Robert H. Scannevin

10.5k total citations · 1 hit paper
59 papers, 5.6k citations indexed

About

Robert H. Scannevin is a scholar working on Molecular Biology, Cellular and Molecular Neuroscience and Oncology. According to data from OpenAlex, Robert H. Scannevin has authored 59 papers receiving a total of 5.6k indexed citations (citations by other indexed papers that have themselves been cited), including 37 papers in Molecular Biology, 13 papers in Cellular and Molecular Neuroscience and 11 papers in Oncology. Recurrent topics in Robert H. Scannevin's work include Genomics, phytochemicals, and oxidative stress (12 papers), Neuroinflammation and Neurodegeneration Mechanisms (9 papers) and Multiple Sclerosis Research Studies (8 papers). Robert H. Scannevin is often cited by papers focused on Genomics, phytochemicals, and oxidative stress (12 papers), Neuroinflammation and Neurodegeneration Mechanisms (9 papers) and Multiple Sclerosis Research Studies (8 papers). Robert H. Scannevin collaborates with scholars based in United States, Germany and Italy. Robert H. Scannevin's co-authors include Richard L. Huganir, Kenneth J. Rhodes, James S. Trimmer, Jun Xia, Hee Jung Chung, Xiaoqun Zhang, Matvey Lukashev, Pradeep Bista, Sarah Ryan and Sowmya Chollate and has published in prestigious journals such as Cell, Proceedings of the National Academy of Sciences and Nature Communications.

In The Last Decade

Robert H. Scannevin

58 papers receiving 5.5k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Fumaric acid esters exert neuroprotective effects in neuroinflammation via activation of the Nrf2 antioxidant pathway

2011 892 citations Sarah Ryan, Pradeep Bista et al. profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Robert H. Scannevin United States	30	3.3k	1.6k	877	747	702	59	5.6k
Shobu Namura Japan	28	2.7k 0.8×	1.0k 0.6×	1.1k 1.2×	357 0.5×	442 0.6×	41	4.7k
Emanuela Bonfoco United States	17	3.2k 1.0×	1.5k 0.9×	546 0.6×	300 0.4×	674 1.0×	19	5.3k
Axel Methner Germany	37	2.4k 0.7×	914 0.6×	369 0.4×	309 0.4×	319 0.5×	87	4.4k
Marı́a Domercq Spain	35	1.6k 0.5×	2.3k 1.4×	2.4k 2.7×	493 0.7×	566 0.8×	55	5.7k
Marina Pizzi Italy	42	2.1k 0.7×	1.8k 1.1×	1.2k 1.4×	235 0.3×	653 0.9×	121	5.3k
Karl‐Norbert Klotz Germany	45	4.9k 1.5×	2.0k 1.2×	549 0.6×	299 0.4×	727 1.0×	193	9.2k
Juana M. Pasquini Argentina	36	2.2k 0.7×	784 0.5×	941 1.1×	336 0.4×	468 0.7×	134	4.7k
Jan Lewerenz Germany	29	1.9k 0.6×	950 0.6×	520 0.6×	287 0.4×	272 0.4×	79	4.5k
Benjamin Drukarch Netherlands	43	1.9k 0.6×	1.6k 1.0×	1.1k 1.3×	336 0.4×	377 0.5×	143	5.4k
Weihai Ying China	38	2.7k 0.8×	819 0.5×	815 0.9×	317 0.4×	400 0.6×	109	6.1k

Countries citing papers authored by Robert H. Scannevin

Since Specialization

Citations

This map shows the geographic impact of Robert H. Scannevin's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Robert H. Scannevin with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Robert H. Scannevin more than expected).

Fields of papers citing papers by Robert H. Scannevin

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Robert H. Scannevin. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Robert H. Scannevin. The network helps show where Robert H. Scannevin may publish in the future.

Co-authorship network of co-authors of Robert H. Scannevin

This figure shows the co-authorship network connecting the top 25 collaborators of Robert H. Scannevin. A scholar is included among the top collaborators of Robert H. Scannevin based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Robert H. Scannevin. Robert H. Scannevin is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

Sort: Min cites: Since: Top N: Style:

20 of 20 papers shown

Scannevin, Robert H., et al.. (2025). Fatty acids as potential biomarkers of stearoyl-CoA desaturase inhibition: Variation in healthy subjects and Parkinson's disease patients. SHILAP Revista de lepidopterología. 13. 100132–100132. 1 indexed citations

Burke, Christopher J., Xin Jiang, Ping Xu, et al.. (2022). Patient-derived three-dimensional cortical neurospheres to model Parkinson’s disease. PLoS ONE. 17(12). e0277532–e0277532. 6 indexed citations

Nuber, Silke, Chee Yeun Chung, Daniel F. Tardiff, et al.. (2022). A Brain-Penetrant Stearoyl-CoA Desaturase Inhibitor Reverses α-Synuclein Toxicity. Neurotherapeutics. 19(3). 1018–1036. 11 indexed citations

Nelson, J. Daniel, Takayuki Uozumi, Yasujiro Kiyota, et al.. (2022). Automated algorithm development to assess survival of human neurons using longitudinal single-cell tracking: Application to synucleinopathy. SLAS TECHNOLOGY. 28(2). 63–69.

Brennan, Melanie S., Maria F. Matos, K. Richter, Bing Li, & Robert H. Scannevin. (2017). The NRF2 transcriptional target, OSGIN1, contributes to monomethyl fumarate-mediated cytoprotection in human astrocytes. Scientific Reports. 7(1). 42054–42054. 60 indexed citations

Peng, Haiyan, et al.. (2016). Dimethyl fumarate alters microglia phenotype and protects neurons against proinflammatory toxic microenvironments. Journal of Neuroimmunology. 299. 35–44. 44 indexed citations

Lastres‐Becker, Isabel, Ángel J. García‐Yagüe, Robert H. Scannevin, et al.. (2016). Repurposing the NRF2 Activator Dimethyl Fumarate as Therapy Against Synucleinopathy in Parkinson's Disease. Antioxidants and Redox Signaling. 25(2). 61–77. 227 indexed citations

Peng, Haiyan, Maria Auxiliadora Costa Matos, Brian T. Wipke, H. Moore Arnold, & Robert H. Scannevin. (2015). Dimethyl Fumarate Alters Microglia Phenotype and Protects Neurons Against Proinflammatory Toxic Microenvironments (P1.163). Neurology. 84(14_supplement). 2 indexed citations

Parodi, Benedetta, Silvia Rossi, Sara Morando, et al.. (2015). Fumarates modulate microglia activation through a novel HCAR2 signaling pathway and rescue synaptic dysregulation in inflamed CNS. Acta Neuropathologica. 130(2). 279–295. 156 indexed citations

10.

Brennan, Melanie S., Maria F. Matos, Bing Li, et al.. (2015). Dimethyl Fumarate and Monoethyl Fumarate Exhibit Differential Effects on KEAP1, NRF2 Activation, and Glutathione Depletion In Vitro. PLoS ONE. 10(3). e0120254–e0120254. 137 indexed citations

11.

Gillard, Geoffrey O., Brian Collette, John E. Anderson, et al.. (2015). DMF, but not other fumarates, inhibits NF-κB activity in vitro in an Nrf2-independent manner. Journal of Neuroimmunology. 283. 74–85. 147 indexed citations

12.

Scannevin, Robert H., Sowmya Chollate, Mi-Young Jung, et al.. (2012). Fumarates Promote Cytoprotection of Central Nervous System Cells against Oxidative Stress via the Nuclear Factor (Erythroid-Derived 2)-Like 2 Pathway. Journal of Pharmacology and Experimental Therapeutics. 341(1). 274–284. 380 indexed citations

13.

Wu, Jing, Ronald S. Petralia, Hiral Patel, et al.. (2011). Arc/Arg3.1 Regulates an Endosomal Pathway Essential for Activity-Dependent β-Amyloid Generation. Cell. 147(3). 615–628. 168 indexed citations

14.

Wilson, Lawrence J., Bingbing Wang, Robert H. Scannevin, et al.. (2011). Discovery of novel Cobactin-T based matrix metalloproteinase inhibitors via a ring closing metathesis strategy. Bioorganic & Medicinal Chemistry Letters. 21(21). 6485–6490. 7 indexed citations

15.

Lundgren, Karen, Hong Zhang, Nanni Huser, et al.. (2009). BIIB021, an orally available, fully synthetic small-molecule inhibitor of the heat shock protein Hsp90. Molecular Cancer Therapeutics. 8(4). 921–929. 140 indexed citations

16.

Murata, Yoshihiro, Anna Rosell, Robert H. Scannevin, et al.. (2008). Extension of the Thrombolytic Time Window With Minocycline in Experimental Stroke. Stroke. 39(12). 3372–3377. 183 indexed citations

17.

Scannevin, Robert H., Bingbing Wang, Sharon L. Burke, et al.. (2007). β-N-Biaryl ether sulfonamide hydroxamates as potent gelatinase inhibitors: Part 1. Design, synthesis, and lead identification. Bioorganic & Medicinal Chemistry Letters. 18(3). 1135–1139. 24 indexed citations

18.

Zhang, Yuemei, et al.. (2007). Syntheses and in vitro evaluation of arylsulfone-based MMP inhibitors with heterocycle-derived zinc-binding groups (ZBGs). Bioorganic & Medicinal Chemistry Letters. 18(1). 405–408. 36 indexed citations

19.

Liao, Dezhi, Robert H. Scannevin, & Richard L. Huganir. (2001). Activation of Silent Synapses by Rapid Activity-Dependent Synaptic Recruitment of AMPA Receptors. Journal of Neuroscience. 21(16). 6008–6017. 235 indexed citations

20.

Lim, Seung T., Dana Antonucci-Durgan, Robert H. Scannevin, & James S. Trimmer. (2000). A Novel Targeting Signal for Proximal Clustering of the Kv2.1 K+ Channel in Hippocampal Neurons. Neuron. 25(2). 385–397. 227 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

Explore authors with similar magnitude of impact