Shali Wang

692 total citations
28 papers, 554 citations indexed

About

Shali Wang is a scholar working on Molecular Biology, Cognitive Neuroscience and Genetics. According to data from OpenAlex, Shali Wang has authored 28 papers receiving a total of 554 indexed citations (citations by other indexed papers that have themselves been cited), including 13 papers in Molecular Biology, 8 papers in Cognitive Neuroscience and 8 papers in Genetics. Recurrent topics in Shali Wang's work include Genetics and Neurodevelopmental Disorders (7 papers), Autism Spectrum Disorder Research (7 papers) and Ginseng Biological Effects and Applications (4 papers). Shali Wang is often cited by papers focused on Genetics and Neurodevelopmental Disorders (7 papers), Autism Spectrum Disorder Research (7 papers) and Ginseng Biological Effects and Applications (4 papers). Shali Wang collaborates with scholars based in China and United States. Shali Wang's co-authors include Yan Wang, Liu Tu, Feng Yang, Di Chen, Yingbo Li, Yan Wang, Jingjing Shen, Ping Xiang, Di Chen and Di Chen and has published in prestigious journals such as SHILAP Revista de lepidopterología, Journal of Pharmacology and Experimental Therapeutics and Advanced Science.

In The Last Decade

Shali Wang

28 papers receiving 551 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Shali Wang China 11 232 119 102 58 48 28 554
Chunli Zhao China 14 305 1.3× 49 0.4× 58 0.6× 120 2.1× 67 1.4× 48 777
Hossein Mostafavi Iran 14 201 0.9× 51 0.4× 45 0.4× 44 0.8× 61 1.3× 50 577
Shuang Yu China 15 232 1.0× 54 0.5× 21 0.2× 53 0.9× 31 0.6× 43 702
Paula C. Reggiani Argentina 13 159 0.7× 51 0.4× 19 0.2× 47 0.8× 61 1.3× 38 580
Isabel Paiva Portugal 12 264 1.1× 88 0.7× 15 0.1× 29 0.5× 56 1.2× 67 771
Zhuo Liu China 18 312 1.3× 28 0.2× 40 0.4× 162 2.8× 141 2.9× 40 811
Chenyi Yang China 14 280 1.2× 23 0.2× 27 0.3× 29 0.5× 39 0.8× 44 647
Reza Ahadi Iran 11 207 0.9× 19 0.2× 67 0.7× 19 0.3× 28 0.6× 35 667

Countries citing papers authored by Shali Wang

Since Specialization
Citations

This map shows the geographic impact of Shali Wang's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Shali Wang with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Shali Wang more than expected).

Fields of papers citing papers by Shali Wang

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Shali Wang. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Shali Wang. The network helps show where Shali Wang may publish in the future.

Co-authorship network of co-authors of Shali Wang

This figure shows the co-authorship network connecting the top 25 collaborators of Shali Wang. A scholar is included among the top collaborators of Shali Wang based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Shali Wang. Shali Wang is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
2.
Zheng, Wenxia, et al.. (2024). Sortilin is associated with progranulin deficiency and autism‐like behaviors in valproic acid‐induced autism rats. CNS Neuroscience & Therapeutics. 30(9). e70015–e70015. 1 indexed citations
3.
Wang, Yan, Jing Deng, Xiaoya Wang, et al.. (2023). Melatonin ameliorates sleep–wake disturbances and autism‐like behaviors in the Ctnnd2 knock out mouse model of autism spectrum disorders. Genes Brain & Behavior. 22(4). e12852–e12852. 5 indexed citations
4.
Xu, Man, Yan Wang, Feifei Wang, et al.. (2023). Melatonin improves synapse development by PI3K/Akt signaling in a mouse model of autism spectrum disorder. Neural Regeneration Research. 19(7). 1618–1624. 7 indexed citations
5.
Wang, Xiaoya, et al.. (2023). Effects of chronic sleep restriction on the neuro‐phenotypes of Ctnnd2 knockout mice. Brain and Behavior. 13(7). e3075–e3075. 1 indexed citations
6.
Wang, Lili, Jianhui Chen, Wenxia Zheng, et al.. (2022). Progranulin improves neural development via the PI3K/Akt/GSK-3β pathway in the cerebellum of a VPA-induced rat model of ASD. Translational Psychiatry. 12(1). 114–114. 28 indexed citations
7.
Wang, Xiaoqing, Wenxia Zheng, Shali Wang, et al.. (2021). Abnormal spatiotemporal expression pattern of progranulin and neurodevelopment impairment in VPA-induced ASD rat model. Neuropharmacology. 196. 108689–108689. 13 indexed citations
8.
Wang, Yan, et al.. (2020). Regulation of progranulin expression and location by sortilin in oxygen–glucose deprivation/reoxygenation injury. Neuroscience Letters. 738. 135394–135394. 5 indexed citations
9.
Yu, Yang, Chen Chen, Jinmu Deng, et al.. (2019). ATP1A1 Integrates AKT and ERK Signaling via Potential Interaction With Src to Promote Growth and Survival in Glioma Stem Cells. Frontiers in Oncology. 9. 320–320. 16 indexed citations
10.
Zheng, Wenxia, et al.. (2019). [Improvement of a mouse model of valproic acid-induced autism].. PubMed. 39(6). 718–723. 9 indexed citations
11.
Wang, Yan, et al.. (2018). A Developmental Study of Abnormal Behaviors and Altered GABAergic Signaling in the VPA-Treated Rat Model of Autism. Frontiers in Behavioral Neuroscience. 12. 182–182. 86 indexed citations
12.
Tu, Liu, Yan Wang, Di Chen, et al.. (2018). Protective Effects of Notoginsenoside R1 via Regulation of the PI3K-Akt-mTOR/JNK Pathway in Neonatal Cerebral Hypoxic–Ischemic Brain Injury. Neurochemical Research. 43(6). 1210–1226. 78 indexed citations
13.
Wang, Yan, et al.. (2016). Notoginsenoside R1 Protects against Neonatal Cerebral Hypoxic-Ischemic Injury through Estrogen Receptor–Dependent Activation of Endoplasmic Reticulum Stress Pathways. Journal of Pharmacology and Experimental Therapeutics. 357(3). 591–605. 46 indexed citations
14.
15.
Li, Xiangyun, Ming Zhu, Wei Guo, et al.. (2015). Ligustrazine Suppresses the Growth of HRPC Cells through the Inhibition of Cap- Dependent Translation Via Both the mTOR and the MEK/ERK Pathways. Anti-Cancer Agents in Medicinal Chemistry. 15(6). 764–772. 18 indexed citations
16.
Xia, Hai‐Feng, et al.. (2014). Molecular Modification of Protein A to Improve the Elution pH and Alkali Resistance in Affinity Chromatography. Applied Biochemistry and Biotechnology. 172(8). 4002–4012. 13 indexed citations
17.
Dai, Hui, Hong Huang, Shali Wang, et al.. (2012). [Role of tumor necrosis factor α in endothelial-mesenchymal transition in vitro].. PubMed. 28(1). 19–24. 2 indexed citations
18.
Xu, Xiang, Fangqiang Zhu, Meng Zhang, et al.. (2012). Stromal Cell-Derived Factor-1 Enhances Wound Healing through Recruiting Bone Marrow-Derived Mesenchymal Stem Cells to the Wound Area and Promoting Neovascularization. Cells Tissues Organs. 197(2). 103–113. 104 indexed citations
19.
Guo, Rui, Zuoren Yu, Jikui Guan, et al.. (2004). Stage‐specific and tissue‐specific expression characteristics of differentially expressed genes during mouse spermatogenesis. Molecular Reproduction and Development. 67(3). 264–272. 56 indexed citations
20.
Wang, Shali, et al.. (2003). [Modulation of expression of human GM-CSF and GM-CSFRalpha by total saponins of Panax ginseng].. PubMed. 55(4). 487–92. 2 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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