Sara Ortiz-Vega

2.9k total citations · 1 hit paper
14 papers, 2.4k citations indexed

About

Sara Ortiz-Vega is a scholar working on Molecular Biology, Cell Biology and Pharmacology. According to data from OpenAlex, Sara Ortiz-Vega has authored 14 papers receiving a total of 2.4k indexed citations (citations by other indexed papers that have themselves been cited), including 13 papers in Molecular Biology, 3 papers in Cell Biology and 2 papers in Pharmacology. Recurrent topics in Sara Ortiz-Vega's work include PI3K/AKT/mTOR signaling in cancer (9 papers), Protein Kinase Regulation and GTPase Signaling (6 papers) and Polyamine Metabolism and Applications (4 papers). Sara Ortiz-Vega is often cited by papers focused on PI3K/AKT/mTOR signaling in cancer (9 papers), Protein Kinase Regulation and GTPase Signaling (6 papers) and Polyamine Metabolism and Applications (4 papers). Sara Ortiz-Vega collaborates with scholars based in United States, Japan and Taiwan. Sara Ortiz-Vega's co-authors include Joseph Avruch, Samuel Long, Yenshou Lin, Kazuyoshi Yonezawa, Maria Praskova, Joseph Rapley, Angela Papageorgiou, Ning Dai, Xianfeng Zhang and Kenta Hara and has published in prestigious journals such as Journal of Biological Chemistry, Oncogene and Current Biology.

In The Last Decade

Sara Ortiz-Vega

14 papers receiving 2.3k citations

Hit Papers

Rheb Binds and Regulates the mTOR Kinase 2005 2026 2012 2019 2005 250 500 750
What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

  1. Rheb Binds and Regulates the mTOR Kinase
    2005 771 citations Samuel Long, Yenshou Lin et al. Current Biology profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Sara Ortiz-Vega United States 13 1.9k 691 253 238 193 14 2.4k
Stephen G. Dann United States 16 1.8k 1.0× 410 0.6× 434 1.7× 322 1.4× 263 1.4× 20 2.6k
Christopher Belham United Kingdom 15 2.3k 1.2× 629 0.9× 359 1.4× 163 0.7× 309 1.6× 16 3.0k
Xiaodong Shu China 20 1.8k 0.9× 658 1.0× 178 0.7× 163 0.7× 349 1.8× 40 2.5k
Karolina Pakos‐Zebrucka Ireland 4 1.2k 0.7× 657 1.0× 209 0.8× 300 1.3× 182 0.9× 4 1.8k
Laura R. Pearce United Kingdom 8 1.6k 0.9× 309 0.4× 190 0.8× 146 0.6× 162 0.8× 9 2.1k
Jeroen van der Kaay United Kingdom 18 2.0k 1.1× 534 0.8× 201 0.8× 86 0.4× 250 1.3× 26 2.6k
Clark D. Wells United States 25 1.7k 0.9× 910 1.3× 122 0.5× 189 0.8× 222 1.2× 33 2.4k
Hiroyuki Okamoto Japan 28 2.5k 1.3× 738 1.1× 402 1.6× 96 0.4× 248 1.3× 55 3.4k
Eileen L. Whiteman United States 15 1.1k 0.6× 443 0.6× 328 1.3× 261 1.1× 115 0.6× 17 1.8k
David Frith United Kingdom 19 1.2k 0.6× 484 0.7× 174 0.7× 515 2.2× 161 0.8× 28 2.0k

Countries citing papers authored by Sara Ortiz-Vega

Since Specialization
Citations

This map shows the geographic impact of Sara Ortiz-Vega's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Sara Ortiz-Vega with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Sara Ortiz-Vega more than expected).

Fields of papers citing papers by Sara Ortiz-Vega

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Sara Ortiz-Vega. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Sara Ortiz-Vega. The network helps show where Sara Ortiz-Vega may publish in the future.

Co-authorship network of co-authors of Sara Ortiz-Vega

This figure shows the co-authorship network connecting the top 25 collaborators of Sara Ortiz-Vega. A scholar is included among the top collaborators of Sara Ortiz-Vega based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Sara Ortiz-Vega. Sara Ortiz-Vega is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

14 of 14 papers shown
1.
Yao, Chien‐An, et al.. (2017). Association of mSin1 with mTORC2 Ras and Akt reveals a crucial domain on mSin1 involved in Akt phosphorylation. Oncotarget. 8(38). 63392–63404. 20 indexed citations
2.
Rapley, Joseph, Noriko Oshiro, Sara Ortiz-Vega, & Joseph Avruch. (2011). The Mechanism of Insulin-stimulated 4E-BP Protein Binding to Mammalian Target of Rapamycin (mTOR) Complex 1 and Its Contribution to mTOR Complex 1 Signaling. Journal of Biological Chemistry. 286(44). 38043–38053. 35 indexed citations
3.
Avruch, Joseph, Samuel Long, Yenshou Lin, et al.. (2009). Activation of mTORC1 in two steps: Rheb-GTP activation of catalytic function and increased binding of substrates to raptor1. Biochemical Society Transactions. 37(1). 223–226. 58 indexed citations
4.
Avruch, Joseph, Samuel Long, Sara Ortiz-Vega, et al.. (2008). Amino acid regulation of TOR complex 1. American Journal of Physiology-Endocrinology and Metabolism. 296(4). E592–E602. 302 indexed citations
5.
Long, Samuel, Yenshou Lin, Sara Ortiz-Vega, Susann Busch, & Joseph Avruch. (2007). The Rheb Switch 2 Segment Is Critical for Signaling to Target of Rapamycin Complex 1. Journal of Biological Chemistry. 282(25). 18542–18551. 34 indexed citations
6.
Avruch, Joseph, Maria Praskova, Sara Ortiz-Vega, Matthew Liu, & Xianfeng Zhang. (2006). Nore1 and RASSF1 Regulation of Cell Proliferation and of the MST1/2 Kinases. Methods in enzymology on CD-ROM/Methods in enzymology. 407. 290–310. 69 indexed citations
7.
Avruch, Joseph, Kenta Hara, Yining Lin, et al.. (2006). Insulin and amino-acid regulation of mTOR signaling and kinase activity through the Rheb GTPase. Oncogene. 25(48). 6361–6372. 258 indexed citations
8.
Long, Samuel, Sara Ortiz-Vega, Yenshou Lin, & Joseph Avruch. (2005). Rheb Binding to Mammalian Target of Rapamycin (mTOR) Is Regulated by Amino Acid Sufficiency. Journal of Biological Chemistry. 280(25). 23433–23436. 271 indexed citations
9.
Long, Samuel, Yenshou Lin, Sara Ortiz-Vega, Kazuyoshi Yonezawa, & Joseph Avruch. (2005). Rheb Binds and Regulates the mTOR Kinase. Current Biology. 15(8). 702–713. 771 indexed citations breakdown →
10.
Avruch, Joseph, Yenshou Lin, Samuel Long, Sid Murthy, & Sara Ortiz-Vega. (2004). Recent advances in the regulation of the TOR pathway by insulin and nutrients. Current Opinion in Clinical Nutrition & Metabolic Care. 8(1). 67–72. 79 indexed citations
11.
Praskova, Maria, et al.. (2004). Regulation of the MST1 kinase by autophosphorylation, by the growth inhibitory proteins, RASSF1 and NORE1, and by Ras. Biochemical Journal. 381(2). 453–462. 288 indexed citations
12.
Ortiz-Vega, Sara, Andrei Khokhlatchev, Xianfeng Zhang, et al.. (2002). The putative tumor suppressor RASSF1A homodimerizes and heterodimerizes with the Ras-GTP binding protein Nore1. Oncogene. 21(9). 1381–1390. 185 indexed citations
13.
Ortiz-Vega, Sara & Barrie Ashby. (1997). Human Prostacyclin Receptor: Cloning and Co-Expression with EP3 Prostaglandin Receptor. Advances in experimental medicine and biology. 433. 235–238. 1 indexed citations
14.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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