Sebastian Bittner

853 total citations
11 papers, 250 citations indexed

About

Sebastian Bittner is a scholar working on Immunology, Molecular Biology and Oncology. According to data from OpenAlex, Sebastian Bittner has authored 11 papers receiving a total of 250 indexed citations (citations by other indexed papers that have themselves been cited), including 8 papers in Immunology, 7 papers in Molecular Biology and 2 papers in Oncology. Recurrent topics in Sebastian Bittner's work include Cell death mechanisms and regulation (6 papers), Immunotherapy and Immune Responses (4 papers) and T-cell and B-cell Immunology (4 papers). Sebastian Bittner is often cited by papers focused on Cell death mechanisms and regulation (6 papers), Immunotherapy and Immune Responses (4 papers) and T-cell and B-cell Immunology (4 papers). Sebastian Bittner collaborates with scholars based in Germany, United Kingdom and Japan. Sebastian Bittner's co-authors include Martin Ehrenschwender, Gertrud Knoll, Markus Feuerer, Thomas Hehlgans, Harald Wajant, Jonathan Jantsch, Michael Rehli, W. Zeller, Asmita Pant and Nicholas Strieder and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Immunity and FEBS Letters.

In The Last Decade

Sebastian Bittner

11 papers receiving 244 citations

Peers

Sebastian Bittner

IMMUN

MB

ONCOL

EPIDE

CR

Jutta Kollet Germany

Madhura Modak Austria

Dang-Nghiem Vo France

Marilyn Marron United States

Boyoung Shin United States

Sara Rhost Sweden

Anaïs Jiménez-Reinoso Spain

Rémy Char France

Jack Bibby United States

Xuexiao Jin China

Jutta Kollet Germany

Sebastian Bittner

132 ×1.0

IMMUN

84 ×0.8

MB

96 ×1.5

ONCOL

14 ×0.5

EPIDE

20 ×1.0

CR

Citations per year, relative to Sebastian Bittner Sebastian Bittner (= 1×) peers Jutta Kollet

Countries citing papers authored by Sebastian Bittner

Since Specialization

Citations

This map shows the geographic impact of Sebastian Bittner's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Sebastian Bittner with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Sebastian Bittner more than expected).

Fields of papers citing papers by Sebastian Bittner

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Sebastian Bittner. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Sebastian Bittner. The network helps show where Sebastian Bittner may publish in the future.

Co-authorship network of co-authors of Sebastian Bittner

This figure shows the co-authorship network connecting the top 25 collaborators of Sebastian Bittner. A scholar is included among the top collaborators of Sebastian Bittner based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Sebastian Bittner. Sebastian Bittner is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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11 of 11 papers shown

1.

Simon, Malte, Lisa Schmidleithner, Sebastian Bittner, et al.. (2024). Single-cell chromatin accessibility and transposable element landscapes reveal shared features of tissue-residing immune cells. Immunity. 57(8). 1975–1993.e10. 7 indexed citations

2.

Bittner, Sebastian, Thomas Hehlgans, & Markus Feuerer. (2023). Engineered Treg cells as putative therapeutics against inflammatory diseases and beyond. Trends in Immunology. 44(6). 468–483. 56 indexed citations

3.

Bittner, Sebastian, Lisa Schmidleithner, Asmita Pant, et al.. (2022). Biosensors for inflammation as a strategy to engineer regulatory T cells for cell therapy. Proceedings of the National Academy of Sciences. 119(40). e2208436119–e2208436119. 23 indexed citations

4.

Bittner, Sebastian, Gertrud Knoll, & Martin Ehrenschwender. (2017). Hyperosmotic stress enhances cytotoxicity of SMAC mimetics. Cell Death and Disease. 8(8). e2967–e2967. 10 indexed citations

5.

Bittner, Sebastian, Gertrud Knoll, & Martin Ehrenschwender. (2017). Death receptor 3 signaling enhances proliferation of human regulatory T cells. FEBS Letters. 591(8). 1187–1195. 17 indexed citations

6.

Bittner, Sebastian & Martin Ehrenschwender. (2017). Multifaceted death receptor 3 signaling—promoting survival and triggering death. FEBS Letters. 591(17). 2543–2555. 26 indexed citations

7.

Bittner, Sebastian, Gertrud Knoll, & Martin Ehrenschwender. (2016). Death receptor 3 mediates necroptotic cell death. Cellular and Molecular Life Sciences. 74(3). 543–554. 24 indexed citations

8.

Knoll, Gertrud, et al.. (2016). Hypoxia regulates TRAIL sensitivity of colorectal cancer cells through mitochondrial autophagy. Oncotarget. 7(27). 41488–41504. 18 indexed citations

9.

Bittner, Sebastian, et al.. (2015). Soluble TL1A is sufficient for activation of death receptor 3. FEBS Journal. 283(2). 323–336. 26 indexed citations

10.

Ehrenschwender, Martin, et al.. (2014). XIAP-targeting drugs re-sensitize PIK3CA-mutated colorectal cancer cells for death receptor-induced apoptosis. Cell Death and Disease. 5(12). e1570–e1570. 26 indexed citations

11.

Itoh, Katsuhiko, Jutta Friel, Christine Laker, et al.. (1997). The role of soluble growth factors in inducing transient growth and clonal extinction of stroma cell dependent erythroblastic leukemia cells. Leukemia. 11(10). 1753–1761. 17 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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