Robert Seed

851 total citations
18 papers, 547 citations indexed

About

Robert Seed is a scholar working on Oncology, Endocrinology, Diabetes and Metabolism and Immunology and Allergy. According to data from OpenAlex, Robert Seed has authored 18 papers receiving a total of 547 indexed citations (citations by other indexed papers that have themselves been cited), including 6 papers in Oncology, 6 papers in Endocrinology, Diabetes and Metabolism and 5 papers in Immunology and Allergy. Recurrent topics in Robert Seed's work include Cell Adhesion Molecules Research (5 papers), Pituitary Gland Disorders and Treatments (4 papers) and Cancer-related Molecular Pathways (3 papers). Robert Seed is often cited by papers focused on Cell Adhesion Molecules Research (5 papers), Pituitary Gland Disorders and Treatments (4 papers) and Cancer-related Molecular Pathways (3 papers). Robert Seed collaborates with scholars based in United Kingdom, United States and Italy. Robert Seed's co-authors include Saburo Ito, Stephen L. Nishimura, James D. Marks, Yifan Cheng, Jody L. Baron, Anthony Cormier, Jianlong Lou, Melody G. Campbell, Christopher J. McCabe and Anne T. Collins and has published in prestigious journals such as Cell, PLoS ONE and Cancer Research.

In The Last Decade

Robert Seed

18 papers receiving 542 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Robert Seed United Kingdom	13	236	192	136	113	80	18	547
Olivier Léger United Kingdom	8	228 1.0×	108 0.6×	101 0.7×	57 0.5×	54 0.7×	13	428
Melissa C Adriance United States	7	472 2.0×	380 2.0×	150 1.1×	154 1.4×	58 0.7×	7	805
Birgitte Grum-Schwensen Denmark	9	558 2.4×	199 1.0×	233 1.7×	270 2.4×	58 0.7×	9	725
Bruce D. Cohen United States	10	420 1.8×	322 1.7×	57 0.4×	127 1.1×	51 0.6×	12	810
Marie-Pierre Podgorniak France	14	364 1.5×	337 1.8×	105 0.8×	130 1.2×	52 0.7×	20	648
Kelly Foley United States	10	335 1.4×	357 1.9×	266 2.0×	124 1.1×	31 0.4×	14	695
Ioannis Roxanis United Kingdom	10	187 0.8×	306 1.6×	74 0.5×	61 0.5×	38 0.5×	18	553
Nobuaki Akakura United States	7	328 1.4×	154 0.8×	76 0.6×	311 2.8×	75 0.9×	9	596
Jun Ida Japan	8	189 0.8×	389 2.0×	241 1.8×	68 0.6×	76 0.9×	14	574
Carolyn V. Ustach United States	10	395 1.7×	165 0.9×	64 0.5×	126 1.1×	43 0.5×	11	726

Countries citing papers authored by Robert Seed

Since Specialization

Citations

This map shows the geographic impact of Robert Seed's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Robert Seed with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Robert Seed more than expected).

Fields of papers citing papers by Robert Seed

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Robert Seed. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Robert Seed. The network helps show where Robert Seed may publish in the future.

Co-authorship network of co-authors of Robert Seed

This figure shows the co-authorship network connecting the top 25 collaborators of Robert Seed. A scholar is included among the top collaborators of Robert Seed based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Robert Seed. Robert Seed is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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18 of 18 papers shown

Jin, Mingliang, Robert Seed, Guoqing Cai, et al.. (2024). Dynamic allostery drives autocrine and paracrine TGF-β signaling. Cell. 187(22). 6200–6219.e23. 20 indexed citations

Yadav, Preeti, Tatsuya Tsukui, Dean Sheppard, et al.. (2022). Macrophage Cx43 Is Necessary for Fibroblast Cytosolic Calcium and Lung Fibrosis After Injury. Frontiers in Immunology. 13. 880887–880887. 12 indexed citations

Seed, Robert, Kenji Kobayashi, Saburo Ito, et al.. (2021). A tumor-specific mechanism of T _reg enrichment mediated by the integrin αvβ8. Science Immunology. 6(57). 22 indexed citations

Seed, Robert, et al.. (2021). A Systematic Comparison of Antiandrogens Identifies Androgen Receptor Protein Stability as an Indicator for Treatment Response. Life. 11(9). 874–874. 11 indexed citations

Campbell, Melody G., Anthony Cormier, Saburo Ito, et al.. (2020). Cryo-EM Reveals Integrin-Mediated TGF-β Activation without Release from Latent TGF-β. Cell. 180(3). 490–501.e16. 114 indexed citations

Erb, Holger H.H., et al.. (2019). PIAS1 is not suitable as a urothelial carcinoma biomarker protein and pharmacological target. PLoS ONE. 14(10). e0224085–e0224085. 2 indexed citations

Seed, Robert, Alberto J. Taurozzi, Giovanna Nappo, et al.. (2019). The putative tumour suppressor protein Latexin is secreted by prostate luminal cells and is downregulated in malignancy. Scientific Reports. 9(1). 5120–5120. 15 indexed citations

Takasaka, Naoki, Robert Seed, Anthony Cormier, et al.. (2018). Integrin αvβ8–expressing tumor cells evade host immunity by regulating TGF-β activation in immune cells. JCI Insight. 3(20). 89 indexed citations

Taurozzi, Alberto J., Michelle Wantoch, Marie‐Christine Labarthe, et al.. (2017). Spontaneous development of Epstein-Barr Virus associated human lymphomas in a prostate cancer xenograft program. PLoS ONE. 12(11). e0188228–e0188228. 17 indexed citations

10.

Nappo, Giovanna, Florian Handle, Frédéric R. Santer, et al.. (2017). The immunosuppressive cytokine interleukin-4 increases the clonogenic potential of prostate stem-like cells by activation of STAT6 signalling. Oncogenesis. 6(5). e342–e342. 78 indexed citations

11.

Read, Martin L., Robert Seed, Neil Sharma, et al.. (2014). The proto‐oncogene PBF binds p53 and is associated with prognostic features in colorectal cancer. Molecular Carcinogenesis. 55(1). 15–26. 26 indexed citations

12.

Read, Martin L., Robert Seed, Rachel Watkins, et al.. (2014). The PTTG1-Binding Factor (PBF/PTTG1IP) Regulates p53 Activity in Thyroid Cells. Endocrinology. 155(4). 1222–1234. 30 indexed citations

13.

Read, Martin L., Robert Seed, Neil Sharma, et al.. (2013). Regulation of Pituitary Tumor Transforming Gene (PTTG) Expression and Phosphorylation in Thyroid Cells. Endocrinology. 154(11). 4408–4422. 10 indexed citations

14.

Smith, Vicki E., Martin L. Read, Andrew S. Turnell, et al.. (2012). PTTG-Binding Factor (PBF) Is a Novel Regulator of the Thyroid Hormone Transporter MCT8. Endocrinology. 153(7). 3526–3536. 20 indexed citations

15.

Sharma, Neil, et al.. (2012). The pituitary tumor transforming gene in thyroid cancer.. PubMed. 35(4). 425–33. 10 indexed citations

16.

Read, Martin L., Neil Sharma, Robert Seed, et al.. (2011). Proto-oncogene PBF/PTTG1IP Regulates Thyroid Cell Growth and Represses Radioiodide Treatment. Cancer Research. 71(19). 6153–6164. 40 indexed citations

17.

Forrester, Natalie, Thomas Speiseder, Peter Groitl, et al.. (2011). Adenovirus E4orf3 Targets Transcriptional Intermediary Factor 1γ for Proteasome-Dependent Degradation during Infection. Journal of Virology. 86(6). 3167–3179. 30 indexed citations

18.

Seed, Robert, et al.. (2001). The Geophysics of the Anjing Hitam Deposit: From mapping shales to a major discovery. ASEG Extended Abstracts. 2001(1). 1–4. 1 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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