Sanami Takada

980 total citations
26 papers, 640 citations indexed

About

Sanami Takada is a scholar working on Molecular Biology, Epidemiology and Hepatology. According to data from OpenAlex, Sanami Takada has authored 26 papers receiving a total of 640 indexed citations (citations by other indexed papers that have themselves been cited), including 13 papers in Molecular Biology, 11 papers in Epidemiology and 6 papers in Hepatology. Recurrent topics in Sanami Takada's work include Hepatitis B Virus Studies (8 papers), DNA Repair Mechanisms (6 papers) and Hepatitis C virus research (5 papers). Sanami Takada is often cited by papers focused on Hepatitis B Virus Studies (8 papers), DNA Repair Mechanisms (6 papers) and Hepatitis C virus research (5 papers). Sanami Takada collaborates with scholars based in Japan, Netherlands and Chile. Sanami Takada's co-authors include Katsuro Koike, Masayuki Arii, Yasuhiro Gotoh, Shigenobu Hayashi, Motonobu Yoshida, Katsuyuki Yaginuma, Ikuo Nakamura, Takahiro Mori, Hiroshi Kido and A Fukutomi and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Virology and Annals of the New York Academy of Sciences.

In The Last Decade

Sanami Takada

25 papers receiving 635 citations

Peers

Sanami Takada
Edward S. Dornan United Kingdom
J S Twu United States
C. Van Sant United States
L P Ting China
Edward S. Dornan United Kingdom
Sanami Takada
Citations per year, relative to Sanami Takada Sanami Takada (= 1×) peers Edward S. Dornan

Countries citing papers authored by Sanami Takada

Since Specialization
Citations

This map shows the geographic impact of Sanami Takada's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Sanami Takada with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Sanami Takada more than expected).

Fields of papers citing papers by Sanami Takada

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Sanami Takada. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Sanami Takada. The network helps show where Sanami Takada may publish in the future.

Co-authorship network of co-authors of Sanami Takada

This figure shows the co-authorship network connecting the top 25 collaborators of Sanami Takada. A scholar is included among the top collaborators of Sanami Takada based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Sanami Takada. Sanami Takada is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Venegas, Viviana, et al.. (2025). A Unique Case of MBD5 and CCM2 Deletions Leading to a Severe Neurological Phenotype With Prolonged Status Epilepticus. Clinical Genetics. 107(6). 663–667. 1 indexed citations
2.
Takada, Sanami, Hiroki Tanaka, Joaquı́n Zúñiga, et al.. (2024). A Novel AGR2 Variant Causing Aberrant Monomer-Dimer Equilibrium Leading to Severe Respiratory and Digestive Symptoms. Journal of Clinical Immunology. 45(1). 55–55. 1 indexed citations
3.
Takada, Sanami, Nienke J.H. van Os, Likun Du, et al.. (2024). Causative mechanisms and clinical impact of immunoglobulin deficiencies in ataxia telangiectasia. Journal of Allergy and Clinical Immunology. 153(5). 1392–1405. 4 indexed citations
4.
Takada, Sanami, Ghada M. H. Abdel‐Salam, Mohamed S. Abdel‐Hamid, et al.. (2024). Biallelic loss-of-function variants in GON4L cause microcephaly and brain structure abnormalities. npj Genomic Medicine. 9(1). 55–55.
5.
Takada, Sanami, et al.. (2023). Human phenotype caused by biallelic KDM4B frameshift variant. Clinical Genetics. 105(1). 72–76. 2 indexed citations
6.
Hitomi, Yuki, Kazuko Ueno, Yoshihiro Aiba, et al.. (2023). rs10924104 in the expression enhancer motif of CD58 confers susceptibility to human autoimmune diseases. Human Genetics. 143(1). 19–33. 2 indexed citations
7.
Hitomi, Yuki, Yoshihiro Aiba, Kazuko Ueno, et al.. (2022). rs2013278 in the multiple immunological-trait susceptibility locus CD28 regulates the production of non-functional splicing isoforms. Human Genomics. 16(1). 46–46. 8 indexed citations
8.
Takada, Sanami, et al.. (2021). ATM: Translating the DNA Damage Response to Adaptive Immunity. Trends in Immunology. 42(4). 350–365. 34 indexed citations
9.
Takada, Sanami, Tessa van Dijk, Merel W. Boogaard, et al.. (2021). Primary Ovarian Failure in Addition to Classical Clinical Features of Coats Plus Syndrome in a Female Carrying 2 Truncating Variants of CTC1. Hormone Research in Paediatrics. 94(11-12). 448–455. 3 indexed citations
10.
Takada, Sanami, Marcel van Deuren, Charlotte J. Dommering, et al.. (2021). Considerations for radiotherapy in Bloom Syndrome: A case series. European Journal of Medical Genetics. 64(10). 104293–104293. 3 indexed citations
11.
Takada, Sanami, Megumu K. Saito, & Naotomo Kambe. (2020). Blau Syndrome: NOD2-related systemic autoinflammatory granulomatosis. Giornale Italiano di Dermatologia e Venereologia. 155(5). 537–541. 9 indexed citations
12.
Takada, Sanami, Erik‐Jan Kamsteeg, Sjoert Pegge, et al.. (2020). The Phenotypic Spectrum of PNKP-Associated Disease and the Absence of Immunodeficiency and Cancer Predisposition in a Dutch Cohort. Pediatric Neurology. 113. 26–32. 6 indexed citations
13.
Takada, Sanami, Naotomo Kambe, Yuri Kawasaki, et al.. (2017). Pluripotent stem cell models of Blau syndrome reveal an IFN-γ–dependent inflammatory response in macrophages. Journal of Allergy and Clinical Immunology. 141(1). 339–349.e11. 41 indexed citations
14.
Ishida, Susumu, Koichi Shudo, Sanami Takada, & Katsuro Koike. (1995). A direct role of transcription factor E2F in c-myc gene expression during granulocytic and macrophage-like differentiation of HL60 cells.. PubMed. 6(3). 229–37. 18 indexed citations
15.
Koike, Katsuro, Toshiko Hara, Yukihiko Aramaki, Sanami Takada, & Satoshi Tsuchiya. (1994). Receptor‐Mediated Gene Transfer into Hepatic Cells Using Asialoglycoprotein‐Labeled Liposomes. Annals of the New York Academy of Sciences. 716(1). 331–333. 8 indexed citations
16.
Takada, Sanami, Hiroshi Kido, A Fukutomi, Takahiro Mori, & K Koike. (1994). Interaction of hepatitis B virus X protein with a serine protease, tryptase TL2 as an inhibitor.. PubMed. 9(2). 341–8. 44 indexed citations
17.
Yaginuma, Katsuyuki, Ikuo Nakamura, Sanami Takada, & Katsuro Koike. (1993). A transcription initiation site for the hepatitis B virus X gene is directed by the promoter-binding protein. Journal of Virology. 67(5). 2559–2565. 17 indexed citations
18.
Takada, Sanami. (1990). Trans-activation function of a 3' truncated X gene-cell fusion product from integrated HBV DNA in chronic hepatitis tissues. Proc Natl Acad Sci USA. 87. 5628–5632. 53 indexed citations
19.
Takada, Sanami, Yasuhiro Gotoh, Shigenobu Hayashi, Motonobu Yoshida, & Katsuro Koike. (1990). Structural rearrangement of integrated hepatitis B virus DNA as well as cellular flanking DNA is present in chronically infected hepatic tissues. Journal of Virology. 64(2). 822–828. 70 indexed citations
20.
Koike, Katsuro, Yumiko Shirakata, Katsuyuki Yaginuma, et al.. (1983). Oncogenic potential of hepatitis B virus.. PubMed. 4(1). 1–2. 54 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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