Seiichi Ishizuka
- Pathology and Forensic Medicine top 0.5%
- Genetics top 2%
- Molecular Biology
- Cell Biology top 2%
- Oncology top 10%
- Co-authors
- Anthony W. NormanDaishiro MiuraNoriyoshi KuriharaSachio IshimotoKeiichi OzonoKazuya TakenouchiMasayoshi KumegawaJill E. Bishop
- Topics
- Vitamin D Research Studies (84 papers)Biotin and Related Studies (35 papers)Estrogen and related hormone effects (29 papers)
- Journals
- Proceedings of the National Academy of SciencesNucleic Acids ResearchJournal of Biological Chemistry
- Partner nations
- JapanUnited StatesNew Zealand
In The Last Decade
Seiichi Ishizuka
105 papers receiving 2.3k citations
Peers
Comparison fields: 5 of 94
- Pathology and Forensic Medicine 1.6k
- Genetics 825
- Molecular Biology 691
- Cell Biology 532
- Oncology 332
Countries citing papers authored by Seiichi Ishizuka
This map shows the geographic impact of Seiichi Ishizuka's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Seiichi Ishizuka with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Seiichi Ishizuka more than expected).
Fields of papers citing papers by Seiichi Ishizuka
This network shows the impact of papers produced by Seiichi Ishizuka. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Seiichi Ishizuka. The network helps show where Seiichi Ishizuka may publish in the future.
Co-authorship network of co-authors of Seiichi Ishizuka
This figure shows the co-authorship network connecting the top 25 collaborators of Seiichi Ishizuka. A scholar is included among the top collaborators of Seiichi Ishizuka based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Seiichi Ishizuka. Seiichi Ishizuka is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 4 | |
| 2 | 25 | |
| 3 | 9 | |
| 4 | 13 | |
| 5 | 10 | |
| 6 | 67 | |
| 7 | 33 | |
| 8 | 100 | |
| 9 | 80 | |
| 10 | 35 | |
| 11 | 16 | |
| 12 | 25 | |
| 13 | 18 | |
| 14 | 52 | |
| 15 | 8 | |
| 16 | 27 | |
| 17 | 15 | |
| 18 | 16 | |
| 19 | 1 | |
| 20 | 4 |
About Seiichi Ishizuka
Seiichi Ishizuka is a scholar working on Pathology and Forensic Medicine, Cell Biology and Genetics, having authored 105 papers that have together received 2.4k indexed citations. Recurring topics across this work include Vitamin D Research Studies (84 papers), Biotin and Related Studies (35 papers) and Estrogen and related hormone effects (29 papers). The work is most often cited by research in Pathology and Forensic Medicine (1.6k citations), Cell Biology (532 citations) and Genetics (825 citations). Seiichi Ishizuka has collaborated with scholars based in Japan, United States and New Zealand. Frequent co-authors include Anthony W. Norman, Daishiro Miura, Noriyoshi Kurihara, Sachio Ishimoto, Keiichi Ozono, Kazuya Takenouchi, Masayoshi Kumegawa, Jill E. Bishop, Hiroaki Takayama and Kenji Manabe. Their work appears in journals such as Proceedings of the National Academy of Sciences, Nucleic Acids Research and Journal of Biological Chemistry.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.