Ruth N. MacKinnon

1.0k total citations
31 papers, 739 citations indexed

About

Ruth N. MacKinnon is a scholar working on Molecular Biology, Genetics and Hematology. According to data from OpenAlex, Ruth N. MacKinnon has authored 31 papers receiving a total of 739 indexed citations (citations by other indexed papers that have themselves been cited), including 21 papers in Molecular Biology, 18 papers in Genetics and 10 papers in Hematology. Recurrent topics in Ruth N. MacKinnon's work include Genomic variations and chromosomal abnormalities (11 papers), Acute Myeloid Leukemia Research (9 papers) and Chromosomal and Genetic Variations (7 papers). Ruth N. MacKinnon is often cited by papers focused on Genomic variations and chromosomal abnormalities (11 papers), Acute Myeloid Leukemia Research (9 papers) and Chromosomal and Genetic Variations (7 papers). Ruth N. MacKinnon collaborates with scholars based in Australia, United Kingdom and United States. Ruth N. MacKinnon's co-authors include Lynda J. Campbell, Mark C. Hirst, M.V. Bell, A. Roche, Yutaka Nakahori, Kay E. Davies, Tracey Flint, Robin Winter, Niels Tommerup and U. Froster‐Iskenius and has published in prestigious journals such as Cell, Nucleic Acids Research and Scientific Reports.

In The Last Decade

Ruth N. MacKinnon

30 papers receiving 725 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Ruth N. MacKinnon Australia 15 523 449 136 92 82 31 739
Marijke Bauters Belgium 17 809 1.5× 911 2.0× 199 1.5× 138 1.5× 96 1.2× 25 1.3k
Donna M. Muzny United States 15 551 1.1× 456 1.0× 103 0.8× 34 0.4× 21 0.3× 20 854
M.V. Bell United Kingdom 13 790 1.5× 482 1.1× 164 1.2× 46 0.5× 22 0.3× 15 1.1k
Gordana Raca United States 12 421 0.8× 235 0.5× 29 0.2× 33 0.4× 107 1.3× 28 667
Pamela Magini Italy 16 347 0.7× 349 0.8× 35 0.3× 47 0.5× 23 0.3× 38 717
M. F. Bertheas France 9 889 1.7× 1.1k 2.4× 519 3.8× 46 0.5× 90 1.1× 17 1.5k
Benno Röthlisberger Switzerland 17 313 0.6× 462 1.0× 25 0.2× 92 1.0× 44 0.5× 34 729
Tiina Alitalo Finland 20 604 1.2× 332 0.7× 47 0.3× 61 0.7× 21 0.3× 30 964
Kenji Kokura Japan 13 784 1.5× 204 0.5× 53 0.4× 33 0.4× 25 0.3× 27 912
Devin McQuaid United States 10 312 0.6× 337 0.8× 132 1.0× 61 0.7× 10 0.1× 12 562

Countries citing papers authored by Ruth N. MacKinnon

Since Specialization
Citations

This map shows the geographic impact of Ruth N. MacKinnon's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Ruth N. MacKinnon with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Ruth N. MacKinnon more than expected).

Fields of papers citing papers by Ruth N. MacKinnon

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Ruth N. MacKinnon. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Ruth N. MacKinnon. The network helps show where Ruth N. MacKinnon may publish in the future.

Co-authorship network of co-authors of Ruth N. MacKinnon

This figure shows the co-authorship network connecting the top 25 collaborators of Ruth N. MacKinnon. A scholar is included among the top collaborators of Ruth N. MacKinnon based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Ruth N. MacKinnon. Ruth N. MacKinnon is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
2.
Ku, Matthew, Ruth N. MacKinnon, Meaghan Wall, et al.. (2019). Hemopoietic Cell Kinase amplification with Protein Tyrosine Phosphatase Receptor T depletion leads to polycythemia, aberrant marrow erythoid maturation, and splenomegaly. Scientific Reports. 9(1). 7050–7050. 4 indexed citations
3.
MacKinnon, Ruth N.. (2018). Analysis of Chromothripsis by Combined FISH and Microarray Analysis. Methods in molecular biology. 53–77. 3 indexed citations
4.
MacKinnon, Ruth N., Hendrika M. Duivenvoorden, Lynda J. Campbell, & Meaghan Wall. (2016). The Dicentric Chromosome dic(20;22) Is a Recurrent Abnormality in Myelodysplastic Syndromes and Is a Product of Telomere Fusion. Cytogenetic and Genome Research. 150(3-4). 262–272. 2 indexed citations
5.
MacKinnon, Ruth N. & Lynda J. Campbell. (2013). Chromothripsis under the microscope: a cytogenetic perspective of two cases of AML with catastrophic chromosome rearrangement. Cancer Genetics. 206(6). 238–251. 23 indexed citations
6.
MacKinnon, Ruth N., et al.. (2012). CGH and SNP array using DNA extracted from fixed cytogenetic preparations and long-term refrigerated bone marrow specimens. Molecular Cytogenetics. 5(1). 10–10. 6 indexed citations
7.
MacKinnon, Ruth N., George Kannourakis, Meaghan Wall, & Lynda J. Campbell. (2011). A cryptic deletion in 5q31.2 provides further evidence for a minimally deleted region in myelodysplastic syndromes. Cancer Genetics. 204(4). 187–194. 22 indexed citations
8.
MacKinnon, Ruth N. & Ilse Chudoba. (2011). The Use of M-FISH and M-BAND to Define Chromosome Abnormalities. Methods in molecular biology. 730. 203–218. 8 indexed citations
9.
MacKinnon, Ruth N., Hendrika M. Duivenvoorden, & Lynda J. Campbell. (2011). Unbalanced translocations of 20q in AML and MDS often involve interstitial rather than terminal deletions of 20q. Cancer Genetics. 204(3). 153–161. 5 indexed citations
10.
MacKinnon, Ruth N., Carly Selan, Meaghan Wall, et al.. (2010). The paradox of 20q11.21 amplification in a subset of cases of myeloid malignancy with chromosome 20 deletion. Genes Chromosomes and Cancer. 49(11). 998–1013. 39 indexed citations
11.
MacKinnon, Ruth N. & Lynda J. Campbell. (2007). Dicentric chromosomes and 20q11.2 amplification in MDS/AML with apparent monosomy 20. Cytogenetic and Genome Research. 119(3-4). 211–220. 14 indexed citations
12.
13.
MacKinnon, Ruth N. & Lynda J. Campbell. (2005). A comparison of two contrasting recurrent isochromosomes 20 found in myelodysplastic syndromes suggests that retention of proximal 20q is a significant factor in myeloid malignancies. Cancer Genetics and Cytogenetics. 163(2). 176–179. 13 indexed citations
14.
MacKinnon, Ruth N., et al.. (2005). Recurrent duplication of Xq27∼qter in hematological malignancies revealed by multicolor fluorescence in situ hybridization and multicolor banding. Cancer Genetics and Cytogenetics. 161(2). 125–129. 2 indexed citations
15.
Blair, Helen J., Mark C. Hirst, Ruth N. MacKinnon, Kay E. Davies, & Yvonne Boyd. (1994). Evaluation of human microdissection clones from the FRAXA region as tools for comparative mapping in the mouse: isolation of a conserved genomic clone close to FMR1. Mammalian Genome. 5(9). 584–587. 2 indexed citations
16.
Bell, M.V., Mark C. Hirst, Yutaka Nakahori, et al.. (1991). Physical mapping across the fragile X: Hypermethylation and clinical expression of the fragile X syndrome. Cell. 64(4). 861–866. 287 indexed citations
17.
Hirst, Mark C., M.V. Bell, Ruth N. MacKinnon, et al.. (1991). Mapping of a cerebellar degeneration related protein and DXS304 around the fragile site. American Journal of Medical Genetics. 38(2-3). 354–356. 7 indexed citations
18.
Hirst, Mark C., Katrina Rack, Yutaka Nakahori, et al.. (1991). A YAC contig across the fragile X site defines the region of fragility. Nucleic Acids Research. 19(12). 3283–3288. 35 indexed citations
19.
MacKinnon, Philip J., Barry C. Powell, George E. Rogers, et al.. (1991). An ultrahigh-sulphur keratin gene of the human hair cuticle is located at 11q13 and cross-hybridizes with sequences at 11p15. Mammalian Genome. 1(1). 53–56. 14 indexed citations
20.
Hyland, V.J., Graeme Suthers, Kathryn Friend, et al.. (1990). Probe, VK5B, is located in the same interval as the autosomal dominant adult polycystic kidney disease locus, PKD1. Human Genetics. 84(3). 286–8. 23 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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