Sarah B. Daly

3.3k total citations
20 papers, 1.4k citations indexed

About

Sarah B. Daly is a scholar working on Molecular Biology, Hematology and Public Health, Environmental and Occupational Health. According to data from OpenAlex, Sarah B. Daly has authored 20 papers receiving a total of 1.4k indexed citations (citations by other indexed papers that have themselves been cited), including 13 papers in Molecular Biology, 10 papers in Hematology and 4 papers in Public Health, Environmental and Occupational Health. Recurrent topics in Sarah B. Daly's work include Acute Myeloid Leukemia Research (9 papers), Acute Lymphoblastic Leukemia research (4 papers) and Renal and related cancers (2 papers). Sarah B. Daly is often cited by papers focused on Acute Myeloid Leukemia Research (9 papers), Acute Lymphoblastic Leukemia research (4 papers) and Renal and related cancers (2 papers). Sarah B. Daly collaborates with scholars based in United Kingdom, Italy and United States. Sarah B. Daly's co-authors include Robert K. Hills, Keith Wheatley, Alan K. Burnett, John A. Liu Yin, Jill Urquhart, Sanjeev S. Bhaskar, James O’Sullivan, Graeme C. Black, William G. Newman and Fabienne Hermitte and has published in prestigious journals such as Journal of Clinical Oncology, Blood and Neurology.

In The Last Decade

Sarah B. Daly

20 papers receiving 1.4k citations

Peers

Sarah B. Daly
Rob Dee Netherlands
K Michalová Czechia
A J Carroll United States
Saskia Langemeijer Netherlands
DG Gilliland United States
Sarah B. Daly
Citations per year, relative to Sarah B. Daly Sarah B. Daly (= 1×) peers Takashi Taga

Countries citing papers authored by Sarah B. Daly

Since Specialization
Citations

This map shows the geographic impact of Sarah B. Daly's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Sarah B. Daly with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Sarah B. Daly more than expected).

Fields of papers citing papers by Sarah B. Daly

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Sarah B. Daly. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Sarah B. Daly. The network helps show where Sarah B. Daly may publish in the future.

Co-authorship network of co-authors of Sarah B. Daly

This figure shows the co-authorship network connecting the top 25 collaborators of Sarah B. Daly. A scholar is included among the top collaborators of Sarah B. Daly based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Sarah B. Daly. Sarah B. Daly is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Sharkia, Rajech, Stavit A. Shalev, Nathan Watemberg, et al.. (2017). Homozygous mutation in PTRH2 gene causes progressive sensorineural deafness and peripheral neuropathy. American Journal of Medical Genetics Part A. 173(4). 1051–1055. 9 indexed citations
2.
Ghosh, Arunabha, Jill Urquhart, Sarah B. Daly, et al.. (2017). Phenotypic Heterogeneity in a Congenital Disorder of Glycosylation Caused by Mutations in STT3A. Journal of Child Neurology. 32(6). 560–565. 11 indexed citations
3.
Daly, Sarah B., Hitesh Shah, James O’Sullivan, et al.. (2014). Exome Sequencing Identifies a Dominant <b><i>TNNT3</i></b> Mutation in a Large Family with Distal Arthrogryposis. Molecular Syndromology. 5(5). 218–228. 14 indexed citations
4.
Smith, Miriam J., Christian Beetz, Simon G. Williams, et al.. (2014). Mutations in LZTR1 add to the complex heterogeneity of schwannomatosis. Neurology. 84(2). 141–147. 78 indexed citations
5.
Banka, Siddharth, Stuart A. Cain, Sarah B. Daly, et al.. (2014). Leri’s pleonosteosis, a congenital rheumatic disease, results from microduplication at 8q22.1 encompassing GDF6 and SDC2 and provides insight into systemic sclerosis pathogenesis. Annals of the Rheumatic Diseases. 74(6). 1249–1256. 15 indexed citations
6.
Smith, Miriam J., Christian Beetz, Simon G. Williams, et al.. (2014). Germline Mutations in SUFU Cause Gorlin Syndrome–Associated Childhood Medulloblastoma and Redefine the Risk Associated With PTCH1 Mutations. Journal of Clinical Oncology. 32(36). 4155–4161. 180 indexed citations
8.
Hanson, Dan, Philip Murray, James O’Sullivan, et al.. (2011). Exome Sequencing Identifies CCDC8 Mutations in 3-M Syndrome, Suggesting that CCDC8 Contributes in a Pathway with CUL7 and OBSL1 to Control Human Growth. The American Journal of Human Genetics. 89(1). 148–153. 88 indexed citations
9.
O’Sullivan, James, Carolina Cavalcante Bitu, Sarah B. Daly, et al.. (2011). Whole-Exome Sequencing Identifies FAM20A Mutations as a Cause of Amelogenesis Imperfecta and Gingival Hyperplasia Syndrome. The American Journal of Human Genetics. 88(5). 616–620. 126 indexed citations
10.
O’Driscoll, Mary, Sarah B. Daly, Jill Urquhart, et al.. (2010). Recessive Mutations in the Gene Encoding the Tight Junction Protein Occludin Cause Band-like Calcification with Simplified Gyration and Polymicrogyria. The American Journal of Human Genetics. 87(3). 354–364. 92 indexed citations
11.
Daly, Sarah B., Eibhlin Conneally, & Stephen E. Langabeer. (2009). Identification of <i>MPL</i> W515L/K Mutations in Patients with Primary Myelofibrosis and Essential Thrombocythaemia by Allele-Specific Polymerase Chain Reaction. Acta Haematologica. 121(4). 221–222. 4 indexed citations
12.
Cilloni, Daniela, Aline Renneville, Fabienne Hermitte, et al.. (2009). Real-Time Quantitative Polymerase Chain Reaction Detection of Minimal Residual Disease by Standardized WT1 Assay to Enhance Risk Stratification in Acute Myeloid Leukemia: A European LeukemiaNet Study. Journal of Clinical Oncology. 27(31). 5195–5201. 327 indexed citations
14.
Daly, Sarah B., Michelle Sale, Stuart A. Green, et al.. (2007). Minimal Residual Disease Monitoring by RQ-PCR in Core Binding Factor Positive AML Allows Risk-Stratification and Predicts Relapse: Results of the UK MRC AML-15 Trial.. Blood. 110(11). 543–543. 3 indexed citations
16.
Ramsden, Simon, Sarah B. Daly, Wolf‐Jochen Geilenkeuser, et al.. (2006). EQUAL-quant: An International External Quality Assessment Scheme for Real-Time PCR. Clinical Chemistry. 52(8). 1584–1591. 36 indexed citations
17.
Sweetser, David A., Andrew Peniket, Yuntian Zhang, et al.. (2005). Delineation of the minimal commonly deleted segment and identification of candidate tumor‐suppressor genes in del(9q) acute myeloid leukemia. Genes Chromosomes and Cancer. 44(3). 279–291. 45 indexed citations
18.
Peniket, Andrew, James S. Wainscoat, Lucy Side, et al.. (2005). Del (9q) AML: clinical and cytological characteristics and prognostic implications. British Journal of Haematology. 129(2). 210–220. 29 indexed citations
19.
Gottardi, Enrico, Daniela Cilloni, Sarah B. Daly, et al.. (2005). Standardization of WT1 mRNA Quantification for Minimal Residual Disease (MRD) Monitoring in Acute Leukemia Patients: A European LeukemiaNet Concerted Action.. Blood. 106(11). 3295–3295. 2 indexed citations
20.
Daly, Sarah B., J. S. Wainscoat, Elaine R. Levy, et al.. (2001). Physical and transcriptional characterization of the commonly deleted region in del(9q) AML.. Blood. 98. 1 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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