Robert S. Mathias

1.0k total citations
18 papers, 699 citations indexed

About

Robert S. Mathias is a scholar working on Molecular Biology, Nephrology and Genetics. According to data from OpenAlex, Robert S. Mathias has authored 18 papers receiving a total of 699 indexed citations (citations by other indexed papers that have themselves been cited), including 7 papers in Molecular Biology, 5 papers in Nephrology and 4 papers in Genetics. Recurrent topics in Robert S. Mathias's work include Ion Transport and Channel Regulation (4 papers), Parathyroid Disorders and Treatments (3 papers) and Electrolyte and hormonal disorders (3 papers). Robert S. Mathias is often cited by papers focused on Ion Transport and Channel Regulation (4 papers), Parathyroid Disorders and Treatments (3 papers) and Electrolyte and hormonal disorders (3 papers). Robert S. Mathias collaborates with scholars based in United States, United Kingdom and Japan. Robert S. Mathias's co-authors include Stephen E. Gitelman, Anthony A. Portale, Stephen M. Rosenthal, H. H. Bengele, E. A. Alexander, Mina Matsuda‐Abedini, Brian J. Feldman, Eric A. Huang, Walter L. Miller and Gabriel Vargas and has published in prestigious journals such as New England Journal of Medicine, Journal of Biological Chemistry and PEDIATRICS.

In The Last Decade

Robert S. Mathias

18 papers receiving 682 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Robert S. Mathias United States 13 300 257 145 145 118 18 699
Katalin Dittrich Germany 14 147 0.5× 110 0.4× 64 0.4× 277 1.9× 96 0.8× 33 625
Kenichi Kano Japan 14 106 0.4× 73 0.3× 119 0.8× 111 0.8× 28 0.2× 72 574
Erum A. Hartung United States 15 238 0.8× 109 0.4× 80 0.6× 142 1.0× 185 1.6× 45 728
Annie Varennes France 12 112 0.4× 29 0.1× 83 0.6× 162 1.1× 33 0.3× 14 488
Jacques Corvilain Belgium 17 188 0.6× 164 0.6× 96 0.7× 259 1.8× 106 0.9× 82 1.1k
Ayşe Binnur Erbağcı Türkiye 11 66 0.2× 119 0.5× 105 0.7× 86 0.6× 153 1.3× 17 652
Flavia Napoli Italy 19 347 1.2× 224 0.9× 147 1.0× 30 0.2× 156 1.3× 58 997
William H. Bergstrom United States 15 153 0.5× 152 0.6× 53 0.4× 150 1.0× 130 1.1× 43 665
Diana Swolin‐Eide Sweden 15 182 0.6× 43 0.2× 107 0.7× 65 0.4× 111 0.9× 49 689
Pat Mahachoklertwattana Thailand 20 254 0.8× 65 0.3× 112 0.8× 27 0.2× 215 1.8× 82 1.2k

Countries citing papers authored by Robert S. Mathias

Since Specialization
Citations

This map shows the geographic impact of Robert S. Mathias's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Robert S. Mathias with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Robert S. Mathias more than expected).

Fields of papers citing papers by Robert S. Mathias

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Robert S. Mathias. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Robert S. Mathias. The network helps show where Robert S. Mathias may publish in the future.

Co-authorship network of co-authors of Robert S. Mathias

This figure shows the co-authorship network connecting the top 25 collaborators of Robert S. Mathias. A scholar is included among the top collaborators of Robert S. Mathias based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Robert S. Mathias. Robert S. Mathias is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

18 of 18 papers shown
1.
Falkner, Bonita, Scott W. Keith, Robert S. Mathias, et al.. (2018). Ambulatory hypertension in a pediatric cohort of sickle cell disease. Journal of the American Society of Hypertension. 12(7). 542–550. 12 indexed citations
3.
Feldman, Brian J., Stephen M. Rosenthal, Gabriel Vargas, et al.. (2005). Nephrogenic Syndrome of Inappropriate Antidiuresis. New England Journal of Medicine. 352(18). 1884–1890. 242 indexed citations
4.
Matsuda‐Abedini, Mina, et al.. (2005). Persistent secondary hyperparathyroidism after renal transplantation in children. Pediatric Nephrology. 21(3). 413–418. 8 indexed citations
5.
Gitelman, Stephen E., et al.. (2002). Increased incidence in post-transplant diabetes mellitus in children: a case-control analysis. Pediatric Nephrology. 17(1). 1–5. 61 indexed citations
6.
Bhargava, Aditi, Robert S. Mathias, James A. McCormick, Mary F. Dallman, & David Pearce. (2002). Glucocorticoids Prolong Ca2+Transients in Hippocampal-Derived H19-7 Neurons by Repressing the Plasma Membrane Ca2+-ATPase-1. Molecular Endocrinology. 16(7). 1629–1637. 34 indexed citations
7.
Mingin, Gerald C., Hiep T. Nguyen, Robert S. Mathias, et al.. (2002). Growth and Metabolic Consequences of Bladder Augmentation in Children With Myelomeningocele and Bladder Exstrophy. PEDIATRICS. 110(6). 1193–1198. 47 indexed citations
8.
Mathias, Robert S., et al.. (2001). Identification of the Calcium-Sensing Receptor in the Developing Tooth Organ. Journal of Bone and Mineral Research. 16(12). 2238–2244. 27 indexed citations
9.
Mathias, Robert S., et al.. (2000). Increased fluoride content in the femur growth plate and cortical bone of uremic rats. Pediatric Nephrology. 14(10-11). 935–939. 3 indexed citations
10.
Mathias, Robert S.. (2000). Rickets in an infant with Williams syndrome. Pediatric Nephrology. 14(6). 489–492. 13 indexed citations
11.
Mathias, Robert S., et al.. (1998). Reduced expression of the renal calcium-sensing receptor in rats with experimental chronic renal insufficiency.. Journal of the American Society of Nephrology. 9(11). 2067–2074. 32 indexed citations
12.
Mathias, Robert S., Katsuhiko Mikoshiba, Takayuki Michikawa, Atsushi Miyawaki, & Harlan E. Ives. (1998). IP3receptor blockade fails to prevent intracellular Ca2+release by ET-1 and α-thrombin. American Journal of Physiology-Cell Physiology. 274(6). C1456–C1465. 11 indexed citations
13.
Mathias, Robert S., et al.. (1997). Non-capacitative Calcium Entry in Chinese Hamster Ovary Cells Expressing the Platelet-derived Growth Factor Receptor. Journal of Biological Chemistry. 272(46). 29076–29082. 11 indexed citations
14.
Mathias, Robert S.. (1997). Chronic anemia as a complication of parvovirus B19 infection in a pediatric kidney transplant patient. Pediatric Nephrology. 11(3). 355–357. 22 indexed citations
15.
Uyeki, Timothy M., et al.. (1993). Successful treatment with hydrochlorothiazide and amiloride in an infant with congenital nephrogenic diabetes insipidus. Pediatric Nephrology. 7(5). 554–556. 6 indexed citations
16.
Mathias, Robert S. & Eric Brown. (1991). Divalent Cations Modulate PTH-Dependent 3′,5′-Cyclic Adenosine Monophosphate Production in Renal Proximal Tubular Cells*. Endocrinology. 128(6). 3005–3012. 17 indexed citations
17.
Bengele, H. H., Robert S. Mathias, & E. A. Alexander. (1981). Impaired Natriuresis after Volume Expansion in the Aged Rat. Kidney & Blood Pressure Research. 4(1). 22–29. 23 indexed citations
18.
Bengele, H. H., Robert S. Mathias, John H. Perkins, & E. A. Alexander. (1981). Urinary concentrating defect in the aged rat. American Journal of Physiology-Renal Physiology. 240(2). F147–F150. 63 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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