Robert J. Goodrow

1.6k total citations
27 papers, 1.2k citations indexed

About

Robert J. Goodrow is a scholar working on Cardiology and Cardiovascular Medicine, Molecular Biology and Cellular and Molecular Neuroscience. According to data from OpenAlex, Robert J. Goodrow has authored 27 papers receiving a total of 1.2k indexed citations (citations by other indexed papers that have themselves been cited), including 26 papers in Cardiology and Cardiovascular Medicine, 24 papers in Molecular Biology and 10 papers in Cellular and Molecular Neuroscience. Recurrent topics in Robert J. Goodrow's work include Cardiac electrophysiology and arrhythmias (25 papers), Ion channel regulation and function (20 papers) and Neuroscience and Neural Engineering (8 papers). Robert J. Goodrow is often cited by papers focused on Cardiac electrophysiology and arrhythmias (25 papers), Ion channel regulation and function (20 papers) and Neuroscience and Neural Engineering (8 papers). Robert J. Goodrow collaborates with scholars based in United States, Denmark and Germany. Robert J. Goodrow's co-authors include Charles Antzelevitch, Andrew C. Zygmunt, Jonathan M. Cordeiro, José M. Di Diego, Guillermo J. Pérez, Jeffrey Fish, Lawrence B. Cohen, Jorge M. Davidenko, Fabiana S. Scornik and Jacqueline A. Treat and has published in prestigious journals such as Circulation, PLoS ONE and International Journal of Molecular Sciences.

In The Last Decade

Robert J. Goodrow

26 papers receiving 1.2k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Robert J. Goodrow United States	15	1.1k	922	316	52	41	27	1.2k
Klaus Schlotthauer United States	7	1.1k 1.0×	884 1.0×	264 0.8×	34 0.7×	38 0.9×	7	1.2k
Antony J. Workman United Kingdom	19	1.1k 1.0×	489 0.5×	152 0.5×	28 0.5×	37 0.9×	33	1.2k
Massimiliano Zaniboni Italy	15	502 0.5×	443 0.5×	157 0.5×	38 0.7×	25 0.6×	32	670
M R Rosen United States	16	820 0.8×	574 0.6×	239 0.8×	59 1.1×	46 1.1×	30	1.0k
J. Toyama Japan	17	571 0.5×	407 0.4×	196 0.6×	26 0.5×	31 0.8×	46	720
Roel L. H. M. G. Spätjens Netherlands	12	864 0.8×	682 0.7×	147 0.5×	43 0.8×	19 0.5×	18	908
Constanze Schmidt Germany	19	525 0.5×	475 0.5×	129 0.4×	20 0.4×	39 1.0×	64	810
Francien J. G. Wilms-Schopman Netherlands	14	1.1k 1.1×	638 0.7×	102 0.3×	25 0.5×	27 0.7×	17	1.2k
Péter Schäffer Austria	16	369 0.3×	299 0.3×	197 0.6×	23 0.4×	40 1.0×	38	629
S S Sheu United States	9	485 0.5×	587 0.6×	311 1.0×	71 1.4×	16 0.4×	10	788

Countries citing papers authored by Robert J. Goodrow

Since Specialization

Citations

This map shows the geographic impact of Robert J. Goodrow's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Robert J. Goodrow with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Robert J. Goodrow more than expected).

Fields of papers citing papers by Robert J. Goodrow

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Robert J. Goodrow. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Robert J. Goodrow. The network helps show where Robert J. Goodrow may publish in the future.

Co-authorship network of co-authors of Robert J. Goodrow

This figure shows the co-authorship network connecting the top 25 collaborators of Robert J. Goodrow. A scholar is included among the top collaborators of Robert J. Goodrow based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Robert J. Goodrow. Robert J. Goodrow is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Treat, Jacqueline A., Ryan Pfeiffer, Robert J. Goodrow, et al.. (2024). Identification and characterization of two novel KCNH2 mutations contributing to long QT syndrome. PLoS ONE. 19(1). e0287206–e0287206.

Treat, Jacqueline A., Ryan Pfeiffer, Héctor Barajas-Martínez, et al.. (2021). Overlap Arrhythmia Syndromes Resulting from Multiple Genetic Variations Studied in Human Induced Pluripotent Stem Cell-Derived Cardiomyocytes. International Journal of Molecular Sciences. 22(13). 7108–7108. 5 indexed citations

Marszalec, William, Shin Yoo, Gary L. Aistrup, et al.. (2020). Triggered Ca ²⁺ Waves Induce Depolarization of Maximum Diastolic Potential and Action Potential Prolongation in Dog Atrial Myocytes. Circulation Arrhythmia and Electrophysiology. 13(6). e008179–e008179. 7 indexed citations

Barajas-Martínez, Héctor, Dan Hu, Robert J. Goodrow, et al.. (2020). Susceptibility to Ventricular Arrhythmias Resulting from Mutations in FKBP1B, PXDNL, and SCN9A Evaluated in hiPSC Cardiomyocytes. Stem Cells International. 2020. 1–16. 14 indexed citations

Treat, Jacqueline A., et al.. (2019). Pharmacological enhancement of repolarization reserve in human induced pluripotent stem cells derived cardiomyocytes. Biochemical Pharmacology. 169. 113608–113608. 10 indexed citations

Calløe, Kirstine, Gary L. Aistrup, José M. Di Diego, et al.. (2018). Interventricular differences in sodium current and its potential role in Brugada syndrome. Physiological Reports. 6(14). e13787–e13787. 13 indexed citations

Goodrow, Robert J., Jacqueline A. Treat, Brian K. Panama, et al.. (2017). Biophysical comparison of sodium currents in native cardiac myocytes and human induced pluripotent stem cell-derived cardiomyocytes. Journal of Pharmacological and Toxicological Methods. 90. 19–30. 18 indexed citations

Barajas-Martínez, Héctor, Robert J. Goodrow, Dan Hu, et al.. (2017). Biophysical and molecular comparison of sodium current in cells isolated from canine atria and pulmonary vein. Pflügers Archiv - European Journal of Physiology. 469(5-6). 703–712. 9 indexed citations

Cordeiro, Jonathan M., Robert J. Goodrow, Aaron D. Kaplan, et al.. (2015). Regional variation of the inwardly rectifying potassium current in the canine heart and the contributions to differences in action potential repolarization. Journal of Molecular and Cellular Cardiology. 84. 52–60. 23 indexed citations

10.

Burashnikov, Alexander, José M. Di Diego, Robert J. Goodrow, Luiz Belardinelli, & Charles Antzelevitch. (2015). Atria are More Sensitive Than Ventricles to GS-458967-Induced Inhibition of Late Sodium Current. Journal of Cardiovascular Pharmacology and Therapeutics. 20(5). 501–508. 15 indexed citations

11.

Panama, Brian K., et al.. (2013). Developmental Changes in Potassium Channel Expression in the Canine Heart: Implications for Sudden Infant Death Caused by Arrhythmias. Biophysical Journal. 104(2). 295a–295a. 1 indexed citations

12.

Cordeiro, Jonathan M., Vladislav V. Nesterenko, Serge Sicouri, et al.. (2013). Identification and characterization of a transient outward K+ current in human induced pluripotent stem cell-derived cardiomyocytes. Journal of Molecular and Cellular Cardiology. 60. 36–46. 52 indexed citations

13.

Calløe, Kirstine, Robert J. Goodrow, Søren‐Peter Olesen, Charles Antzelevitch, & Jonathan M. Cordeiro. (2013). Tissue-specific effects of acetylcholine in the canine heart. American Journal of Physiology-Heart and Circulatory Physiology. 305(1). H66–H75. 20 indexed citations

14.

Cordeiro, Jonathan M., Brian K. Panama, Robert J. Goodrow, et al.. (2013). Developmental changes in expression and biophysics of ion channels in the canine ventricle. Journal of Molecular and Cellular Cardiology. 64. 79–89. 14 indexed citations

15.

Doss, Michael Xavier, José M. Di Diego, Robert J. Goodrow, et al.. (2012). Maximum Diastolic Potential of Human Induced Pluripotent Stem Cell-Derived Cardiomyocytes Depends Critically on IKr. PLoS ONE. 7(7). e40288–e40288. 127 indexed citations

16.

Cordeiro, Jonathan M., et al.. (2008). Functionally distinct sodium channels in ventricular epicardial and endocardial cells contribute to a greater sensitivity of the epicardium to electrical depression. American Journal of Physiology-Heart and Circulatory Physiology. 295(1). H154–H162. 41 indexed citations

17.

Sicouri, Serge, Katherine W. Timothy, Andrew C. Zygmunt, et al.. (2007). Cellular basis for the electrocardiographic and arrhythmic manifestations of Timothy syndrome: Effects of ranolazine. Heart Rhythm. 4(5). 638–647. 65 indexed citations

18.

Diego, José M. Di, Jonathan M. Cordeiro, Robert J. Goodrow, et al.. (2002). Ionic and Cellular Basis for the Predominance of the Brugada Syndrome Phenotype in Males. Circulation. 106(15). 2004–2011. 263 indexed citations

19.

Antzelevitch, Charles, Jorge M. Davidenko, Serge Sicouri, et al.. (1989). Quinidine‐Induced Early Afterdepolarizations and Triggered Activity. 3(5). 323–338. 13 indexed citations

20.

Davidenko, Jorge M., Lawrence B. Cohen, Robert J. Goodrow, & Charles Antzelevitch. (1989). Quinidine-induced action potential prolongation, early afterdepolarizations, and triggered activity in canine Purkinje fibers. Effects of stimulation rate, potassium, and magnesium.. Circulation. 79(3). 674–686. 157 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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