Sam Wormald

1.6k total citations · 1 hit paper
11 papers, 1.2k citations indexed

About

Sam Wormald is a scholar working on Molecular Biology, Oncology and Endocrine and Autonomic Systems. According to data from OpenAlex, Sam Wormald has authored 11 papers receiving a total of 1.2k indexed citations (citations by other indexed papers that have themselves been cited), including 5 papers in Molecular Biology, 4 papers in Oncology and 2 papers in Endocrine and Autonomic Systems. Recurrent topics in Sam Wormald's work include Cytokine Signaling Pathways and Interactions (4 papers), Pluripotent Stem Cells Research (3 papers) and CRISPR and Genetic Engineering (3 papers). Sam Wormald is often cited by papers focused on Cytokine Signaling Pathways and Interactions (4 papers), Pluripotent Stem Cells Research (3 papers) and CRISPR and Genetic Engineering (3 papers). Sam Wormald collaborates with scholars based in Australia, United Kingdom and Netherlands. Sam Wormald's co-authors include Douglas J. Hilton, Warren S. Alexander, Donald Metcalf, Ben A. Croker, Andrew W. Roberts, Christopher J. Greenhalgh, Lorraine Robb, Jian‐Guo Zhang, Danielle L. Krebs and Nicos A. Nicola and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Nucleic Acids Research and Blood.

In The Last Decade

Sam Wormald

11 papers receiving 1.1k citations

Hit Papers

SOCS3 negatively regulates IL-6 signaling in vivo 2003 2026 2010 2018 2003 200 400 600

Peers

Sam Wormald
Sam Wormald
Citations per year, relative to Sam Wormald Sam Wormald (= 1×) peers Fabrizio Antonangeli

Countries citing papers authored by Sam Wormald

Since Specialization
Citations

This map shows the geographic impact of Sam Wormald's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Sam Wormald with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Sam Wormald more than expected).

Fields of papers citing papers by Sam Wormald

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Sam Wormald. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Sam Wormald. The network helps show where Sam Wormald may publish in the future.

Co-authorship network of co-authors of Sam Wormald

This figure shows the co-authorship network connecting the top 25 collaborators of Sam Wormald. A scholar is included among the top collaborators of Sam Wormald based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Sam Wormald. Sam Wormald is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

11 of 11 papers shown
1.
McWilliam, Hamish E. G., Jeffrey Y. W. Mak, Wael Awad, et al.. (2020). Endoplasmic reticulum chaperones stabilize ligand-receptive MR1 molecules for efficient presentation of metabolite antigens. Proceedings of the National Academy of Sciences. 117(40). 24974–24985. 35 indexed citations
2.
Fisher, Cynthia L., Hendrik Marks, Robert Andrews, et al.. (2017). An efficient method for generation of bi-allelic null mutant mouse embryonic stem cells and its application for investigating epigenetic modifiers. Nucleic Acids Research. 45(21). e174–e174. 46 indexed citations
3.
Babon, Jeffrey J., John Wagner, Jonathan H. Manton, et al.. (2017). TGF-β and IL-6 family signalling crosstalk: an integrated model. Growth Factors. 35(2-3). 100–124. 7 indexed citations
4.
Coelho, Paula A., Leah Bury, Marta N. Shahbazi, et al.. (2015). Over-expression of Plk4 induces centrosome amplification, loss of primary cilia and associated tissue hyperplasia in the mouse. Open Biology. 5(12). 150209–150209. 122 indexed citations
5.
Melidoni, Anna N., Michael R. Dyson, Sam Wormald, & John McCafferty. (2013). Selecting antagonistic antibodies that control differentiation through inducible expression in embryonic stem cells. Proceedings of the National Academy of Sciences. 110(44). 17802–17807. 15 indexed citations
6.
Sachithanandan, Nirupa, Barbara C. Fam, Stacey Fynch, et al.. (2010). Liver-Specific Suppressor of Cytokine Signaling-3 Deletion in Mice Enhances Hepatic Insulin Sensitivity and Lipogenesis Resulting in Fatty Liver and Obesity. Hepatology. 52(5). 1632–1642. 90 indexed citations
7.
Croker, Ben A., Lisa A. Mielke, Sam Wormald, et al.. (2008). Socs3 maintains the specificity of biological responses to cytokine signals during granulocyte and macrophage differentiation. Experimental Hematology. 36(7). 786–798. 23 indexed citations
8.
Laslett, Andrew L., Sean M. Grimmond, Brooke Gardiner, et al.. (2007). Transcriptional analysis of early lineage commitment in human embryonic stem cells. BMC Developmental Biology. 7(1). 12–12. 70 indexed citations
10.
Steinberg, Gregory R., et al.. (2004). Endurance training partially reverses dietary-induced leptin resistance in rodent skeletal muscle. American Journal of Physiology-Endocrinology and Metabolism. 286(1). E57–E63. 66 indexed citations
11.
Croker, Ben A., Danielle L. Krebs, Jian‐Guo Zhang, et al.. (2003). SOCS3 negatively regulates IL-6 signaling in vivo. Nature Immunology. 4(6). 540–545. 684 indexed citations breakdown →

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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