S. Sandler

7.0k total citations
94 papers, 2.9k citations indexed

About

S. Sandler is a scholar working on Surgery, Genetics and Endocrinology, Diabetes and Metabolism. According to data from OpenAlex, S. Sandler has authored 94 papers receiving a total of 2.9k indexed citations (citations by other indexed papers that have themselves been cited), including 76 papers in Surgery, 48 papers in Genetics and 47 papers in Endocrinology, Diabetes and Metabolism. Recurrent topics in S. Sandler's work include Pancreatic function and diabetes (76 papers), Diabetes and associated disorders (48 papers) and Diabetes Management and Research (25 papers). S. Sandler is often cited by papers focused on Pancreatic function and diabetes (76 papers), Diabetes and associated disorders (48 papers) and Diabetes Management and Research (25 papers). S. Sandler collaborates with scholars based in Sweden, Denmark and United States. S. Sandler's co-authors include Décio L. Eizirik, Leif Jansson, Arne Andersson, Nils Welsh, Claes Hellerström, Malin Flodström‐Tullberg, Klaus Bendtzen, Eva Strandell, Marina Ćetković-Cvrlje and Olle Korsgren and has published in prestigious journals such as Journal of Clinical Investigation, The Journal of Clinical Endocrinology & Metabolism and Diabetes.

In The Last Decade

S. Sandler

94 papers receiving 2.8k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
S. Sandler Sweden 31 1.8k 1.3k 1.1k 669 449 94 2.9k
Alberto M. Davalli Italy 28 1.9k 1.0× 1.1k 0.8× 1.1k 1.0× 840 1.3× 374 0.8× 65 3.3k
Ν. Kaiser Israel 27 1.2k 0.6× 638 0.5× 798 0.7× 1.0k 1.5× 438 1.0× 46 2.5k
Joachim Størling Denmark 29 1.4k 0.8× 982 0.8× 736 0.7× 1.2k 1.8× 385 0.9× 81 3.2k
Sarah A. Tersey United States 29 1.2k 0.6× 742 0.6× 558 0.5× 917 1.4× 364 0.8× 77 2.3k
Hiroto Furuta Japan 32 1.8k 1.0× 1.2k 1.0× 1.0k 0.9× 1.5k 2.2× 547 1.2× 98 3.7k
Asllan Gjinovci Switzerland 29 2.0k 1.1× 651 0.5× 1.2k 1.1× 1.4k 2.1× 484 1.1× 52 3.1k
Stewart A. Metz United States 37 1.9k 1.0× 554 0.4× 923 0.8× 2.1k 3.1× 478 1.1× 113 3.6k
Hans Kofod Denmark 22 1.4k 0.7× 508 0.4× 1.5k 1.3× 853 1.3× 378 0.8× 47 2.7k
Mitsuhisa Komatsu Japan 29 1.4k 0.8× 479 0.4× 1.0k 0.9× 1.2k 1.7× 330 0.7× 114 2.6k
María E. Pueyo France 21 641 0.3× 330 0.3× 488 0.4× 818 1.2× 679 1.5× 34 2.3k

Countries citing papers authored by S. Sandler

Since Specialization
Citations

This map shows the geographic impact of S. Sandler's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by S. Sandler with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites S. Sandler more than expected).

Fields of papers citing papers by S. Sandler

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by S. Sandler. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by S. Sandler. The network helps show where S. Sandler may publish in the future.

Co-authorship network of co-authors of S. Sandler

This figure shows the co-authorship network connecting the top 25 collaborators of S. Sandler. A scholar is included among the top collaborators of S. Sandler based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with S. Sandler. S. Sandler is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Börjesson, Anna, Sif G. Rønn, Allan E. Karlsen, Nils Billestrup, & S. Sandler. (2011). β-cell specific overexpression of suppressor of cytokine signalling-3 does not protect against multiple low dose streptozotocin induced type 1 diabetes in mice. Immunology Letters. 136(1). 74–79. 4 indexed citations
2.
Hässler, Signe, Leena Peltonen, S. Sandler, & Ola Winqvist. (2008). Aire Deficiency Causes Increased Susceptibility to Streptozotocin‐induced Murine Type 1 Diabetes. Scandinavian Journal of Immunology. 67(6). 569–580. 5 indexed citations
3.
Stridsberg, Mats, et al.. (2005). Expression of somatostatin receptor subtypes 1–5 in pancreatic islets of normoglycaemic and diabetic NOD mice. European Journal of Endocrinology. 153(3). 445–454. 8 indexed citations
4.
Størling, Joachim, Anna Andersson, Richard Züllig, et al.. (2005). Nitric oxide contributes to cytokine-induced apoptosis in pancreatic beta cells via potentiation of JNK activity and inhibition of Akt. Diabetologia. 48(10). 2039–2050. 124 indexed citations
6.
Panerai, Alberto E., Ignacio Conget, A Bartorelli, et al.. (2001). MFP14, a multifunctional emerging protein with immunomodulatory properties, prevents spontaneous and recurrent autoimmune diabetes in NOD mice. Diabetologia. 44(7). 839–847. 8 indexed citations
7.
Sandler, S., et al.. (1999). Prolactin protects against diabetes induced by multiple low doses of streptozotocin in mice. Journal of Endocrinology. 163(2). 229–234. 32 indexed citations
8.
Eizirik, Décio L., et al.. (1997). IL-1 receptor antagonist inhibits recurrence of disease after syngeneic pancreatic islet transplantation to spontaneously diabetic non-obese diabetic (NOD) mice. Clinical & Experimental Immunology. 108(2). 314–317. 65 indexed citations
9.
Strandell, Eva & S. Sandler. (1997). In vitro response to interleukin-1β and streptozotocin in pancreatic islets isolated from male and female nonobese diabetic mice. Journal of Endocrinology. 153(1). 81–86. 5 indexed citations
10.
Reddy, Shiva & S. Sandler. (1995). Age-Dependent Sensitivity to Streptozotocin of Pancreatic Islets Isolated from Female NOD Mice. Autoimmunity. 22(2). 121–126. 5 indexed citations
11.
Eizirik, Décio L., S. Sandler, Nils Welsh, et al.. (1994). Cytokines suppress human islet function irrespective of their effects on nitric oxide generation.. Journal of Clinical Investigation. 93(5). 1968–1974. 272 indexed citations
12.
Welsh, Nils, Boris A. Margulis, Klaus Bendtzen, & S. Sandler. (1994). Liposomal delivery of antioxidant enzymes protects against hydrogen peroxide- but not interleukin-1β-induced inhibition of glucose metabolism in rat pancreatic islets. Journal of Endocrinology. 143(1). 151–156. 15 indexed citations
14.
Sandler, S., Leif Jansson, & Nils Welsh. (1992). Adaptive response in β-cell function in pancreatic islets isolated from partially pancreatectomized rats. Molecular and Cellular Endocrinology. 86(3). 149–156. 4 indexed citations
15.
Eizirik, Décio L., Daniel E. Tracey, Klaus Bendtzen, & S. Sandler. (1991). An interleukin-1 receptor antagonist protein protects insulin-producing Beta cells against suppressive effects of interleukin-1?. Diabetologia. 34(6). 445–448. 71 indexed citations
16.
Eizirik, Décio L., Eva Strandell, & S. Sandler. (1991). Prolonged exposure of pancreatic islets isolated from ?pre-diabetic? non-obese diabetic mice to a high glucose concentration does not impair Beta-cell function. Diabetologia. 34(1). 6–11. 15 indexed citations
17.
Korsgren, Olle, Leif Jansson, S. Sandler, & Arne Andersson. (1990). Hyperglycemia-induced B cell toxicity. The fate of pancreatic islets transplanted into diabetic mice is dependent on their genetic background.. Journal of Clinical Investigation. 86(6). 2161–2168. 74 indexed citations
18.
19.
Strandell, Eva, Arne Andersson, C. G. Groth, & S. Sandler. (1989). Effects of (‐)15‐Deoxyspergualin on Pancreatic Islet B‐Cell Function in Vitro and on the Development of Diabetes after Multiple Low Dose Streptozotocin Administration. Pharmacology & Toxicology. 65(2). 114–118. 10 indexed citations
20.
Jansson, Leif & S. Sandler. (1988). The protective effect of glucose, but not 3-O-methyl glucose, against alloxan-induced diabetes depends upon the route of hexose administration.. PubMed. 293. 257–64. 1 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

Explore authors with similar magnitude of impact

Rankless by CCL
2026