Sébastien Pinte

1.9k total citations
25 papers, 1.4k citations indexed

About

Sébastien Pinte is a scholar working on Molecular Biology, Immunology and Oncology. According to data from OpenAlex, Sébastien Pinte has authored 25 papers receiving a total of 1.4k indexed citations (citations by other indexed papers that have themselves been cited), including 22 papers in Molecular Biology, 5 papers in Immunology and 4 papers in Oncology. Recurrent topics in Sébastien Pinte's work include Epigenetics and DNA Methylation (10 papers), DNA Repair Mechanisms (4 papers) and RNA modifications and cancer (4 papers). Sébastien Pinte is often cited by papers focused on Epigenetics and DNA Methylation (10 papers), DNA Repair Mechanisms (4 papers) and RNA modifications and cancer (4 papers). Sébastien Pinte collaborates with scholars based in France, United States and Switzerland. Sébastien Pinte's co-authors include Dominique Leprince, Cateline Guérardel, Sophie Deltour, Didier Monté, Nicolas Stankovic‐Valentin, Jean Coll, Jean‐Jacques Curgy, Brigitte Quatannens, Éric Adriaenssens and Séverine Lottin and has published in prestigious journals such as Journal of Biological Chemistry, Nature Communications and PLoS ONE.

In The Last Decade

Sébastien Pinte

25 papers receiving 1.4k citations

Peers

Sébastien Pinte
Mark Eckersdorff United States
Gerald Chu United States
Roy Blum United States
Suzan Lazo-Kallanian United States
M. Phillip DeYoung United States
Mark Eckersdorff United States
Sébastien Pinte
Citations per year, relative to Sébastien Pinte Sébastien Pinte (= 1×) peers Mark Eckersdorff

Countries citing papers authored by Sébastien Pinte

Since Specialization
Citations

This map shows the geographic impact of Sébastien Pinte's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Sébastien Pinte with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Sébastien Pinte more than expected).

Fields of papers citing papers by Sébastien Pinte

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Sébastien Pinte. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Sébastien Pinte. The network helps show where Sébastien Pinte may publish in the future.

Co-authorship network of co-authors of Sébastien Pinte

This figure shows the co-authorship network connecting the top 25 collaborators of Sébastien Pinte. A scholar is included among the top collaborators of Sébastien Pinte based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Sébastien Pinte. Sébastien Pinte is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
2.
Nassour, Joe, Sébastien Martien, Nathalie Martin, et al.. (2016). Defective DNA single-strand break repair is responsible for senescence and neoplastic escape of epithelial cells. Nature Communications. 7(1). 10399–10399. 96 indexed citations
3.
Pinte, Sébastien, Alexandra Le Bras, Gaëlle Villain, et al.. (2016). Endothelial Cell Activation Is Regulated by Epidermal Growth Factor-like Domain 7 (Egfl7) during Inflammation. Journal of Biological Chemistry. 291(46). 24017–24028. 28 indexed citations
4.
Baranzelli, Marie‐Christine, Chantal Samson, Charles Fournier, et al.. (2013). Expression of Egfl7 correlates with low-grade invasive lesions in human breast cancer. International Journal of Oncology. 42(4). 1367–1375. 19 indexed citations
5.
Pinte, Sébastien, et al.. (2013). The Reelin receptors ApoER2 and VLDLR are direct target genes of HIC1 (Hypermethylated In Cancer 1). Biochemical and Biophysical Research Communications. 440(3). 424–430. 9 indexed citations
6.
Galati, Alessandra, Frédérique Magdinier, Serge Bauwens, et al.. (2012). TRF2 Controls Telomeric Nucleosome Organization in a Cell Cycle Phase-Dependent Manner. PLoS ONE. 7(4). e34386–e34386. 36 indexed citations
7.
Pinte, Sébastien & Fabrice Soncin. (2012). Egfl7 promotes tumor escape from immunity. OncoImmunology. 1(3). 375–376. 9 indexed citations
8.
Dehennaut, Vanessa, et al.. (2012). Molecular dissection of the interaction between HIC1 and SIRT1. Biochemical and Biophysical Research Communications. 421(2). 384–388. 12 indexed citations
9.
Foveau, Bénédicte, Gaylor Boulay, Sébastien Pinte, et al.. (2011). The Receptor Tyrosine Kinase EphA2 Is a Direct Target Gene of Hypermethylated in Cancer 1 (HIC1). Journal of Biological Chemistry. 287(8). 5366–5378. 28 indexed citations
10.
Rechem, Capucine Van, Brian R. Rood, Sébastien Pinte, et al.. (2009). Scavenger Chemokine (CXC Motif) Receptor 7 (CXCR7) Is a Direct Target Gene of HIC1 (Hypermethylated in Cancer 1). Journal of Biological Chemistry. 284(31). 20927–20935. 66 indexed citations
11.
Amiard, Simon, Michel Doudeau, Sébastien Pinte, et al.. (2007). A topological mechanism for TRF2-enhanced strand invasion. SPIRE - Sciences Po Institutional REpository. 6 indexed citations
12.
Amiard, Simon, Michel Doudeau, Sébastien Pinte, et al.. (2007). A topological mechanism for TRF2-enhanced strand invasion. Nature Structural & Molecular Biology. 14(2). 147–154. 149 indexed citations
13.
Berteaux, Nathalie, Séverine Lottin, Didier Monté, et al.. (2005). H19 mRNA-like Noncoding RNA Promotes Breast Cancer Cell Proliferation through Positive Control by E2F1. Journal of Biological Chemistry. 280(33). 29625–29636. 303 indexed citations
14.
Pinte, Sébastien, Nicolas Stankovic‐Valentin, Sophie Deltour, et al.. (2004). The Tumor Suppressor Gene HIC1 (Hypermethylated in Cancer 1) Is a Sequence-specific Transcriptional Repressor. Journal of Biological Chemistry. 279(37). 38313–38324. 71 indexed citations
15.
Lefebvre, Tony, Sébastien Pinte, Cateline Guérardel, et al.. (2004). The tumor suppressor HIC1 (hypermethylated in cancer 1) is O‐GlcNAc glycosylated. European Journal of Biochemistry. 271(19). 3843–3854. 25 indexed citations
16.
Pinte, Sébastien, et al.. (2004). Identification of a second G–C-rich promoter conserved in the human, murine and rat tumor suppressor genes HIC1. Oncogene. 23(22). 4023–4031. 15 indexed citations
17.
Bertrand, Stéphanie, Sébastien Pinte, Nicolas Stankovic‐Valentin, et al.. (2004). Identification and developmental expression of the zebrafish orthologue of the tumor suppressor gene HIC1. Biochimica et Biophysica Acta (BBA) - Gene Structure and Expression. 1678(1). 57–66. 11 indexed citations
18.
Deltour, Sophie, Sébastien Pinte, Cateline Guérardel, Bohdan Wasylyk, & Dominique Leprince. (2002). The Human Candidate Tumor Suppressor Gene HIC1 Recruits CtBP through a Degenerate GLDLSKK Motif. Molecular and Cellular Biology. 22(13). 4890–4901. 84 indexed citations
19.
Deltour, Sophie, Sébastien Pinte, Cateline Guérardel, & Dominique Leprince. (2001). Characterization of HRG22, a Human Homologue of the Putative Tumor Suppressor Gene HIC1. Biochemical and Biophysical Research Communications. 287(2). 427–434. 33 indexed citations
20.
Guérardel, Cateline, Sophie Deltour, Sébastien Pinte, et al.. (2001). Identification in the Human Candidate Tumor Suppressor GeneHIC-1 of a New Major Alternative TATA-less Promoter Positively Regulated by p53. Journal of Biological Chemistry. 276(5). 3078–3089. 67 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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