Rüdiger Streicher

705 total citations
17 papers, 519 citations indexed

About

Rüdiger Streicher is a scholar working on Molecular Biology, Surgery and Cancer Research. According to data from OpenAlex, Rüdiger Streicher has authored 17 papers receiving a total of 519 indexed citations (citations by other indexed papers that have themselves been cited), including 9 papers in Molecular Biology, 5 papers in Surgery and 4 papers in Cancer Research. Recurrent topics in Rüdiger Streicher's work include Metabolism, Diabetes, and Cancer (4 papers), Cancer, Lipids, and Metabolism (3 papers) and Protein Kinase Regulation and GTPase Signaling (3 papers). Rüdiger Streicher is often cited by papers focused on Metabolism, Diabetes, and Cancer (4 papers), Cancer, Lipids, and Metabolism (3 papers) and Protein Kinase Regulation and GTPase Signaling (3 papers). Rüdiger Streicher collaborates with scholars based in Germany, Czechia and Austria. Rüdiger Streicher's co-authors include Stefanie Maurer, Dirk Müller‐Wieland, Wilhelm Krone, Regina Brigelius‐Flohé, Gerhard Siemeister, Jörg Kotzka, Martina Munck, Haluk Avci, Olivier Potterat and Jürgen Mack and has published in prestigious journals such as Journal of Biological Chemistry, Biochemical Journal and Biochemical and Biophysical Research Communications.

In The Last Decade

Rüdiger Streicher

17 papers receiving 507 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Rüdiger Streicher Germany 12 298 130 84 76 60 17 519
John Babiak United States 14 165 0.6× 202 1.6× 95 1.1× 153 2.0× 28 0.5× 23 488
Mark T. Yates United States 13 204 0.7× 170 1.3× 98 1.2× 98 1.3× 49 0.8× 19 731
Cassandra L. Henry United States 11 285 1.0× 70 0.5× 80 1.0× 42 0.6× 117 1.9× 13 657
Laurie L. McLeod United States 8 240 0.8× 153 1.2× 79 0.9× 55 0.7× 19 0.3× 8 511
Ying Yao United States 12 196 0.7× 104 0.8× 45 0.5× 75 1.0× 38 0.6× 23 487
Lawrence M. Zaccaro United States 9 269 0.9× 116 0.9× 41 0.5× 60 0.8× 23 0.4× 12 535
Martin J. Smit Netherlands 13 279 0.9× 212 1.6× 72 0.9× 166 2.2× 13 0.2× 15 671
Aaron T. Lada United States 6 235 0.8× 270 2.1× 51 0.6× 62 0.8× 26 0.4× 7 464
Shuzo Moritani Japan 13 274 0.9× 51 0.4× 44 0.5× 41 0.5× 77 1.3× 19 783
Anna Gantman Israel 8 130 0.4× 59 0.5× 67 0.8× 66 0.9× 35 0.6× 10 378

Countries citing papers authored by Rüdiger Streicher

Since Specialization
Citations

This map shows the geographic impact of Rüdiger Streicher's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Rüdiger Streicher with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Rüdiger Streicher more than expected).

Fields of papers citing papers by Rüdiger Streicher

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Rüdiger Streicher. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Rüdiger Streicher. The network helps show where Rüdiger Streicher may publish in the future.

Co-authorship network of co-authors of Rüdiger Streicher

This figure shows the co-authorship network connecting the top 25 collaborators of Rüdiger Streicher. A scholar is included among the top collaborators of Rüdiger Streicher based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Rüdiger Streicher. Rüdiger Streicher is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

17 of 17 papers shown
1.
Hofmann, Anja, Coy Brunßen, Mirko Peitzsch, et al.. (2016). Aldosterone Synthase Inhibition Improves Glucose Tolerance in Zucker Diabetic Fatty (ZDF) Rats. Endocrinology. 157(10). 3844–3855. 14 indexed citations
2.
Trieselmann, Thomas, Holger Wagner, Klaus Fuchs, et al.. (2014). Potent Cholesteryl Ester Transfer Protein Inhibitors of Reduced Lipophilicity: 1,1′-Spiro-Substituted Hexahydrofuroquinoline Derivatives. Journal of Medicinal Chemistry. 57(21). 8766–8776. 24 indexed citations
3.
Müller‐Wieland, Dirk, Rüdiger Streicher, Gerhard Siemeister, & Wilhelm Krone. (2009). Molecular Biology of Insulin Resistance. Experimental and Clinical Endocrinology & Diabetes. 101(1). 17–29. 1 indexed citations
4.
Pautsch, Alexander, et al.. (2008). Molecular Recognition of the Protein Phosphatase 1 Glycogen Targeting Subunit by Glycogen Phosphorylase. Journal of Biological Chemistry. 283(14). 8913–8918. 25 indexed citations
5.
Zibrova, Darya, Rolf Grempler, Rüdiger Streicher, & Stefan G. Kauschke. (2008). Inhibition of the interaction between protein phosphatase 1 glycogen-targeting subunit and glycogen phosphorylase increases glycogen synthesis in primary rat hepatocytes. Biochemical Journal. 412(2). 359–366. 21 indexed citations
6.
Potterat, Olivier, Klaus Wagner, Gerd Gemmecker, et al.. (2004). BI-32169, a Bicyclic 19-Peptide with Strong Glucagon Receptor Antagonist Activity from Streptomyces sp.. Journal of Natural Products. 67(9). 1528–1531. 67 indexed citations
7.
Streicher, Rüdiger, Jörg Kotzka, Dirk Müller‐Wieland, & Wilhelm Krone. (1998). Regulation of the LDL receptor gene expression by hormones.. PubMed. 37 Suppl 1. 85–7. 3 indexed citations
8.
Brigelius‐Flohé, R., et al.. (1997). Determinants of PHGPx expression in a cultured endothelial cell line.. PubMed. 10(2-3). 163–76. 17 indexed citations
9.
Knebel, Birgit, Jörg Kotzka, Gerhard Siemeister, et al.. (1997). Defects of Insulin and IGF-1 Action at Receptor and Postreceptor Level in a Patient with Type A Syndrome of Insulin Resistance. Biochemical and Biophysical Research Communications. 234(3). 626–630. 7 indexed citations
10.
11.
Streicher, Rüdiger, Jörg Kotzka, Dirk Müller‐Wieland, et al.. (1996). SREBP-1 Mediates Activation of the Low Density Lipoprotein Receptor Promoter by Insulin and Insulin-like Growth Factor-I. Journal of Biological Chemistry. 271(12). 7128–7133. 133 indexed citations
12.
Streicher, Rüdiger, et al.. (1996). Structure of the human apolipoprotein C-II gene promoter.. PubMed. 34 Suppl 3. 49–50. 2 indexed citations
13.
Streicher, Rüdiger, et al.. (1996). A single nucleotide substitution in the promoter region of the apolipoprotein C-II gene identified in individuals with chylomicronemia. Journal of Lipid Research. 37(12). 2599–2607. 17 indexed citations
14.
Siemeister, Gerhard, Hadi Al‐Hasani, Helmut W. Klein, et al.. (1995). Recombinant Human Insulin Receptor Substrate-1 Protein. Journal of Biological Chemistry. 270(9). 4870–4874. 19 indexed citations
15.
Becker, Walter, et al.. (1993). Heterogeneity of insulin receptors in rat tissues as detected with the partial agonist B29,B29'-suberoyl-insulin.. Molecular Pharmacology. 44(2). 271–276. 5 indexed citations
16.
Müller‐Wieland, Dirk, Eric R. van der Vorm, Rüdiger Streicher, et al.. (1993). An in-frame insertion in exon 3 and a nonsense mutation in exon 2 of the insulin receptor gene associated with severe insulin resistance in a patient with Rabson-Mendenhall syndrome. Diabetologia. 36(11). 1168–1174. 17 indexed citations
17.
Streicher, Rüdiger & W. Stoffel. (1989). The Organization of the Human Myelin Basic Protein Gene. Comparison with the Mouse Gene. Biological Chemistry Hoppe-Seyler. 370(1). 503–510. 20 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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