Ruthie E. Amir

6.9k total citations · 1 hit paper
21 papers, 5.1k citations indexed

About

Ruthie E. Amir is a scholar working on Molecular Biology, Genetics and Cardiology and Cardiovascular Medicine. According to data from OpenAlex, Ruthie E. Amir has authored 21 papers receiving a total of 5.1k indexed citations (citations by other indexed papers that have themselves been cited), including 10 papers in Molecular Biology, 10 papers in Genetics and 7 papers in Cardiology and Cardiovascular Medicine. Recurrent topics in Ruthie E. Amir's work include Genetics and Neurodevelopmental Disorders (8 papers), Epigenetics and DNA Methylation (5 papers) and Autism Spectrum Disorder Research (5 papers). Ruthie E. Amir is often cited by papers focused on Genetics and Neurodevelopmental Disorders (8 papers), Epigenetics and DNA Methylation (5 papers) and Autism Spectrum Disorder Research (5 papers). Ruthie E. Amir collaborates with scholars based in Israel, United States and Portugal. Ruthie E. Amir's co-authors include Huda Y. Zoghbi, Uta Francke, Mimi Wan, Ignatia B. Van den Veyver, Aaron Ciechanover, Michael Sagiv, H.-R. Song, Xianyu Zhang, José Luiz Pinto Pereira and N. Carolyn Schanen and has published in prestigious journals such as Journal of Biological Chemistry, Nature Genetics and Oncogene.

In The Last Decade

Ruthie E. Amir

21 papers receiving 5.0k citations

Hit Papers

Rett syndrome is caused by mutations in X-linked MECP2, e... 1999 2026 2008 2017 1999 1000 2.0k 3.0k

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Ruthie E. Amir Israel 14 4.1k 3.0k 2.3k 541 260 21 5.1k
Santhosh Girirajan United States 34 3.4k 0.8× 2.5k 0.8× 1.0k 0.5× 134 0.2× 399 1.5× 82 5.0k
Jeffrey L. Neul United States 45 6.1k 1.5× 3.2k 1.0× 4.4k 1.9× 1.6k 3.0× 612 2.4× 129 7.6k
B. A. Oostra Netherlands 35 2.1k 0.5× 1.9k 0.6× 999 0.4× 212 0.4× 422 1.6× 72 3.7k
Jacky Guy United Kingdom 27 3.9k 1.0× 4.3k 1.4× 1.9k 0.8× 188 0.3× 362 1.4× 33 5.7k
Michele Zappella Italy 30 3.5k 0.8× 1.5k 0.5× 2.5k 1.1× 833 1.5× 147 0.6× 96 4.2k
James S. Sutcliffe United States 45 6.7k 1.6× 5.1k 1.7× 3.9k 1.7× 467 0.9× 1.4k 5.3× 92 9.5k
N. Carolyn Schanen United States 27 3.1k 0.8× 2.0k 0.7× 1.9k 0.8× 474 0.9× 271 1.0× 45 3.8k
Sakkubai Naidu United States 33 1.0k 0.3× 3.2k 1.1× 645 0.3× 211 0.4× 351 1.4× 80 4.4k
Maria H. Chahrour United States 17 3.0k 0.7× 2.8k 0.9× 1.8k 0.8× 191 0.4× 632 2.4× 31 4.5k
Hyejung Won United States 21 2.1k 0.5× 2.3k 0.7× 1.4k 0.6× 125 0.2× 518 2.0× 45 4.0k

Countries citing papers authored by Ruthie E. Amir

Since Specialization
Citations

This map shows the geographic impact of Ruthie E. Amir's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Ruthie E. Amir with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Ruthie E. Amir more than expected).

Fields of papers citing papers by Ruthie E. Amir

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Ruthie E. Amir. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Ruthie E. Amir. The network helps show where Ruthie E. Amir may publish in the future.

Co-authorship network of co-authors of Ruthie E. Amir

This figure shows the co-authorship network connecting the top 25 collaborators of Ruthie E. Amir. A scholar is included among the top collaborators of Ruthie E. Amir based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Ruthie E. Amir. Ruthie E. Amir is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Eynon, Nir, Yoav Meckel, Michael Sagiv, et al.. (2010). Do PPARGC1A and PPARα polymorphisms influence sprint or endurance phenotypes?. Scandinavian Journal of Medicine and Science in Sports. 20(1). e145–50. 67 indexed citations
2.
Amir, Offer, Moran Sagiv, Nir Eynon, et al.. (2010). The response of circulating brain natriuretic peptide to academic stress in college students. Stress. 13(1). 83–90. 13 indexed citations
3.
Amir, Ruthie E., et al.. (2010). Effect of different sprint training regimes on the oxygen delivery-extraction in elite sprinters.. PubMed. 50(2). 121–5. 1 indexed citations
4.
Amir, Offer, Ori Rogowski, Marina Bar‐Shai, et al.. (2009). Serum Oxidative Stress Level Correlates with Clinical Parameters in Chronic Systolic Heart Failure Patients. Clinical Cardiology. 32(4). 199–203. 24 indexed citations
5.
Amir, Offer, et al.. (2009). Acute incremental exercise to maximal performance does not cause alterations in serum oxidant levels of healthy young individuals.. PubMed. 49(1). 105–11. 3 indexed citations
6.
Chen, Yamin, José Alberto Duarte, José Oliveira, et al.. (2008). IL6 (-174) and TNFA (-308) promoter polymorphisms are associated with systemic creatine kinase response to eccentric exercise. European Journal of Applied Physiology. 104(3). 579–586. 74 indexed citations
7.
Amir, Offer, et al.. (2008). Aldosterone Synthase Gene Polymorphism as a Determinant of Atrial Fibrillation in Patients With Heart Failure. The American Journal of Cardiology. 102(3). 326–329. 35 indexed citations
8.
Amir, Offer, et al.. (2008). Relation between AT1R Gene Polymorphism and Long-Term Outcome in Patients with Heart Failure. Cardiology. 112(2). 151–157. 11 indexed citations
9.
10.
Chen, Yamin, Offer Amir, Moran Sagiv, et al.. (2007). ACE ID genotype affects blood creatine kinase response to eccentric exercise. Journal of Applied Physiology. 103(6). 2057–2061. 54 indexed citations
11.
Amir, Ruthie E., et al.. (2005). Mutations in exon 1 of MECP2 are a rare cause of Rett syndrome. Journal of Medical Genetics. 42(2). e15–e15. 45 indexed citations
12.
Amir, Ruthie E., V. Reid Sutton, & Ignatia B. Van den Veyver. (2005). Newborn Screening and Prenatal Diagnosis for Rett Syndrome: Implications for Therapy. Journal of Child Neurology. 20(9). 779–783. 8 indexed citations
14.
Amir, Ruthie E., Kazuhiro Iwaï, & Aaron Ciechanover. (2002). The NEDD8 Pathway Is Essential for SCFβ-TrCP-mediated Ubiquitination and Processing of the NF-κB Precursor p105. Journal of Biological Chemistry. 277(26). 23253–23259. 66 indexed citations
15.
Amir, Ruthie E., Aaron Ciechanover, & Shai Cohen. (2001). [The ubiquitin-proteasome system: the relationship between protein degradation and human diseases].. PubMed. 140(12). 1172–6, 1229. 8 indexed citations
16.
Buyse, I., et al.. (2000). Diagnostic Testing for Rett Syndrome by DHPLC and Direct Sequencing Analysis of the MECP2 Gene: Identification of Several Novel Mutations and Polymorphisms. The American Journal of Human Genetics. 67(6). 1428–1436. 132 indexed citations
17.
Amir, Ruthie E. & Huda Y. Zoghbi. (2000). Rett syndrome: Methyl-CpG-binding protein 2 mutations and phenotype-genotype correlations. American Journal of Medical Genetics. 97(2). 147–152. 148 indexed citations
18.
Amir, Ruthie E., Ignatia B. Van den Veyver, Denise Malicki, et al.. (2000). Influence of mutation type and X chromosome inactivation on Rett syndrome phenotypes. Annals of Neurology. 47(5). 670–679. 28 indexed citations
19.
Wan, Mimi, Xianyu Zhang, Elisa J. F. Houwink, et al.. (1999). Rett Syndrome and Beyond: Recurrent Spontaneous and Familial MECP2 Mutations at CpG Hotspots. The American Journal of Human Genetics. 65(6). 1520–1529. 369 indexed citations
20.
Amir, Ruthie E., et al.. (1999). Rett syndrome is caused by mutations in X-linked MECP2, encoding methyl-CpG-binding protein 2. Nature Genetics. 23(2). 185–188. 3664 indexed citations breakdown →

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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