Saeed Solali

1.2k total citations
41 papers, 896 citations indexed

About

Saeed Solali is a scholar working on Molecular Biology, Cancer Research and Genetics. According to data from OpenAlex, Saeed Solali has authored 41 papers receiving a total of 896 indexed citations (citations by other indexed papers that have themselves been cited), including 33 papers in Molecular Biology, 19 papers in Cancer Research and 7 papers in Genetics. Recurrent topics in Saeed Solali's work include Epigenetics and DNA Methylation (12 papers), MicroRNA in disease regulation (10 papers) and Cell death mechanisms and regulation (8 papers). Saeed Solali is often cited by papers focused on Epigenetics and DNA Methylation (12 papers), MicroRNA in disease regulation (10 papers) and Cell death mechanisms and regulation (8 papers). Saeed Solali collaborates with scholars based in Iran, Denmark and Iraq. Saeed Solali's co-authors include Majid Farshdousti Hagh, Mohammad Reza Alivand, Ali Hassanzadeh, Faroogh Marofi, Maryam Zare, Milad Bastami, Behzad Baradaran, Mehdi Talebi, Behzad Mansoori and Karim Shamsasenjan and has published in prestigious journals such as SHILAP Revista de lepidopterología, Biochemical and Biophysical Research Communications and Journal of Cellular Physiology.

In The Last Decade

Saeed Solali

39 papers receiving 888 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Saeed Solali Iran 16 603 271 129 117 110 41 896
Majid Farshdousti Hagh Iran 20 733 1.2× 251 0.9× 176 1.4× 130 1.1× 202 1.8× 57 1.1k
Yu Hou China 20 680 1.1× 287 1.1× 103 0.8× 103 0.9× 81 0.7× 55 1.0k
Soňa Hubáčková Czechia 15 646 1.1× 208 0.8× 158 1.2× 246 2.1× 72 0.7× 27 1.1k
Ping Tang China 14 591 1.0× 218 0.8× 159 1.2× 130 1.1× 40 0.4× 25 862
Maria Mesuraca Italy 20 631 1.0× 181 0.7× 129 1.0× 69 0.6× 80 0.7× 41 966
Peter Breslin United States 16 424 0.7× 184 0.7× 84 0.7× 176 1.5× 72 0.7× 34 737
Christina Körbel Germany 19 630 1.0× 234 0.9× 264 2.0× 154 1.3× 60 0.5× 39 1.3k

Countries citing papers authored by Saeed Solali

Since Specialization
Citations

This map shows the geographic impact of Saeed Solali's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Saeed Solali with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Saeed Solali more than expected).

Fields of papers citing papers by Saeed Solali

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Saeed Solali. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Saeed Solali. The network helps show where Saeed Solali may publish in the future.

Co-authorship network of co-authors of Saeed Solali

This figure shows the co-authorship network connecting the top 25 collaborators of Saeed Solali. A scholar is included among the top collaborators of Saeed Solali based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Saeed Solali. Saeed Solali is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
2.
Solali, Saeed, et al.. (2024). Study of TRAIL and SAHA Co-Treatment on Leukemia K562 Cell Line. Cell Biochemistry and Biophysics. 83(1). 1087–1096.
3.
Rasoulzadeh, Yahya, et al.. (2023). Hematotoxicity induced by simultaneous exposure to noise and toluene in New Zealand white rabbits: Synergistic and antagonistic effects. Toxicology and Industrial Health. 39(8). 451–463. 2 indexed citations
4.
Kazemi, Tohid, et al.. (2023). Pharmacological and therapeutic potentials of cordycepin in hematological malignancies. Biochemical and Biophysical Research Communications. 678. 135–143. 4 indexed citations
5.
Najafi, Fatemeh, et al.. (2022). The contributing factors of resistance or sensitivity to epigenetic drugs in the treatment of AML. Clinical & Translational Oncology. 24(7). 1250–1261. 4 indexed citations
6.
Najafi, Fatemeh, et al.. (2022). The role of miRNA-424 and miR-631 in various cancers: Focusing on drug resistance and sensitivity. Pathology - Research and Practice. 239. 154130–154130. 5 indexed citations
7.
Farsani, Mehdi Allahbakhshian, et al.. (2021). Investigating the expression pattern of the angiopoietin-Tie system in ALL and its correlation with baseline characteristics. Blood Research. 56(2). 79–85. 3 indexed citations
8.
Rahman, Heshu Sulaiman, Marwah Suliman Maashi, Svetlana Danshina, et al.. (2021). MicroRNAs and JAK/STAT3 signaling: A new promising therapeutic axis in blood cancers. Genes & Diseases. 9(4). 849–867. 25 indexed citations
9.
Mohammadi, Ali, et al.. (2021). Effect of Mimic Hypoxia on the Proliferation and Expression of miR-27a, miR-9, miR-370 and their Target Genes in MOLT-4 and KG1a Cell Lines. Asian Pacific Journal of Cancer Prevention. 22(6). 1975–1984. 1 indexed citations
10.
Rahman, Heshu Sulaiman, Masoud Soleimani, Mohammad Asghari Jafarabadi, et al.. (2021). Kaempferol sensitizes tumor necrosis factor-related apoptosis-inducing ligand-resistance chronic myelogenous leukemia cells to apoptosis. Molecular Biology Reports. 49(1). 19–29. 9 indexed citations
11.
Mohammadi, Ali, et al.. (2020). microRNAs involved in T‐cell development, selection, activation, and hemostasis. Journal of Cellular Physiology. 235(11). 8461–8471. 14 indexed citations
12.
Hagh, Majid Farshdousti, et al.. (2019). An investigation of methylation pattern changes in the IKZF1 promoter in patients with childhood B-cell acute lymphoblastic leukemia. Blood Research. 54(2). 144–148. 4 indexed citations
13.
Marofi, Faroogh, et al.. (2018). Gene expression of TWIST1 and ZBTB16 is regulated by methylation modifications during the osteoblastic differentiation of mesenchymal stem cells. Journal of Cellular Physiology. 234(5). 6230–6243. 34 indexed citations
14.
Soleimani, Masoud, et al.. (2018). The role of XIAP in resistance to TNF-related apoptosis-inducing ligand (TRAIL) in Leukemia. Biomedicine & Pharmacotherapy. 107. 1010–1019. 35 indexed citations
15.
Talebi, Mehdi, et al.. (2017). Aberrant DNA methylation of key genes and Acute Lymphoblastic Leukemia. Biomedicine & Pharmacotherapy. 97. 1493–1500. 32 indexed citations
16.
Naimi, Adel, Ali Akbar Movassaghpour, Majid Farshdousti Hagh, et al.. (2017). TNF-related apoptosis-inducing ligand (TRAIL) as the potential therapeutic target in hematological malignancies. Biomedicine & Pharmacotherapy. 98. 566–576. 36 indexed citations
17.
Zare, Maryam, Milad Bastami, Saeed Solali, & Mohammad Reza Alivand. (2017). Aberrant miRNA promoter methylation and EMT‐involving miRNAs in breast cancer metastasis: Diagnosis and therapeutic implications. Journal of Cellular Physiology. 233(5). 3729–3744. 76 indexed citations
18.
Shirjang, Solmaz, Behzad Mansoori, Saeed Solali, Majid Farshdousti Hagh, & Karim Shamsasenjan. (2016). Toll-like receptors as a key regulator of mesenchymal stem cell function: An up-to-date review. Cellular Immunology. 315. 1–10. 84 indexed citations
19.
Solali, Saeed, et al.. (2015). Isolation and characterization of mesenchymal stem cells derived from adipose tissue.. Koomesh Journal. 16(4). 505–511. 1 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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