S Lachance

444 total citations
19 papers, 369 citations indexed

About

S Lachance is a scholar working on Molecular Biology, Cardiology and Cardiovascular Medicine and Endocrinology, Diabetes and Metabolism. According to data from OpenAlex, S Lachance has authored 19 papers receiving a total of 369 indexed citations (citations by other indexed papers that have themselves been cited), including 10 papers in Molecular Biology, 6 papers in Cardiology and Cardiovascular Medicine and 6 papers in Endocrinology, Diabetes and Metabolism. Recurrent topics in S Lachance's work include Renin-Angiotensin System Studies (6 papers), Receptor Mechanisms and Signaling (5 papers) and Hormonal Regulation and Hypertension (5 papers). S Lachance is often cited by papers focused on Renin-Angiotensin System Studies (6 papers), Receptor Mechanisms and Signaling (5 papers) and Hormonal Regulation and Hypertension (5 papers). S Lachance collaborates with scholars based in Canada, India and United States. S Lachance's co-authors include John S.D. Chan, S Carrière, Aline Delalandre, Claude Perreault, Chantal Baron, János G. Filep, Chantale Lapierre, Géraldine Mathonnet, Moulay A. Alaoui‐Jamali and Elliot Drobetsky and has published in prestigious journals such as Blood, Biochemical Journal and Kidney International.

In The Last Decade

S Lachance

18 papers receiving 362 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
S Lachance Canada 13 149 95 83 64 59 19 369
Juan Carlos Monge Canada 13 153 1.0× 160 1.7× 56 0.7× 146 2.3× 46 0.8× 18 431
Christian Böhm Germany 11 248 1.7× 75 0.8× 57 0.7× 92 1.4× 106 1.8× 16 488
K A Chandrabose United States 8 135 0.9× 62 0.7× 39 0.5× 67 1.0× 60 1.0× 11 418
О. Е. Мустафина Russia 10 97 0.7× 56 0.6× 56 0.7× 50 0.8× 72 1.2× 76 352
Wendell-Lamar B. Blackwell United States 8 176 1.2× 159 1.7× 72 0.9× 38 0.6× 45 0.8× 8 458
J M Grognet France 10 212 1.4× 156 1.6× 61 0.7× 33 0.5× 35 0.6× 19 498
J H Zhang United States 5 132 0.9× 37 0.4× 121 1.5× 56 0.9× 97 1.6× 7 503
Carina Hallberg Sweden 7 156 1.0× 41 0.4× 85 1.0× 37 0.6× 38 0.6× 9 360
Camille Malaval France 6 233 1.6× 53 0.6× 87 1.0× 87 1.4× 58 1.0× 7 483

Countries citing papers authored by S Lachance

Since Specialization
Citations

This map shows the geographic impact of S Lachance's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by S Lachance with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites S Lachance more than expected).

Fields of papers citing papers by S Lachance

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by S Lachance. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by S Lachance. The network helps show where S Lachance may publish in the future.

Co-authorship network of co-authors of S Lachance

This figure shows the co-authorship network connecting the top 25 collaborators of S Lachance. A scholar is included among the top collaborators of S Lachance based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with S Lachance. S Lachance is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

19 of 19 papers shown
1.
Roy, Denis‐Claude, S Lachance, Jean Roy, et al.. (2016). Donor lymphocytes depleted of alloreactive T-cells (ATIR101) improve overall survival and reduce transplant related mortality in a T-cell depleted haploidentical HSCT: Results from a Phase 2 trial in patients with AML and ALL. Bone Marrow Transplantation. 51.
2.
Lachance, S, Anna Christofides, Laurie H. Sehn, et al.. (2016). A Canadian Perspective on the Use of Immunoglobulin Therapy to Reduce Infectious Complications in Chronic Lymphocytic Leukemia. Current Oncology. 23(1). 42–51. 25 indexed citations
3.
4.
Mathonnet, Géraldine, S Lachance, Moulay A. Alaoui‐Jamali, & Elliot Drobetsky. (2004). Expression of hepatitis B virus X oncoprotein inhibits transcription-coupled nucleotide excision repair in human cells. Mutation research. Fundamental and molecular mechanisms of mutagenesis. 554(1-2). 305–318. 37 indexed citations
5.
Qian, Jing, Jie Wu, Chunxi Ge, et al.. (1997). Angiotensinogen gene expression is stimulated by the cAMP-responsive element binding protein in opossum kidney cells.. Journal of the American Society of Nephrology. 8(7). 1072–1079. 23 indexed citations
6.
Filep, János G., Chantale Lapierre, S Lachance, & John S.D. Chan. (1997). Nitric oxide co-operates with hydrogen peroxide in inducing DNA fragmentation and cell lysis in murine lymphoma cells. Biochemical Journal. 321(3). 897–901. 41 indexed citations
7.
Chan, Wynnie, Kenneth D. Roberts, Michel Bouvier, et al.. (1996). β-Adrenoceptors and dexamethasone synergistically stimulate the expression of the angiotensinogen gene in opossum kidney cells. Kidney International. 50(1). 94–101. 15 indexed citations
8.
Baron, Chantal, et al.. (1996). Involvement of nitric oxide in target-cell lysis and DNA fragmentation induced by murine natural killer cells. Blood. 87(12). 5136–5143. 53 indexed citations
9.
Lachance, S, et al.. (1996). Dopaminergic receptors and angiotensinogen gene expression in opossum kidney cells. American Journal of Physiology-Regulatory, Integrative and Comparative Physiology. 271(3). R519–R527. 8 indexed citations
10.
Lachance, S, et al.. (1995). Alpha-adrenoceptors and angiotensinogen gene expression in opossum kidney cells. Kidney International. 48(1). 139–145. 7 indexed citations
11.
Lachance, S, et al.. (1995). Expression of the angiotensinogen gene is synergistically stimulated by 8-BrcAMP and Dex in opossum kidney cells. American Journal of Physiology-Regulatory, Integrative and Comparative Physiology. 268(1). R105–R111. 8 indexed citations
12.
Wu, Jie, et al.. (1995). β-Adrenergic Receptors and Angiotensinogen Gene Expression in Mouse Hepatoma Cells In Vitro. Hypertension. 25(1). 105–109. 14 indexed citations
13.
Chen, Ming, et al.. (1994). Isoproterenol and 8-bromo-cyclic adenosine monophosphate stimulate the expression of the angiotensinogen gene in opossum kidney cells. Kidney International. 46(3). 703–710. 17 indexed citations
14.
Sikstrom, R, et al.. (1993). Hormonal Regulation of Expression of the Angiotensinoeen Gene in Cultured Mouse Hepatoma Cells. American Journal of Hypertension. 6(2). 141–148. 10 indexed citations
15.
Chan, John S.D., et al.. (1992). Hormonal regulation of expression of the angiotensinogen gene in cultured opossum kidney proximal tubular cells.. Journal of the American Society of Nephrology. 2(10). 1516–1522. 19 indexed citations
16.
Chan, John S.D., et al.. (1992). Thyroid hormone, L-T3, stimulates the expression of the angiotensinogen gene in cultured opossum kidney (OK) cells.. Journal of the American Society of Nephrology. 2(8). 1360–1367. 17 indexed citations
17.
Chan, John S.D., et al.. (1990). Molecular cloning and expression of the rat angiotensinogen gene. Pediatric Nephrology. 4(4). 429–435. 25 indexed citations
18.
Lachance, S, et al.. (1983). Turnover of free and conjugated serum catecholamines during hemodialysis.. PubMed. 6(1). 11–7. 7 indexed citations
19.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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