Sébastien Gaumer

1.3k total citations
23 papers, 1.0k citations indexed

About

Sébastien Gaumer is a scholar working on Molecular Biology, Epidemiology and Cell Biology. According to data from OpenAlex, Sébastien Gaumer has authored 23 papers receiving a total of 1.0k indexed citations (citations by other indexed papers that have themselves been cited), including 15 papers in Molecular Biology, 8 papers in Epidemiology and 6 papers in Cell Biology. Recurrent topics in Sébastien Gaumer's work include Autophagy in Disease and Therapy (7 papers), Cell death mechanisms and regulation (7 papers) and Endoplasmic Reticulum Stress and Disease (6 papers). Sébastien Gaumer is often cited by papers focused on Autophagy in Disease and Therapy (7 papers), Cell death mechanisms and regulation (7 papers) and Endoplasmic Reticulum Stress and Disease (6 papers). Sébastien Gaumer collaborates with scholars based in France, United States and Norway. Sébastien Gaumer's co-authors include Bernard Mignotte, Isabelle Guénal, Didier Contamine, Sylvain Brun, Harald Stenmark, Andreas Brech, Kim D. Finley, Tor Erik Rusten, Ioannis P. Nezis and Antonia P. Sagona and has published in prestigious journals such as Journal of Biological Chemistry, The Journal of Cell Biology and The EMBO Journal.

In The Last Decade

Sébastien Gaumer

22 papers receiving 1.0k citations

Peers

Sébastien Gaumer

EPIDE

CMN

IMMUN

Christine Powers United States

Catherine Creppe Belgium

Monika Bug Germany

Misako Okuno Japan

Sharan Swarup United States

Steve Jean Canada

Robert Köchl United Kingdom

Mónika Lippai Hungary

Takahide Matsui Japan

H.C. Ardley United Kingdom

Christine Powers United States

Sébastien Gaumer

768 ×1.3

345 ×1.0

EPIDE

331 ×1.3

60 ×0.4

CMN

77 ×0.6

IMMUN

Citations per year, relative to Sébastien Gaumer Sébastien Gaumer (= 1×) peers Christine Powers

Countries citing papers authored by Sébastien Gaumer

Since Specialization

Citations

This map shows the geographic impact of Sébastien Gaumer's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Sébastien Gaumer with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Sébastien Gaumer more than expected).

Fields of papers citing papers by Sébastien Gaumer

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Sébastien Gaumer. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Sébastien Gaumer. The network helps show where Sébastien Gaumer may publish in the future.

Co-authorship network of co-authors of Sébastien Gaumer

This figure shows the co-authorship network connecting the top 25 collaborators of Sébastien Gaumer. A scholar is included among the top collaborators of Sébastien Gaumer based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Sébastien Gaumer. Sébastien Gaumer is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Estaquier, Jérôme, et al.. (2024). ATF4 Signaling in HIV-1 Infection: Viral Subversion of a Stress Response Transcription Factor. Biology. 13(3). 146–146. 4 indexed citations

Gaumer, Sébastien, et al.. (2023). DREAM a little dREAM of DRM: Model organisms and conservation of DREAM‐like complexes. BioEssays. 46(2). e2300125–e2300125. 3 indexed citations

Bréban, Maxime, et al.. (2021). Lessons on SpA pathogenesis from animal models. Seminars in Immunopathology. 43(2). 207–219. 14 indexed citations

Mignotte, Bernard, et al.. (2019). The endoplasmic reticulum unfolded protein response varies depending on the affected region of the tissue but independently from the source of stress. Cell Stress and Chaperones. 24(4). 817–824.

Araujo, Luiza M., Simon Glatigny, Gilles Chiocchia, et al.. (2019). HLA-B27 alters BMP/TGFβ signalling in Drosophila, revealing putative pathogenic mechanism for spondyloarthritis. Annals of the Rheumatic Diseases. 78(12). 1653–1662. 23 indexed citations

Rincheval, Vincent, et al.. (2016). Identification of MARCKS, FLJ11383 and TAF1B as putative novel target genes in colorectal carcinomas with mocrosatellite instability. YUHSpace (Yonsei University Medical Library). 12 indexed citations

Gaumer, Sébastien. (2016). SP0138 The Fruit Fly, as A Tool To Study ER Stress. Annals of the Rheumatic Diseases. 75. 33–33. 1 indexed citations

Mignotte, Bernard, et al.. (2015). Quantification of Ataxin-3 and Ataxin-7 aggregates formed in vivo in Drosophila reveals a threshold of aggregated polyglutamine proteins associated with cellular toxicity. Biochemical and Biophysical Research Communications. 464(4). 1060–1065. 2 indexed citations

Colin, Jessie, et al.. (2014). The drosophila Bcl-2 family protein Debcl is targeted to the proteasome by the β-TrCP homologue slimb. APOPTOSIS. 19(10). 1444–1456. 6 indexed citations

10.

Saitoh, Yuji, Nobuhiro Fujikake, H. Akiko Popiel, et al.. (2014). p62 Plays a Protective Role in the Autophagic Degradation of Polyglutamine Protein Oligomers in Polyglutamine Disease Model Flies. Journal of Biological Chemistry. 290(3). 1442–1453. 42 indexed citations

11.

Mignotte, Bernard, et al.. (2014). The PERK pathway independently triggers apoptosis and a Rac1/Slpr/JNK/Dilp8 signaling favoring tissue homeostasis in a chronic ER stress Drosophila model. Cell Death and Disease. 5(10). e1452–e1452. 43 indexed citations

12.

Gaumer, Sébastien, et al.. (2012). zVAD-fmk upregulates caspase-9 cleavage and activity in etoposide-induced cell death of mouse embryonic fibroblasts. Biochimica et Biophysica Acta (BBA) - Molecular Cell Research. 1823(8). 1343–1352. 16 indexed citations

13.

Boivin, Antoine, et al.. (2008). Life span extension by dietary restriction is reduced but not abolished by loss of both SIR2 and HST2 in Podospora anserina. Mechanisms of Ageing and Development. 129(12). 714–721. 4 indexed citations

14.

Lindmo, Karine, Andreas Brech, Kim D. Finley, et al.. (2008). The PI 3-kinase regulator Vps15 is required for autophagic clearance of protein aggregates. Autophagy. 4(4). 500–506. 55 indexed citations

15.

Nezis, Ioannis P., Anne Simonsen, Antonia P. Sagona, et al.. (2008). Ref(2)P, the Drosophila melanogaster homologue of mammalian p62, is required for the formation of protein aggregates in adult brain. The Journal of Cell Biology. 180(6). 1065–1071. 291 indexed citations

16.

Boeddrich, Annett, Sébastien Gaumer, Annette Haacke, et al.. (2006). An arginine/lysine‐rich motif is crucial for VCP/p97‐mediated modulation of ataxin‐3 fibrillogenesis. The EMBO Journal. 25(7). 1547–1558. 131 indexed citations

17.

Brun, Sylvain, Vincent Rincheval, Sébastien Gaumer, Bernard Mignotte, & Isabelle Guénal. (2002). reaper and bax initiate two different apoptotic pathways affecting mitochondria and antagonized by bcl-2 in Drosophila. Oncogene. 21(42). 6458–6470. 95 indexed citations

18.

Gaumer, Sébastien, Isabelle Guénal, Sylvain Brun, & Bernard Mignotte. (2002). L’apoptose chez la drosophile : conservation et originalité. médecine/sciences. 18(8-9). 875–880. 1 indexed citations

19.

Gaumer, Sébastien, Isabelle Guénal, Sylvain Brun, Laurent Théodore, & Bernard Mignotte. (2000). Bcl-2 and Bax mammalian regulators of apoptosis are functional in Drosophila. Cell Death and Differentiation. 7(9). 804–814. 111 indexed citations

20.

Guénal, Isabelle, et al.. (1997). Bcl-2 and Hsp27 act at different levels to suppress programmed cell death. Oncogene. 15(3). 347–360. 88 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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