Sarah Jackson

9.2k total citations · 2 hit papers
36 papers, 2.5k citations indexed

About

Sarah Jackson is a scholar working on Epidemiology, Immunology and Parasitology. According to data from OpenAlex, Sarah Jackson has authored 36 papers receiving a total of 2.5k indexed citations (citations by other indexed papers that have themselves been cited), including 26 papers in Epidemiology, 17 papers in Immunology and 9 papers in Parasitology. Recurrent topics in Sarah Jackson's work include Cytomegalovirus and herpesvirus research (25 papers), Herpesvirus Infections and Treatments (15 papers) and Immune Cell Function and Interaction (10 papers). Sarah Jackson is often cited by papers focused on Cytomegalovirus and herpesvirus research (25 papers), Herpesvirus Infections and Treatments (15 papers) and Immune Cell Function and Interaction (10 papers). Sarah Jackson collaborates with scholars based in United Kingdom, United States and Netherlands. Sarah Jackson's co-authors include Mark R. Wills, Gavin M. Mason, Arne N. Akbar, Milica Vukmanovic‐Stejic, John Sinclair, Noam Stern‐Ginossar, M. Scott Harris, Gaëlle J.S. Talhouarne, Gloria A. Brar and Jonathan S. Weissman and has published in prestigious journals such as Proceedings of the National Academy of Sciences, The Journal of Experimental Medicine and Blood.

In The Last Decade

Sarah Jackson

35 papers receiving 2.5k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Ribosome Profiling Reveals Pervasive Translation Outside of Annotated Protein-Coding Genes

2014 480 citations Nicholas T. Ingolia, Gloria A. Brar et al. Cell Reports profile →
Spontaneous, persistent, T cell–dependent IFN-γ release in patients who progress to Long Covid

2024 41 citations Benjamin A. Krishna, Eleanor Y. Lim et al. Science Advances profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Sarah Jackson United Kingdom	24	1.2k	1.1k	658	245	205	36	2.5k
Gary C. Chan United States	26	1.2k 1.0×	694 0.6×	404 0.6×	133 0.5×	102 0.5×	51	1.9k
Susmit Suvas United States	24	604 0.5×	1.6k 1.4×	304 0.5×	215 0.9×	98 0.5×	51	2.5k
Anna Cerny United States	25	606 0.5×	1.0k 0.9×	518 0.8×	179 0.7×	96 0.5×	29	1.9k
Bence Réthi Sweden	23	282 0.2×	985 0.9×	478 0.7×	181 0.7×	73 0.4×	70	1.8k
Sally S. Atherton United States	28	1.4k 1.2×	530 0.5×	297 0.5×	180 0.7×	155 0.8×	94	2.3k
Ester B. M. Remmerswaal Netherlands	34	1.5k 1.2×	2.9k 2.6×	461 0.7×	801 3.3×	113 0.6×	85	4.3k
Robert N. Lausch United States	31	1.4k 1.2×	1.6k 1.4×	338 0.5×	340 1.4×	83 0.4×	86	3.1k
Sinéad E. Keating Ireland	15	536 0.4×	2.1k 1.9×	1.0k 1.5×	551 2.2×	87 0.4×	18	2.7k
Kenji Akita Japan	11	395 0.3×	1.7k 1.5×	1.4k 2.1×	312 1.3×	205 1.0×	31	2.8k
Sabine M. Lang United States	18	592 0.5×	1.2k 1.1×	1.1k 1.7×	249 1.0×	142 0.7×	25	2.5k

Countries citing papers authored by Sarah Jackson

Since Specialization

Citations

This map shows the geographic impact of Sarah Jackson's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Sarah Jackson with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Sarah Jackson more than expected).

Fields of papers citing papers by Sarah Jackson

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Sarah Jackson. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Sarah Jackson. The network helps show where Sarah Jackson may publish in the future.

Co-authorship network of co-authors of Sarah Jackson

This figure shows the co-authorship network connecting the top 25 collaborators of Sarah Jackson. A scholar is included among the top collaborators of Sarah Jackson based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Sarah Jackson. Sarah Jackson is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

Sort: Min cites: Since: Top N: Style:

20 of 20 papers shown

Krishna, Benjamin A., Eleanor Y. Lim, Sarah Jackson, et al.. (2024). Spontaneous, persistent, T cell–dependent IFN-γ release in patients who progress to Long Covid. Science Advances. 10(8). eadi9379–eadi9379. 41 indexed citations breakdown →

Davies, Emma L., Eleanor Y. Lim, Charlotte J. Houldcroft, et al.. (2022). HCMV carriage in the elderly diminishes anti-viral functionality of the adaptive immune response resulting in virus replication at peripheral sites. Frontiers in Immunology. 13. 1083230–1083230. 6 indexed citations

Poole, Emma, Ian J. Groves, Sarah Jackson, Mark R. Wills, & John Sinclair. (2021). Using Primary Human Cells to Analyze Human Cytomegalovirus Biology. Methods in molecular biology. 2244. 51–81. 11 indexed citations

Jackson, Sarah, Kevin Chen, Ian J. Groves, et al.. (2021). Latent Cytomegalovirus-Driven Recruitment of Activated CD4+ T Cells Promotes Virus Reactivation. Frontiers in Immunology. 12. 657945–657945. 13 indexed citations

Houldcroft, Charlotte J., Sarah Jackson, Eleanor Y. Lim, et al.. (2020). Assessing Anti-HCMV Cell Mediated Immune Responses in Transplant Recipients and Healthy Controls Using a Novel Functional Assay. Frontiers in Cellular and Infection Microbiology. 10. 275–275. 13 indexed citations

Nikolich‐Žugich, Janko, Luka Čičin‐Šain, Donna Collins-McMillen, et al.. (2020). Advances in cytomegalovirus (CMV) biology and its relationship to health, diseases, and aging. GeroScience. 42(2). 495–504. 31 indexed citations

Lim, Eleanor Y., Sarah Jackson, & Mark R. Wills. (2020). The CD4+ T Cell Response to Human Cytomegalovirus in Healthy and Immunocompromised People. Frontiers in Cellular and Infection Microbiology. 10. 202–202. 54 indexed citations

Jackson, Sarah, et al.. (2019). Generation, maintenance and tissue distribution of T cell responses to human cytomegalovirus in lytic and latent infection. Medical Microbiology and Immunology. 208(3-4). 375–389. 37 indexed citations

Poole, Emma, Jessica L. Forbester, Miri Shnayder, et al.. (2019). An iPSC-Derived Myeloid Lineage Model of Herpes Virus Latency and Reactivation. Frontiers in Microbiology. 10. 2233–2233. 15 indexed citations

10.

Jackson, Sarah, et al.. (2017). Latent Cytomegalovirus (CMV) Infection Does Not Detrimentally Alter T Cell Responses in the Healthy Old, But Increased Latent CMV Carriage Is Related to Expanded CMV-Specific T Cells. Frontiers in Immunology. 8. 733–733. 44 indexed citations

11.

Krishna, Benjamin A., Betty Lau, Sarah Jackson, et al.. (2016). Transient activation of human cytomegalovirus lytic gene expression during latency allows cytotoxic T cell killing of latently infected cells. Scientific Reports. 6(1). 24674–24674. 47 indexed citations

12.

Vukmanovic‐Stejic, Milica, Judith A. Seidel, T Sobande, et al.. (2015). The Characterization of Varicella Zoster Virus–Specific T Cells in Skin and Blood during Aging. Journal of Investigative Dermatology. 135(7). 1752–1762. 79 indexed citations

13.

Ingolia, Nicholas T., Gloria A. Brar, Noam Stern‐Ginossar, et al.. (2014). Ribosome Profiling Reveals Pervasive Translation Outside of Annotated Protein-Coding Genes. Cell Reports. 8(5). 1365–1379. 480 indexed citations breakdown →

14.

Mason, Gavin M., Sarah Jackson, Georgina Okecha, et al.. (2013). Human Cytomegalovirus Latency-Associated Proteins Elicit Immune-Suppressive IL-10 Producing CD4+ T Cells. PLoS Pathogens. 9(10). e1003635–e1003635. 66 indexed citations

15.

Jackson, Sarah, John W. Holloway, Jane A. Warner, & Anthony P. Sampson. (2011). Interleukin-13, but Not Indomethacin, Increases Cysteinyl-Leukotriene Synthesis in Human Lung Macrophages. PubMed. 2012. 1–6. 4 indexed citations

16.

Libri, Valentina, Rita I Azevedo, Sarah Jackson, et al.. (2011). Cytomegalovirus infection induces the accumulation of short-lived, multifunctional CD4+ CD45RA+ CD27− T cells: the potential involvement of interleukin-7 in this process. Immunology. 132(3). 326–339. 87 indexed citations

17.

Berg, Pablo J. E. J. van de, Stephen Griffiths, Si‐La Yong, et al.. (2010). Cytomegalovirus Infection Reduces Telomere Length of the Circulating T Cell Pool. The Journal of Immunology. 184(7). 3417–3423. 122 indexed citations

18.

Jackson, Sarah, Gavin M. Mason, & Mark R. Wills. (2010). Human cytomegalovirus immunity and immune evasion. Virus Research. 157(2). 151–160. 203 indexed citations

19.

Fletcher, Jean M., Milica Vukmanovic‐Stejic, Pádraic J. Dunne, et al.. (2005). Cytomegalovirus-Specific CD4+ T Cells in Healthy Carriers Are Continuously Driven to Replicative Exhaustion. The Journal of Immunology. 175(12). 8218–8225. 226 indexed citations

20.

Reed, John R., Milica Vukmanovic‐Stejic, Jean M. Fletcher, et al.. (2004). Telomere Erosion in Memory T Cells Induced by Telomerase Inhibition at the Site of Antigenic Challenge In Vivo. The Journal of Experimental Medicine. 199(10). 1433–1443. 97 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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