Safak Yalcin

1.4k total citations
19 papers, 1.1k citations indexed

About

Safak Yalcin is a scholar working on Molecular Biology, Immunology and Oncology. According to data from OpenAlex, Safak Yalcin has authored 19 papers receiving a total of 1.1k indexed citations (citations by other indexed papers that have themselves been cited), including 14 papers in Molecular Biology, 5 papers in Immunology and 4 papers in Oncology. Recurrent topics in Safak Yalcin's work include FOXO transcription factor regulation (9 papers), Epigenetics and DNA Methylation (2 papers) and Genetics, Aging, and Longevity in Model Organisms (2 papers). Safak Yalcin is often cited by papers focused on FOXO transcription factor regulation (9 papers), Epigenetics and DNA Methylation (2 papers) and Genetics, Aging, and Longevity in Model Organisms (2 papers). Safak Yalcin collaborates with scholars based in United States, Singapore and Colombia. Safak Yalcin's co-authors include Saghi Ghaffari, Xin Zhang, Dragan Marinković, Sathish Kumar Mungamuri, Carlo Brugnara, Tara L. Huber, Rani S. Sellers, Reshma Taneja, Cécile Vercherat and Pauline Rimmelé and has published in prestigious journals such as Journal of Biological Chemistry, Journal of Clinical Investigation and The EMBO Journal.

In The Last Decade

Safak Yalcin

19 papers receiving 1.1k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Safak Yalcin United States 13 722 181 163 159 140 19 1.1k
Pauline Rimmelé United States 13 603 0.8× 189 1.0× 201 1.2× 129 0.8× 80 0.6× 23 882
Mondira Kundu United States 17 756 1.0× 147 0.8× 203 1.2× 117 0.7× 252 1.8× 28 1.3k
Jozef Madžo United States 20 1.0k 1.4× 141 0.8× 222 1.4× 201 1.3× 213 1.5× 52 1.5k
Joshua A. Regal United States 8 349 0.5× 432 2.4× 130 0.8× 82 0.5× 78 0.6× 17 831
Lorenzo Brunetti Italy 19 1.1k 1.5× 116 0.6× 727 4.5× 119 0.7× 168 1.2× 39 1.6k
Hsiang‐Ying Lee Taiwan 18 728 1.0× 129 0.7× 210 1.3× 110 0.7× 137 1.0× 33 1.1k
Karin E. M. Diderich Netherlands 21 822 1.1× 113 0.6× 18 0.1× 142 0.9× 70 0.5× 60 1.6k
Rebecca Hoffman United States 20 682 0.9× 391 2.2× 67 0.4× 78 0.5× 112 0.8× 28 1.3k
Katerina I. Leonova United States 10 672 0.9× 443 2.4× 23 0.1× 133 0.8× 132 0.9× 13 1.3k
Liguang Sun China 18 356 0.5× 84 0.5× 77 0.5× 60 0.4× 104 0.7× 54 1.1k

Countries citing papers authored by Safak Yalcin

Since Specialization
Citations

This map shows the geographic impact of Safak Yalcin's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Safak Yalcin with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Safak Yalcin more than expected).

Fields of papers citing papers by Safak Yalcin

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Safak Yalcin. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Safak Yalcin. The network helps show where Safak Yalcin may publish in the future.

Co-authorship network of co-authors of Safak Yalcin

This figure shows the co-authorship network connecting the top 25 collaborators of Safak Yalcin. A scholar is included among the top collaborators of Safak Yalcin based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Safak Yalcin. Safak Yalcin is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

19 of 19 papers shown
1.
Henry, Cathérine, Naveen K. Mehta, Kavya Rakhra, et al.. (2025). CLN-619, a MICA/B monoclonal antibody that promotes innate immune cell-mediated antitumor activity. Journal for ImmunoTherapy of Cancer. 13(4). e008987–e008987. 2 indexed citations
2.
Mehta, Naveen K., Safak Yalcin, Kristan Meetze, et al.. (2022). Abstract 3506: CLN-619, a clinical-stage MICA/MICB-specific IgG1 antibody, restores the MICA/MICB-NKG2D axis to promote NK-mediated tumor cell lysis. Cancer Research. 82(12_Supplement). 3506–3506. 1 indexed citations
3.
Sharp, Leslie L., Cathy Chang, Gerhard Frey, et al.. (2018). Abstract 827: Anti-tumor efficacy of BA3011, a novel Conditionally Active Biologic (CAB) anti-AXL-ADC. Cancer Research. 78(13_Supplement). 827–827. 16 indexed citations
4.
Sharp, Leslie L., Cathy Chang, Gerhard Frey, et al.. (2018). Abstract 833: Anti-tumor efficacy of BA3021, a novel Conditionally Active Biologic (CAB) anti-ROR2 ADC. Cancer Research. 78(13_Supplement). 833–833. 15 indexed citations
5.
Liang, Raymond, Pauline Rimmelé, Carolina L. Bigarella, Safak Yalcin, & Saghi Ghaffari. (2016). Evidence for AKT-independent regulation of FOXO1 and FOXO3 in haematopoietic stem and progenitor cells. Cell Cycle. 15(6). 861–867. 30 indexed citations
6.
Zhang, Xin, Genís Campreciós, Pauline Rimmelé, et al.. (2014). FOXO3‐mTOR metabolic cooperation in the regulation of erythroid cell maturation and homeostasis. American Journal of Hematology. 89(10). 954–963. 71 indexed citations
7.
Yalcin, Safak, Mark Carty, Joseph Shin, et al.. (2014). Microrna Mediated Regulation of Hematopoietic Stem Cell Aging. Blood. 124(21). 602–602. 2 indexed citations
8.
Park, Chris, Joseph Shin, Safak Yalcin, & Wenhuo Hu. (2013). Low c-Kit expression identifies hematopoietic stem cells with enhanced self-renewal potential. Experimental Hematology. 41(8). S43–S43. 1 indexed citations
9.
Zhang, Xin, et al.. (2011). Regulation and Function of FoxO Transcription Factors in Normal and Cancer Stem Cells: What Have We Learned?. Current Drug Targets. 12(9). 1267–1283. 35 indexed citations
10.
Zhang, Xin, Safak Yalcin, Dung‐Fang Lee, et al.. (2011). FOXO1 is an essential regulator of pluripotency in human embryonic stem cells. Nature Cell Biology. 13(9). 1092–1099. 212 indexed citations
11.
Grzmil, Paweł, et al.. (2010). Early embryonic lethality in gene trap mice with disruption of the Arfgef2 gene. The International Journal of Developmental Biology. 54(8-9). 1259–1266. 15 indexed citations
12.
Yalcin, Safak, Dragan Marinković, Sathish Kumar Mungamuri, et al.. (2010). ROS‐mediated amplification of AKT/mTOR signalling pathway leads to myeloproliferative syndrome in Foxo3−/− mice. The EMBO Journal. 29(24). 4118–4131. 126 indexed citations
13.
Vercherat, Cécile, et al.. (2009). Stra13 regulates oxidative stress mediated skeletal muscle degeneration. Human Molecular Genetics. 18(22). 4304–4316. 33 indexed citations
14.
Doğruman‐Al, Funda, et al.. (2009). A possible relationship betweenToxoplasma gondiiand schizophrenia: A seroprevalence study. International Journal of Psychiatry in Clinical Practice. 13(1). 82–87. 18 indexed citations
15.
Yalcin, Safak, Sathish Kumar Mungamuri, Dragan Marinković, et al.. (2008). Oxidative Stress-Mediated Activation of AKT/mTOR Signaling Pathway Leads to Myeloproliferative Syndrome in FoxO3 Null Mice: A Role for Lnk Adaptor Protein. Blood. 112(11). 509–509. 2 indexed citations
16.
Yalcin, Safak, Xin Zhang, Sathish Kumar Mungamuri, et al.. (2008). Foxo3 Is Essential for the Regulation of Ataxia Telangiectasia Mutated and Oxidative Stress-mediated Homeostasis of Hematopoietic Stem Cells. Journal of Biological Chemistry. 283(37). 25692–25705. 206 indexed citations
17.
Marinković, Dragan, Xin Zhang, Safak Yalcin, et al.. (2007). Foxo3 is required for the regulation of oxidative stress in erythropoiesis. Journal of Clinical Investigation. 117(8). 2133–2144. 243 indexed citations
18.
19.
Yalcin, Safak, Hirohiko Kuratsune, K. Yamaguchi, Teruo Kitani, & Koichi Yamanishi. (1994). Prevalence of Human Herpesvirus 6 Variants A and B in Patients with Chronic Fatigue Syndrome. Microbiology and Immunology. 38(7). 587–590. 43 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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