Roy E. Weiss

11.9k total citations
194 papers, 8.0k citations indexed

About

Roy E. Weiss is a scholar working on Endocrinology, Diabetes and Metabolism, Molecular Biology and Genetics. According to data from OpenAlex, Roy E. Weiss has authored 194 papers receiving a total of 8.0k indexed citations (citations by other indexed papers that have themselves been cited), including 145 papers in Endocrinology, Diabetes and Metabolism, 49 papers in Molecular Biology and 48 papers in Genetics. Recurrent topics in Roy E. Weiss's work include Thyroid Disorders and Treatments (117 papers), Growth Hormone and Insulin-like Growth Factors (78 papers) and Estrogen and related hormone effects (23 papers). Roy E. Weiss is often cited by papers focused on Thyroid Disorders and Treatments (117 papers), Growth Hormone and Insulin-like Growth Factors (78 papers) and Estrogen and related hormone effects (23 papers). Roy E. Weiss collaborates with scholars based in United States, France and Belgium. Roy E. Weiss's co-authors include Samuel Refetoff, A. Hari Reddi, Alexandra M. Dumitrescu, Xiao-Hui Liao, Jessica Hwang, Joachim Pohlenz, Caterina Di Cosmo, Yoshitaka Hayashi, Mark A. Stein and Jacques Samarut and has published in prestigious journals such as Proceedings of the National Academy of Sciences, JAMA and Journal of Clinical Investigation.

In The Last Decade

Roy E. Weiss

189 papers receiving 7.8k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Roy E. Weiss United States 51 5.2k 2.3k 1.6k 700 602 194 8.0k
Michael J. Waters Australia 53 5.0k 1.0× 3.3k 1.5× 1.5k 1.0× 877 1.3× 494 0.8× 200 8.5k
Ze’ev Hochberg Israel 49 3.1k 0.6× 2.2k 1.0× 1.6k 1.0× 605 0.9× 1.1k 1.8× 242 7.4k
Graham R. Williams United Kingdom 51 5.1k 1.0× 2.6k 1.1× 1.5k 1.0× 1.1k 1.6× 546 0.9× 147 8.7k
Sally Radovick United States 50 3.0k 0.6× 2.9k 1.3× 1.8k 1.2× 793 1.1× 601 1.0× 144 7.4k
Vincent Goffin France 49 3.9k 0.8× 3.0k 1.3× 1.9k 1.2× 535 0.8× 421 0.7× 167 8.5k
Derek Le Roith United States 33 3.1k 0.6× 2.8k 1.2× 957 0.6× 789 1.1× 369 0.6× 63 6.0k
Hermann L. Müller Germany 53 5.8k 1.1× 2.1k 0.9× 598 0.4× 1.2k 1.7× 658 1.1× 340 9.3k
Kerstin Hall Sweden 47 4.8k 0.9× 2.8k 1.2× 1.7k 1.0× 853 1.2× 816 1.4× 132 7.3k
Cunming Duan United States 51 3.1k 0.6× 3.0k 1.3× 1.6k 1.0× 747 1.1× 599 1.0× 118 7.9k
Jörgen Isgaard Sweden 47 3.4k 0.7× 2.0k 0.9× 928 0.6× 724 1.0× 518 0.9× 124 6.8k

Countries citing papers authored by Roy E. Weiss

Since Specialization
Citations

This map shows the geographic impact of Roy E. Weiss's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Roy E. Weiss with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Roy E. Weiss more than expected).

Fields of papers citing papers by Roy E. Weiss

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Roy E. Weiss. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Roy E. Weiss. The network helps show where Roy E. Weiss may publish in the future.

Co-authorship network of co-authors of Roy E. Weiss

This figure shows the co-authorship network connecting the top 25 collaborators of Roy E. Weiss. A scholar is included among the top collaborators of Roy E. Weiss based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Roy E. Weiss. Roy E. Weiss is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Abdullah, Mohammed A., Samuel Refetoff, A Bertolini, et al.. (2023). Congenital Hypothyroidism in Two Sudanese Families Harboring a Novel Iodotyrosine Deiodinase Mutation (IYD R279C). Thyroid. 33(2). 261–266. 4 indexed citations
2.
Hirani, Khemraj, M. Bansinath, Rahul Mittal, et al.. (2023). Eyes on the Prize: Decoding the Ophthalmic Product Regulations and Intricacies of the U.S. Food and Drug Administration Approval. Journal of Ocular Pharmacology and Therapeutics. 39(8). 572–582. 3 indexed citations
3.
Dumitrescu, Alexandra M., et al.. (2021). Novel DIO1 Gene Mutation Acting as Phenotype Modifier for Novel Compound Heterozygous TPO Gene Mutations Causing Congenital Hypothyroidism. Thyroid. 31(10). 1589–1591. 4 indexed citations
4.
Fu, Jiao, E. Nazlı Gönç, Laura Sillers, et al.. (2020). Clinical and Molecular Analysis in 2 Families With Novel Compound Heterozygous SBP2 (SECISBP2) Mutations. The Journal of Clinical Endocrinology & Metabolism. 105(3). e6–e11. 11 indexed citations
5.
Abdullah, Mohamed Ahmed, et al.. (2019). Central Congenital Hypothyroidism Caused by a Novel Mutation, C47W, in the Cysteine Knot Region of TSHβ. Hormone Research in Paediatrics. 92(6). 390–394.
6.
Abdullah, Mohamed Ahmed, et al.. (2019). Insertion of an Alu Element in Thyroglobulin Gene as a Novel Cause of Congenital Hypothyroidism. Thyroid. 30(5). 780–782. 4 indexed citations
7.
Ferrara, Alfonso Massimiliano, Xiao-Hui Liao, Honggang Ye, et al.. (2015). The Thyroid Hormone Analog DITPA Ameliorates Metabolic Parameters of Male Mice With Mct8 Deficiency. Endocrinology. 156(11). 3889–3894. 26 indexed citations
8.
Dumitrescu, Alexandra M., et al.. (2013). Incidental Identification of a Thyroid Hormone Receptor Beta ( THRB ) Gene Variant in a Family with Autoimmune Thyroid Disease. Thyroid. 23(12). 1638–1643. 11 indexed citations
9.
Verge, Charles F., Daniel Konrad, Michal Cohen, et al.. (2012). Diiodothyropropionic Acid (DITPA) in the Treatment of MCT8 Deficiency. The Journal of Clinical Endocrinology & Metabolism. 97(12). 4515–4523. 102 indexed citations
10.
Barca‐Mayo, Olga, Xiao-Hui Liao, Manuela Alonso‐Sampedro, et al.. (2011). Thyroid Hormone Receptor α and Regulation of Type 3 Deiodinase. Molecular Endocrinology. 25(4). 575–583. 52 indexed citations
11.
Kocherginsky, Masha, et al.. (2010). Autoimmunity in Patients with Resistance to Thyroid Hormone. The Journal of Clinical Endocrinology & Metabolism. 95(7). 3189–3193. 39 indexed citations
12.
Cosmo, Caterina Di, Xiao-Hui Liao, Alexandra M. Dumitrescu, et al.. (2010). Mice deficient in MCT8 reveal a mechanism regulating thyroid hormone secretion. Journal of Clinical Investigation. 120(9). 3377–3388. 142 indexed citations
13.
Brown, Rebecca, Robert Wollman, & Roy E. Weiss. (2007). Transformation of a Pituitary Macroadenoma into to a Corticotropin-Secreting Carcinoma Over 16 Years. Endocrine Practice. 13(5). 463–471. 14 indexed citations
14.
Dumitrescu, Alexandra M., Xiao-Hui Liao, Roy E. Weiss, Kathleen J. Millen, & Samuel Refetoff. (2006). Tissue-Specific Thyroid Hormone Deprivation and Excess in Monocarboxylate Transporter (Mct) 8-Deficient Mice. Endocrinology. 147(9). 4036–4043. 260 indexed citations
15.
16.
Murphy, Esther, Sabine Costagliola, Gilbert Vassart, et al.. (2006). Congenitally hypothyroid mice with (Pax8 −/− ) or without (hyt/hyt) functional TSH receptors (TSHR) display equivalent skeletal phenotypes. 11. 1 indexed citations
17.
Weiss, Roy E., Martine Géhin, Jianming Xu, et al.. (2002). Thyroid Function in Mice with Compound Heterozygous and Homozygous Disruptions of SRC-1 and TIF-2 Coactivators: Evidence for Haploinsufficiency. Endocrinology. 143(4). 1554–1557. 32 indexed citations
18.
19.
Pohlenz, Joachim, et al.. (1998). Congenital hypothyroidism due to mutations in the sodium/iodide symporter. Identification of a nonsense mutation producing a downstream cryptic 3' splice site.. Journal of Clinical Investigation. 101(5). 1028–1035. 90 indexed citations
20.
Todd, J. R., et al.. (1971). Electrocardiograms of Hypomagnesemic Calves with or Without Development of Tetany. American Journal of Veterinary Research. 32(11). 1851–1857.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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