Ruth Eytner

767 total citations
7 papers, 651 citations indexed

About

Ruth Eytner is a scholar working on Immunology and Allergy, Immunology and Cancer Research. According to data from OpenAlex, Ruth Eytner has authored 7 papers receiving a total of 651 indexed citations (citations by other indexed papers that have themselves been cited), including 6 papers in Immunology and Allergy, 4 papers in Immunology and 3 papers in Cancer Research. Recurrent topics in Ruth Eytner's work include Cell Adhesion Molecules Research (6 papers), T-cell and B-cell Immunology (2 papers) and Immune Response and Inflammation (2 papers). Ruth Eytner is often cited by papers focused on Cell Adhesion Molecules Research (6 papers), T-cell and B-cell Immunology (2 papers) and Immune Response and Inflammation (2 papers). Ruth Eytner collaborates with scholars based in Germany, United States and Indonesia. Ruth Eytner's co-authors include Thomas Moll, Dietmar Vestweber, Renate Hofer-Warbinek, Rainer de Martin, Christopher J. Wrighton, Fritz H. Bach, Eric Borges, Martin Steegmaier, Gunther G. Pendl and Horst Mossmann and has published in prestigious journals such as Journal of Biological Chemistry, The Journal of Experimental Medicine and Blood.

In The Last Decade

Ruth Eytner

7 papers receiving 632 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Ruth Eytner Germany 7 338 282 240 149 119 7 651
Adam Asch United States 8 371 1.1× 289 1.0× 171 0.7× 54 0.4× 83 0.7× 27 689
U Jäger Germany 8 177 0.5× 225 0.8× 163 0.7× 53 0.4× 52 0.4× 12 448
Eric Lynam United States 9 195 0.6× 254 0.9× 231 1.0× 77 0.5× 79 0.7× 11 520
Paul W. Gudewicz United States 12 163 0.5× 212 0.8× 148 0.6× 156 1.0× 76 0.6× 31 541
D.E. Zhang United States 11 455 1.3× 109 0.4× 495 2.1× 124 0.8× 153 1.3× 11 938
DD Hickstein United States 9 351 1.0× 273 1.0× 214 0.9× 41 0.3× 76 0.6× 11 630
Deborah Webb United States 11 502 1.5× 173 0.6× 253 1.1× 78 0.5× 290 2.4× 15 894
Barbara Metzler Switzerland 14 656 1.9× 101 0.4× 208 0.9× 51 0.3× 137 1.2× 19 978
Felipe Vences‐Catalán United States 12 235 0.7× 112 0.4× 228 0.9× 55 0.4× 110 0.9× 17 505
Fui G. Goh United Kingdom 8 283 0.8× 65 0.2× 162 0.7× 96 0.6× 91 0.8× 9 571

Countries citing papers authored by Ruth Eytner

Since Specialization
Citations

This map shows the geographic impact of Ruth Eytner's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Ruth Eytner with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Ruth Eytner more than expected).

Fields of papers citing papers by Ruth Eytner

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Ruth Eytner. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Ruth Eytner. The network helps show where Ruth Eytner may publish in the future.

Co-authorship network of co-authors of Ruth Eytner

This figure shows the co-authorship network connecting the top 25 collaborators of Ruth Eytner. A scholar is included among the top collaborators of Ruth Eytner based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Ruth Eytner. Ruth Eytner is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

7 of 7 papers shown
1.
Strobel, Benjamin, Gudrun Zimmermann, Christine Mayer, et al.. (2019). Standardized, Scalable, and Timely Flexible Adeno-Associated Virus Vector Production Using Frozen High-Density HEK-293 Cell Stocks and CELLdiscs. Human Gene Therapy Methods. 30(1). 23–33. 42 indexed citations
2.
Pendl, Gunther G., Caroline Robert, Meike Steinert, et al.. (2002). Immature mouse dendritic cells enter inflamed tissue, a process that requires E- and P-selectin, but not P-selectin glycoprotein ligand 1. Blood. 99(3). 946–956. 64 indexed citations
3.
Pendl, Gunther G., Caroline Robert, Meike Steinert, et al.. (2002). Immature mouse dendritic cells enter inflamed tissue, a process that requires E- and P-selectin, but not P-selectin glycoprotein ligand 1. Blood. 99(3). 946–956. 12 indexed citations
4.
Moll, Thomas, et al.. (1998). Stimulation of P-selectin glycoprotein ligand-1 on mouse neutrophils activates β2 -integrin mediated cell attachment to ICAM-1. European Journal of Immunology. 28(2). 433–443. 114 indexed citations
5.
Borges, Eric, et al.. (1997). The Binding of T Cell-expressed P-selectin Glycoprotein Ligand-1 to E- and P-selectin Is Differentially Regulated. Journal of Biological Chemistry. 272(45). 28786–28792. 95 indexed citations
6.
Borges, Eric, Ruth Eytner, Thomas Moll, et al.. (1997). The P-Selectin Glycoprotein Ligand-1 Is Important for Recruitment of Neutrophils Into Inflamed Mouse Peritoneum. Blood. 90(5). 1934–1942. 103 indexed citations
7.
Wrighton, Christopher J., Renate Hofer-Warbinek, Thomas Moll, et al.. (1996). Inhibition of endothelial cell activation by adenovirus-mediated expression of I kappa B alpha, an inhibitor of the transcription factor NF-kappa B.. The Journal of Experimental Medicine. 183(3). 1013–1022. 221 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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