Shigemasa Hanazawa

3.5k total citations
102 papers, 3.0k citations indexed

About

Shigemasa Hanazawa is a scholar working on Molecular Biology, Immunology and Periodontics. According to data from OpenAlex, Shigemasa Hanazawa has authored 102 papers receiving a total of 3.0k indexed citations (citations by other indexed papers that have themselves been cited), including 54 papers in Molecular Biology, 43 papers in Immunology and 31 papers in Periodontics. Recurrent topics in Shigemasa Hanazawa's work include Immune Response and Inflammation (37 papers), Oral microbiology and periodontitis research (31 papers) and Bone Metabolism and Diseases (19 papers). Shigemasa Hanazawa is often cited by papers focused on Immune Response and Inflammation (37 papers), Oral microbiology and periodontitis research (31 papers) and Bone Metabolism and Diseases (19 papers). Shigemasa Hanazawa collaborates with scholars based in Japan, United States and United Kingdom. Shigemasa Hanazawa's co-authors include S Kitano, Shigeru Amano, Akira Takeshita, Yoshihiro Ohmori, Shigeo Kitano, Ken Ozaki, Yukio Murakami, Noboru Yamaguchi, Yoshikazu Masuhiro and Yasuhiro Kawata and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Biological Chemistry and Nature Communications.

In The Last Decade

Shigemasa Hanazawa

100 papers receiving 2.9k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Shigemasa Hanazawa Japan 32 1.3k 931 854 456 416 102 3.0k
Tokio Osaki Japan 32 1.0k 0.8× 359 0.4× 648 0.8× 411 0.9× 710 1.7× 144 3.4k
Keith L. Kirkwood United States 39 2.0k 1.6× 1.1k 1.2× 2.1k 2.4× 675 1.5× 814 2.0× 119 5.2k
Yoshihiro Ohmori Japan 35 1.1k 0.8× 2.2k 2.4× 388 0.5× 646 1.4× 1.4k 3.4× 124 4.1k
Kuniaki Okamoto Japan 33 2.0k 1.6× 428 0.5× 1.3k 1.5× 705 1.5× 495 1.2× 102 3.6k
Piotr Mydel Norway 26 825 0.6× 648 0.7× 797 0.9× 145 0.3× 194 0.5× 58 2.6k
Xin Huang China 28 778 0.6× 1.9k 2.0× 350 0.4× 138 0.3× 817 2.0× 93 3.5k
Jeom‐Il Choi South Korea 27 588 0.5× 667 0.7× 897 1.1× 149 0.3× 112 0.3× 85 2.1k
Naoki Takahashi Japan 26 996 0.8× 300 0.3× 976 1.1× 111 0.2× 159 0.4× 96 2.5k
Eisaku Ueta Japan 28 645 0.5× 254 0.3× 476 0.6× 199 0.4× 329 0.8× 81 2.3k
C A Salkowski United States 25 540 0.4× 1.7k 1.9× 128 0.1× 366 0.8× 455 1.1× 36 2.7k

Countries citing papers authored by Shigemasa Hanazawa

Since Specialization
Citations

This map shows the geographic impact of Shigemasa Hanazawa's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Shigemasa Hanazawa with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Shigemasa Hanazawa more than expected).

Fields of papers citing papers by Shigemasa Hanazawa

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Shigemasa Hanazawa. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Shigemasa Hanazawa. The network helps show where Shigemasa Hanazawa may publish in the future.

Co-authorship network of co-authors of Shigemasa Hanazawa

This figure shows the co-authorship network connecting the top 25 collaborators of Shigemasa Hanazawa. A scholar is included among the top collaborators of Shigemasa Hanazawa based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Shigemasa Hanazawa. Shigemasa Hanazawa is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Itoh, Kikuji, Yoshika Momose, Kazumi Kasakura, et al.. (2021). Post-translational suppression of the high affinity IgE receptor expression on mast cells by an intestinal bacterium. Immunobiology. 226(2). 152056–152056. 6 indexed citations
2.
Masuhiro, Yoshikazu, et al.. (2016). TIPE2 (Tumor Necrosis Factor α-induced Protein 8-like 2) Is a Novel Negative Regulator of TAK1 Signal. Journal of Biological Chemistry. 291(43). 22650–22660. 28 indexed citations
3.
Yabu, Takeshi, Hideaki Toda, Yasuhiro Shibasaki, et al.. (2011). Antiviral protection mechanisms mediated by ginbuna crucian carp interferon gamma isoforms 1 and 2 through two distinct interferon gamma-receptors. The Journal of Biochemistry. 150(6). 635–648. 50 indexed citations
4.
Yamaguchi, Naomi, et al.. (2006). Induction ofPorphyromonas gingivalisGroEL signaling via binding to Toll‐like receptors 2 and 4. Oral Microbiology and Immunology. 21(4). 245–251. 33 indexed citations
5.
Murakami, Yukio, Masao Shoji, Atsushi Hirata, et al.. (2004). Interleukin-6 negatively regulatesPorphyromonas gingivalisfimbria-stimulated fibronectin expression in human gingival fibroblasts. FEMS Immunology & Medical Microbiology. 43(2). 205–211. 4 indexed citations
6.
Takeshita, Akira, et al.. (2000). Prostaglandin E2 Stimulates AP-1-Mediated CD14 Expression in Mouse Macrophages Via Cyclic AMP-Dependent Protein Kinase A. The Journal of Immunology. 164(10). 5403–5408. 29 indexed citations
7.
Hanazawa, Shigemasa. (1998). Study on Stimulatory Mechanism of Inflammatory Bone Resorption Induced by Porphyromonas gingivalis Department of Oral Microbiology.. Nippon Saikingaku Zasshi. 53(4). 571–586. 1 indexed citations
8.
Takeda, Hiroyuki, et al.. (1998). Sphingomyelinase and ceramide inhibit formation of F‐actin ring in and bone resorption by rabbit mature osteoclasts. FEBS Letters. 422(2). 255–258. 21 indexed citations
9.
Murakami, Yukio, et al.. (1998). Fibronectin in saliva inhibitsPorphyromonas gingivalisfimbria-induced expression of inflammatory cytokine gene in mouse macrophages. FEMS Immunology & Medical Microbiology. 22(3). 257–262. 21 indexed citations
10.
Murakami, Yukio, et al.. (1994). Porphyromonas gingivalis fimbriae induce a 68-kilodalton phosphorylated protein in macrophages. Infection and Immunity. 62(12). 5242–5246. 22 indexed citations
11.
Kawata, Yasuhiro, Shigemasa Hanazawa, Shigeru Amano, et al.. (1994). Porphyromonas gingivalis fimbriae stimulate bone resorption in vitro. Infection and Immunity. 62(7). 3012–3016. 56 indexed citations
12.
Nagatsuka, Yasuko, et al.. (1994). Anti-bacterial antibodies in Epstein-Barr virus (EBV)-transformed oligoclonal B-cell lines established from normal persons and autoimmune disease patients. Letters in Applied Microbiology. 19(4). 206–209. 1 indexed citations
13.
Takeshita, Akira, et al.. (1993). IL-1 induces expression of monocyte chemoattractant JE in clonal mouse osteoblastic cell line MC3T3-E1. The Journal of Immunology. 150(4). 1554–1562. 23 indexed citations
14.
Ohmori, Yoshihiro, Shigemasa Hanazawa, Shigeru Amano, et al.. (1988). Inducing effect of periodontopathic bacteria on interleukin‐1 production by mouse peritoneal macrophages. Oral Microbiology and Immunology. 3(4). 169–172. 18 indexed citations
15.
Hanazawa, Shigemasa, Shigeru Amano, Kimiharu Hirose, et al.. (1988). Inductive effect of recombinant human interleukin‐1 α and β on differentiation of macrophage‐like tumor cell line P388D1. Journal of Cellular Physiology. 136(3). 543–546. 4 indexed citations
16.
Ohmori, Yoshihiro, et al.. (1986). Functional roles of macrophages in periodontal disease. II Macrophages produce a factor stimulating growth and differentiation of an osteoblastic cell line (MC3T3-E1).:Macrophages produce a factor stimulating growth and differentiation of an osteoblastic cell line (MC3T3-E1). Journal of Oral Biosciences. 28(5). 646–653. 1 indexed citations
17.
Hanazawa, Shigemasa, Yoshihiro Ohmori, Shigeru Amano, et al.. (1986). Human purified interleukin‐1 inhibits DNA synthesis and cell growth of osteoblastic cell line (MC3T3‐E1), but enhances alkaline phosphatase activity in the cells. FEBS Letters. 203(2). 279–284. 27 indexed citations
18.
Hanazawa, Shigemasa, et al.. (1986). Comparison of the activation and interleukin-1 production of mouse peritoneal macrophages by three types of purified carrageenan.. PubMed. 15(3). 557–66. 2 indexed citations
19.
Ohmori, Yoshihiro, et al.. (1985). Functional role of macrophages in periodontal disease: I. Activation of macrophage functions by Bacteroides gingivalis.:I. Activation of macrophage functions by Bacteroides gingivalis. 27(2). 728–732.
20.
Hanazawa, Shigemasa, et al.. (1976). A Comparative Study of Biological Activities among Bacterial Strains of Anaerobic Coryneforms and Propionibacteria. Nippon Saikingaku Zasshi. 31(5). 613–620. 1 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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