Sandrine Prost

1.1k total citations
25 papers, 816 citations indexed

About

Sandrine Prost is a scholar working on Molecular Biology, Oncology and Hepatology. According to data from OpenAlex, Sandrine Prost has authored 25 papers receiving a total of 816 indexed citations (citations by other indexed papers that have themselves been cited), including 17 papers in Molecular Biology, 14 papers in Oncology and 4 papers in Hepatology. Recurrent topics in Sandrine Prost's work include Cancer-related Molecular Pathways (10 papers), Cancer therapeutics and mechanisms (5 papers) and DNA Repair Mechanisms (3 papers). Sandrine Prost is often cited by papers focused on Cancer-related Molecular Pathways (10 papers), Cancer therapeutics and mechanisms (5 papers) and DNA Repair Mechanisms (3 papers). Sandrine Prost collaborates with scholars based in United Kingdom, France and United States. Sandrine Prost's co-authors include David J. Harrison, Christopher Bellamy, G Riou, Helen Caldwell, Michel Barrois, Pin Lü, Andrew H. Wyllie, Christine Théodore, Arnold J. Levine and A J Demetris and has published in prestigious journals such as Nucleic Acids Research, Journal of Biological Chemistry and The Journal of Experimental Medicine.

In The Last Decade

Sandrine Prost

24 papers receiving 806 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Sandrine Prost United Kingdom 16 458 312 149 147 125 25 816
Hien Dang United States 15 479 1.0× 376 1.2× 107 0.7× 119 0.8× 373 3.0× 24 1.0k
Tetsuhisa Yamamoto Japan 15 577 1.3× 519 1.7× 226 1.5× 102 0.7× 106 0.8× 45 1.1k
Wilma Jogunoori United States 13 498 1.1× 390 1.3× 87 0.6× 99 0.7× 197 1.6× 22 871
Mitsukazu Gotoh Japan 18 365 0.8× 300 1.0× 187 1.3× 150 1.0× 83 0.7× 30 820
Insa S. Schroeder Germany 13 536 1.2× 114 0.4× 218 1.5× 32 0.2× 117 0.9× 33 829
Jonathan F. Anker United States 14 617 1.3× 519 1.7× 61 0.4× 233 1.6× 14 0.1× 31 1.1k
Taiji Yamazoe Japan 13 546 1.2× 437 1.4× 140 0.9× 125 0.9× 92 0.7× 26 1.0k
Kesen Xu China 17 271 0.6× 243 0.8× 128 0.9× 103 0.7× 60 0.5× 32 675
Herwig P. Moll Austria 14 323 0.7× 200 0.6× 44 0.3× 180 1.2× 18 0.1× 32 633

Countries citing papers authored by Sandrine Prost

Since Specialization
Citations

This map shows the geographic impact of Sandrine Prost's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Sandrine Prost with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Sandrine Prost more than expected).

Fields of papers citing papers by Sandrine Prost

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Sandrine Prost. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Sandrine Prost. The network helps show where Sandrine Prost may publish in the future.

Co-authorship network of co-authors of Sandrine Prost

This figure shows the co-authorship network connecting the top 25 collaborators of Sandrine Prost. A scholar is included among the top collaborators of Sandrine Prost based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Sandrine Prost. Sandrine Prost is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
O’Connor, Richard A., Lilian Koppensteiner, Irene Young, et al.. (2021). T cells drive negative feedback mechanisms in cancer associated fibroblasts, promoting expression of co-inhibitory ligands, CD73 and IL-27 in non-small cell lung cancer. OncoImmunology. 10(1). 1940675–1940675. 36 indexed citations
2.
Ma, Ruoyu, Hui Zhang, Xuefeng Li, et al.. (2020). Monocyte-derived macrophages promote breast cancer bone metastasis outgrowth. The Journal of Experimental Medicine. 217(11). 115 indexed citations
3.
Prost, Sandrine, et al.. (2016). Working with Commercially Available Quantum Dots for Immunofluorescence on Tissue Sections. PLoS ONE. 11(9). e0163856–e0163856. 12 indexed citations
4.
Prost, Sandrine, et al.. (2016). Choice of Illumination System & Fluorophore for Multiplex Immunofluorescence on FFPE Tissue Sections. PLoS ONE. 11(9). e0162419–e0162419. 8 indexed citations
5.
Bellamy, Christopher, et al.. (2012). The value of immunophenotyping hepatocellular adenomas: consecutive resections at one UK centre. Histopathology. 62(3). 431–445. 24 indexed citations
6.
Bellamy, Christopher, et al.. (2008). TGFbeta induces apoptosis and EMT in primary mouse hepatocytes independently of p53, p21 Cip1 or Rbstatus. BMC Cancer. 8(1). 191–191. 15 indexed citations
7.
Lü, Pin, Sandrine Prost, Helen Caldwell, et al.. (2007). Microarray analysis of gene expression of mouse hepatocytes of different ploidy. Mammalian Genome. 18(9). 617–626. 56 indexed citations
8.
Bellamy, Christopher, et al.. (2007). Deficiency of G1 regulators P53, P21Cip1and/or pRb decreases hepatocyte sensitivity to TGFβ cell cycle arrest. BMC Cancer. 7(1). 215–215. 22 indexed citations
9.
Kirschner, Kristina, Rajinder Singh, Sandrine Prost, & David W. Melton. (2006). Characterisation of Ercc1 deficiency in the liver and in conditional Ercc1-deficient primary hepatocytes in vitro. DNA repair. 6(3). 304–316. 14 indexed citations
10.
Prost, Sandrine, Pin Lü, Helen Caldwell, & David J. Harrison. (2006). E2F regulates DDB2: consequences for DNA repair in Rb-deficient cells. Oncogene. 26(24). 3572–3581. 27 indexed citations
11.
Harrison, David J., et al.. (2003). Additive effect of p53, p21 and Rb deletion in triple knockout primary hepatocytes. Oncogene. 23(8). 1489–1497. 32 indexed citations
12.
Wheelhouse, Nick, et al.. (2003). TNF-α induced DNA damage in primary murine hepatocytes. International Journal of Molecular Medicine. 12(6). 889–94. 58 indexed citations
13.
Prost, Sandrine. (2001). Adenovirus-mediated Cre deletion of floxed sequences in primary mouse cells is an efficient alternative for studies of gene deletion. Nucleic Acids Research. 29(16). 80e–80. 28 indexed citations
14.
Prost, Sandrine, et al.. (1998). Hepatitis B x Protein Inhibits p53-dependent DNA Repair in Primary Mouse Hepatocytes. Journal of Biological Chemistry. 273(50). 33327–33332. 1 indexed citations
15.
Prost, Sandrine, Christopher Bellamy, Dean S. Cunningham, & David J. Harrison. (1998). Altered DNA repair and dysregulation of p53 in IRF‐1 null hepatocytes. The FASEB Journal. 12(2). 181–188. 29 indexed citations
16.
Bellamy, Christopher, Sandrine Prost, Andrew H. Wyllie, & David J. Harrison. (1997). UV but not γ-irradiation induces specific transcriptional activity of p53 in primary hepatocytes. The Journal of Pathology. 183(2). 177–181. 26 indexed citations
17.
Riou, G, et al.. (1995). The p53 and mdm‐2 genes in human testicular germ‐cell tumors. Molecular Carcinogenesis. 12(3). 124–131. 83 indexed citations
18.
Prost, Sandrine. (1995). Mechanisms of resistance to topoisomerases poisons. General Pharmacology The Vascular System. 26(8). 1773–1784. 14 indexed citations
20.
Madelaine, Isabelle, et al.. (1993). Sequential modifications of topoisomerase I activity in a camptothecin-resistant cell line established by progressive adaptation. Biochemical Pharmacology. 45(2). 339–348. 56 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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