Robert J. Vincent

934 total citations
14 papers, 492 citations indexed

About

Robert J. Vincent is a scholar working on Molecular Biology, Cardiology and Cardiovascular Medicine and Surgery. According to data from OpenAlex, Robert J. Vincent has authored 14 papers receiving a total of 492 indexed citations (citations by other indexed papers that have themselves been cited), including 5 papers in Molecular Biology, 5 papers in Cardiology and Cardiovascular Medicine and 4 papers in Surgery. Recurrent topics in Robert J. Vincent's work include Cardiac Fibrosis and Remodeling (5 papers), Mesenchymal stem cell research (4 papers) and Tissue Engineering and Regenerative Medicine (4 papers). Robert J. Vincent is often cited by papers focused on Cardiac Fibrosis and Remodeling (5 papers), Mesenchymal stem cell research (4 papers) and Tissue Engineering and Regenerative Medicine (4 papers). Robert J. Vincent collaborates with scholars based in United States, China and Poland. Robert J. Vincent's co-authors include Buddhadeb Dawn, Magdy Girgis, Anweshan Samanta, Lin Zhao, Yu‐Ting Xuan, Michael Wacker, Yanjuan Yang, Chad D. Touchberry, Lynda F. Bonewald and Guangming Cheng and has published in prestigious journals such as Circulation, Journal of the American College of Cardiology and PLoS ONE.

In The Last Decade

Robert J. Vincent

14 papers receiving 488 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Robert J. Vincent United States 7 249 194 114 87 69 14 492
Wanling Xuan United States 14 266 1.1× 187 1.0× 71 0.6× 76 0.9× 42 0.6× 26 566
Magdy Girgis United States 6 190 0.8× 198 1.0× 113 1.0× 54 0.6× 62 0.9× 11 422
Yoshiki Akakabe Japan 10 155 0.6× 144 0.7× 67 0.6× 77 0.9× 44 0.6× 18 429
Mar Orriols Spain 14 285 1.1× 139 0.7× 31 0.3× 95 1.1× 70 1.0× 20 635
Mariko Fukui Japan 12 197 0.8× 162 0.8× 235 2.1× 34 0.4× 65 0.9× 25 583
Toshihiro Amaki Japan 8 143 0.6× 53 0.3× 216 1.9× 70 0.8× 135 2.0× 10 477
Maki Uraoka Japan 6 168 0.7× 62 0.3× 123 1.1× 45 0.5× 78 1.1× 6 407
Sandhya S. Thomas United States 13 316 1.3× 62 0.3× 162 1.4× 73 0.8× 30 0.4× 20 591
H Nihei Japan 10 242 1.0× 44 0.2× 160 1.4× 41 0.5× 29 0.4× 17 428
Brigitte Dautréaux France 13 232 0.9× 132 0.7× 43 0.4× 117 1.3× 20 0.3× 17 501

Countries citing papers authored by Robert J. Vincent

Since Specialization
Citations

This map shows the geographic impact of Robert J. Vincent's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Robert J. Vincent with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Robert J. Vincent more than expected).

Fields of papers citing papers by Robert J. Vincent

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Robert J. Vincent. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Robert J. Vincent. The network helps show where Robert J. Vincent may publish in the future.

Co-authorship network of co-authors of Robert J. Vincent

This figure shows the co-authorship network connecting the top 25 collaborators of Robert J. Vincent. A scholar is included among the top collaborators of Robert J. Vincent based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Robert J. Vincent. Robert J. Vincent is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

14 of 14 papers shown
1.
Chen, Lei, Anweshan Samanta, Lin Zhao, et al.. (2021). Vitamin D3 induces mesenchymal-to-endothelial transition and promotes a proangiogenic niche through IGF-1 signaling. iScience. 24(4). 102272–102272. 10 indexed citations
2.
Zhao, Lin, Guangming Cheng, Kashyap Choksi, et al.. (2019). Transplantation of Human Umbilical Cord Blood–Derived Cellular Fraction Improves Left Ventricular Function and Remodeling After Myocardial Ischemia/Reperfusion. Circulation Research. 125(8). 759–772. 11 indexed citations
3.
Chen, Lei, Lin Zhao, Anweshan Samanta, et al.. (2017). STAT3 balances myocyte hypertrophy vis-à-vis autophagy in response to Angiotensin II by modulating the AMPKα/mTOR axis. PLoS ONE. 12(7). e0179835–e0179835. 25 indexed citations
4.
Chen, Xing, Guangming Cheng, Shiming Liu, et al.. (2016). PRESSURE-OVERLOAD INDUCED LV HYPERTROPHY AND DYSFUNCTION: CRITICAL ROLES OF CAMKII AND P38 MAP KINASE IN ER STRESS SIGNALING PATHWAY. Journal of the American College of Cardiology. 67(13). 1403–1403. 1 indexed citations
5.
Zhao, Lin, Guangming Cheng, Runming Jin, et al.. (2016). Deletion of Interleukin-6 Attenuates Pressure Overload-Induced Left Ventricular Hypertrophy and Dysfunction. Circulation Research. 118(12). 1918–1929. 202 indexed citations
6.
Zhao, Lin, Guangming Cheng, Runming Jin, et al.. (2015). CAMKII IS CRITICAL FOR THE UPREGULATION OF STAT3 SIGNALING IN PATHOGENESIS OF CARDIOMYOCYTE HYPERTROPHY. Journal of the American College of Cardiology. 65(10). A903–A903. 1 indexed citations
7.
Cazanave, Sophie C., Robert J. Vincent, Jyoti Srivastava, et al.. (2015). P0953 : FXR resistance characterizes human and mouse model of NASH. Journal of Hepatology. 62. S703–S703. 1 indexed citations
8.
Cheng, Guangming, Dominika Berdecka, Yu‐Ting Xuan, et al.. (2015). Abstract 16673: Intramyocardial Injection of MSC-derived Extracellular Vesicles Confers Superior Cardiac Repair After a Reperfused Myocardial Infarction. Circulation. 132(suppl_3). 1 indexed citations
9.
Samanta, Anweshan, Guangming Cheng, Arash Davani, et al.. (2014). Abstract 15693: Genetic Deletion of Interleukin-6 Attenuates Left Ventricular Dysfunction and Remodeling After a Reperfused Myocardial Infarction. Circulation. 130. 1 indexed citations
10.
Cheng, Guangming, Lin Zhao, Lei Chen, et al.. (2014). Abstract 15923: CaMKII Links ER Stress Signaling in Pressure Overload-Induced Left Ventricular Hypertrophy and Failure. Circulation. 130(suppl_2). 1 indexed citations
11.
Touchberry, Chad D., Magdy Girgis, Robert J. Vincent, et al.. (2013). FGF23 is a novel regulator of intracellular calcium and cardiac contractility in addition to cardiac hypertrophy. American Journal of Physiology-Endocrinology and Metabolism. 304(8). E863–E873. 156 indexed citations
12.
Zuba‐Surma, Ewa, Yiru Guo, Santosh K. Sanganalmath, et al.. (2010). Transplantation of expanded bone marrow‐derived very small embryonic‐like stem cells (VSEL‐SCs) improves left ventricular function and remodelling after myocardial infarction. Journal of Cellular and Molecular Medicine. 15(6). 1319–1328. 63 indexed citations
13.
Zuba‐Surma, Ewa, Ahmed Abdel‐Latif, Robert J. Vincent, et al.. (2006). Abstract 937: Antigenically-Defined Subsets of Bone Marrow Mesenchymal Stem Cells Exhibit Differential Cardiomyogenic and Angiogenic Potential. Circulation. 114. 1 indexed citations
14.
Zuba‐Surma, Ewa, Ahmed Abdel‐Latif, Jamie Case, et al.. (2006). Sca-1 expression is associated with decreased cardiomyogenic differentiation potential of skeletal muscle-derived adult primitive cells. Journal of Molecular and Cellular Cardiology. 41(4). 650–660. 18 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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