.4× 31 416
Jerónimo Auzmendi Argentina 15 161 1.0× 128 1.6× 22 0.4× 76 1.5× 32 0.8× 29 536
Yuka Ikeda Japan 12 287 1.8× 34 0.4× 25 0.4× 39 0.8× 68 1.7× 47 512
Yumi Watanabe Japan 13 199 1.2× 73 0.9× 122 2.1× 67 1.3× 11 0.3× 37 444
Lifeng Zheng China 8 119 0.7× 48 0.6× 23 0.4× 33 0.7× 24 0.6× 18 293

Countries citing papers authored by Shimeng Zhou

Since Specialization
Citations

This map shows the geographic impact of Shimeng Zhou's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Shimeng Zhou with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Shimeng Zhou more than expected).

Fields of papers citing papers by Shimeng Zhou

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Shimeng Zhou. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Shimeng Zhou. The network helps show where Shimeng Zhou may publish in the future.

Co-authorship network of co-authors of Shimeng Zhou

This figure shows the co-authorship network connecting the top 25 collaborators of Shimeng Zhou. A scholar is included among the top collaborators of Shimeng Zhou based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Shimeng Zhou. Shimeng Zhou is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Chen, Hongqiang, Yong Zeng, Yu Shi, et al.. (2025). ZNF831-YTHDF1-DNMT1/3a feedback loop regulates lung carcinogenesis and progression through WNT7B-FZD5-β-catenin signalling axis. Free Radical Biology and Medicine. 241. 338–352.
2.
Wang, Bo, Qiaoyu Liu, Sheng Guo, et al.. (2025). MVP-LCN2 axis triggers evasion of ferroptosis to drive hepatocarcinogenesis and sorafenib resistance. Drug Resistance Updates. 81. 101246–101246. 5 indexed citations
3.
Zhou, Shimeng, Yu Shi, Na Wang, et al.. (2025). Bisphenol F induces spermatogenic cell ferroptosis via FTO-mediated m6A regulation of FTH1. Free Radical Biology and Medicine. 229. 364–373. 2 indexed citations
4.
5.
Shi, Yu, Shimeng Zhou, Na Wang, et al.. (2024). FTO mediates bisphenol F-induced blood-testis barrier impairment through regulating ferroptosis via YTHDF1/TfRc and YTHDF2/SLC7A11 signal axis. Environmental Pollution. 359. 124531–124531. 11 indexed citations
6.
Zhou, Shimeng, Wenbo Yuan, Hongqiang Chen, et al.. (2023). RNA binding protein YTHDF1 mediates bisphenol S-induced Leydig cell damage by regulating the mitochondrial pathway of BCL2 and the expression of CDK2-CyclinE1. Environmental Pollution. 321. 121144–121144. 3 indexed citations
7.
Zhou, Shimeng, Yong Zeng, Wenbo Yuan, et al.. (2023). FTO-Nrf2 axis regulates bisphenol F-induced leydig cell toxicity in an m6A-YTHDF2-dependent manner. Environmental Pollution. 325. 121393–121393. 25 indexed citations
8.
Luo, Li, Le Yang, Kun Zhang, et al.. (2023). Caveolin-1-Mediated Cholesterol Accumulation Contributes to Exaggerated mGluR-Dependent Long-Term Depression and Impaired Cognition in Fmr1 Knockout Mice. Molecular Neurobiology. 60(6). 3379–3395. 8 indexed citations
9.
Zeng, Yong, Hongqiang Chen, Zhe Zhang, et al.. (2022). IFI44L as a novel epigenetic silencing tumor suppressor promotes apoptosis through JAK/STAT1 pathway during lung carcinogenesis. Environmental Pollution. 319. 120943–120943. 14 indexed citations
10.
11.
Zeng, Yong, Zhe Zhang, Hongqiang Chen, et al.. (2022). Comprehensive Analysis of Immune Implication and Prognostic Value of IFI44L in Non-Small Cell Lung Cancer. Frontiers in Oncology. 11. 798425–798425. 6 indexed citations
12.
Zhou, Shimeng, Wenbo Yuan, Yong Zeng, et al.. (2022). TET1 involved in bisphenol A induced TM3 Leydig cell toxicity by regulating Cav3.3 hydroxymethylation. Chemosphere. 312(Pt 1). 137171–137171. 4 indexed citations
13.
Zhou, Shimeng, Jinfeng Zhu, Jingwei Xu, et al.. (2022). Anti‐tumour potential of PD‐L1/PD‐1 post‐translational modifications. Immunology. 167(4). 471–481. 25 indexed citations
14.
Yuan, Wenbo, Hongqiang Chen, Shimeng Zhou, et al.. (2021). TET1 mediated male reproductive toxicity induced by Bisphenol A through Catsper-Ca2+ signaling pathway. Environmental Pollution. 296. 118739–118739. 10 indexed citations
15.
Zhou, Shimeng, Le Yang, Liu-kun Yang, et al.. (2017). Cucurbitacin IIa exerts antidepressant-like effects on mice exposed to chronic unpredictable mild stress. Neuroreport. 28(5). 259–267. 19 indexed citations
16.
Shi, Qixin, et al.. (2017). The novel cannabinoid receptor GPR55 mediates anxiolytic-like effects in the medial orbital cortex of mice with acute stress. Molecular Brain. 10(1). 38–38. 47 indexed citations
17.
Zhang, Huinan, Di Qu, Ling Wang, et al.. (2016). An Orally Active Allosteric GLP-1 Receptor Agonist Is Neuroprotective in Cellular and Rodent Models of Stroke. PLoS ONE. 11(2). e0148827–e0148827. 45 indexed citations
18.
Wang, Jing, Le Yang, Shimeng Zhou, et al.. (2016). Effect of Praeruptorin C on 3-nitropropionic acid induced Huntington’s disease-like symptoms in mice. Biomedicine & Pharmacotherapy. 86. 81–87. 48 indexed citations
19.
Zhang, Huinan, Jingru Meng, Shimeng Zhou, et al.. (2015). Intranasal Delivery of Exendin-4 Confers Neuroprotective Effect Against Cerebral Ischemia in Mice. The AAPS Journal. 18(2). 385–394. 33 indexed citations
20.
Zhang, Huinan, Jingru Meng, Xubo Li, et al.. (2015). Pro-GLP-1, a Pro-drug of GLP-1, is neuroprotective in cerebral ischemia. European Journal of Pharmaceutical Sciences. 70. 82–91. 22 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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