Robert E. Blair

2.5k total citations
39 papers, 2.0k citations indexed

About

Robert E. Blair is a scholar working on Cellular and Molecular Neuroscience, Molecular Biology and Pharmacology. According to data from OpenAlex, Robert E. Blair has authored 39 papers receiving a total of 2.0k indexed citations (citations by other indexed papers that have themselves been cited), including 29 papers in Cellular and Molecular Neuroscience, 16 papers in Molecular Biology and 12 papers in Pharmacology. Recurrent topics in Robert E. Blair's work include Neuroscience and Neuropharmacology Research (29 papers), Cannabis and Cannabinoid Research (11 papers) and Ion channel regulation and function (11 papers). Robert E. Blair is often cited by papers focused on Neuroscience and Neuropharmacology Research (29 papers), Cannabis and Cannabinoid Research (11 papers) and Ion channel regulation and function (11 papers). Robert E. Blair collaborates with scholars based in United States, India and United Kingdom. Robert E. Blair's co-authors include Robert J. DeLorenzo, Laxmikant S. Deshpande, Sompong Sombati, Billy R. Martin, Dawn S. Carter, William C. Taft, Melisa J. Wallace, Guy L. Clifton, Severn B. Churn and Mohsin Raza and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Stroke and Brain Research.

In The Last Decade

Robert E. Blair

39 papers receiving 2.0k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Robert E. Blair United States 25 1.3k 766 572 426 322 39 2.0k
Laxmikant S. Deshpande United States 25 1.1k 0.8× 495 0.6× 639 1.1× 636 1.5× 206 0.6× 56 2.1k
Maria F. M. Braga United States 33 1.2k 0.9× 387 0.5× 774 1.4× 288 0.7× 519 1.6× 68 2.5k
Lynn Wecker United States 31 920 0.7× 367 0.5× 1.0k 1.8× 133 0.3× 217 0.7× 86 2.3k
María A. Mena Spain 31 1.1k 0.8× 305 0.4× 634 1.1× 140 0.3× 91 0.3× 56 2.6k
Márcio R. Martins Brazil 21 606 0.5× 126 0.2× 307 0.5× 672 1.6× 247 0.8× 39 1.8k
M. Ruth Pazos Spain 27 1.5k 1.1× 2.4k 3.1× 426 0.7× 86 0.2× 516 1.6× 49 3.0k
Lih‐Chu Chiou Taiwan 28 1.0k 0.8× 341 0.4× 839 1.5× 250 0.6× 646 2.0× 97 2.8k
Erica J. Carrier United States 22 1.6k 1.2× 2.6k 3.4× 415 0.7× 125 0.3× 601 1.9× 31 3.5k
Yukihiro Ohno Japan 32 2.0k 1.5× 334 0.4× 1.2k 2.2× 874 2.1× 206 0.6× 129 3.3k
Toivo Halonen Finland 29 1.4k 1.1× 193 0.3× 582 1.0× 969 2.3× 412 1.3× 74 2.5k

Countries citing papers authored by Robert E. Blair

Since Specialization
Citations

This map shows the geographic impact of Robert E. Blair's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Robert E. Blair with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Robert E. Blair more than expected).

Fields of papers citing papers by Robert E. Blair

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Robert E. Blair. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Robert E. Blair. The network helps show where Robert E. Blair may publish in the future.

Co-authorship network of co-authors of Robert E. Blair

This figure shows the co-authorship network connecting the top 25 collaborators of Robert E. Blair. A scholar is included among the top collaborators of Robert E. Blair based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Robert E. Blair. Robert E. Blair is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Blair, Robert E., et al.. (2023). Chronic Epilepsy and Mossy Fiber Sprouting Following Organophosphate-Induced Status Epilepticus in Rats. Journal of Pharmacology and Experimental Therapeutics. 388(2). 325–332. 8 indexed citations
2.
Deshpande, Laxmikant S., et al.. (2016). Pharmacological blockade of the calcium plateau provides neuroprotection following organophosphate paraoxon induced status epilepticus in rats. Neurotoxicology and Teratology. 56. 81–86. 18 indexed citations
3.
Blair, Robert E., Laxmikant S. Deshpande, & Robert J. DeLorenzo. (2015). Cannabinoids: is there a potential treatment role in epilepsy?. Expert Opinion on Pharmacotherapy. 16(13). 1911–1914. 23 indexed citations
4.
5.
Deshpande, Laxmikant S., Robert E. Blair, & Robert J. DeLorenzo. (2011). Prolonged cannabinoid exposure alters GABAA receptor mediated synaptic function in cultured hippocampal neurons. Experimental Neurology. 229(2). 264–273. 20 indexed citations
6.
Deshpande, Laxmikant S., Dawn S. Carter, Robert E. Blair, & Robert J. DeLorenzo. (2010). Development of a Prolonged Calcium Plateau in Hippocampal Neurons in Rats Surviving Status Epilepticus Induced by the Organophosphate Diisopropylfluorophosphate. Toxicological Sciences. 116(2). 623–631. 96 indexed citations
7.
8.
Blair, Robert E., Sompong Sombati, Severn B. Churn, & Robert J. DeLorenzo. (2008). Epileptogenesis causes an N-methyl-d-aspartate receptor/Ca2+-dependent decrease in Ca2+/calmodulin-dependent protein kinase II activity in a hippocampal neuronal culture model of spontaneous recurrent epileptiform discharges. European Journal of Pharmacology. 588(1). 64–71. 25 indexed citations
9.
Carter, Dawn S., et al.. (2008). Long-term decrease in calbindin-D28K expression in the hippocampus of epileptic rats following pilocarpine-induced status epilepticus. Epilepsy Research. 79(2-3). 213–223. 33 indexed citations
10.
Deshpande, Laxmikant S., et al.. (2007). In vitro status epilepticus but not spontaneous recurrent seizures cause cell death in cultured hippocampal neurons. Epilepsy Research. 75(2-3). 171–179. 26 indexed citations
11.
12.
DeLorenzo, Robert J., David Sun, Robert E. Blair, & Sompong Sombati. (2007). An in vitro model of Stroke‐Induced Epilepsy: Elucidation of The roles of Glutamate and Calcium in The induction and Maintenance of Stroke‐Induced Epileptogenesis. International review of neurobiology. 81. 59–84. 33 indexed citations
13.
Blair, Robert E., et al.. (2006). Activation of the Cannabinoid Type-1 Receptor Mediates the Anticonvulsant Properties of Cannabinoids in the Hippocampal Neuronal Culture Models of Acquired Epilepsy and Status Epilepticus. Journal of Pharmacology and Experimental Therapeutics. 317(3). 1072–1078. 111 indexed citations
14.
Carter, Dawn S., Sajjad Haider, Robert E. Blair, et al.. (2006). Altered Calcium/Calmodulin Kinase II Activity Changes Calcium Homeostasis That Underlies Epileptiform Activity in Hippocampal Neurons in Culture. Journal of Pharmacology and Experimental Therapeutics. 319(3). 1021–1031. 22 indexed citations
15.
Wallace, Melisa J., et al.. (2003). The Endogenous Cannabinoid System Regulates Seizure Frequency and Duration in a Model of Temporal Lobe Epilepsy. Journal of Pharmacology and Experimental Therapeutics. 307(1). 129–137. 309 indexed citations
16.
Sun, David, Sompong Sombati, Robert E. Blair, & Robert J. DeLorenzo. (2002). Calcium‐dependent Epileptogenesis in an In Vitro Model of Stroke‐induced “Epilepsy”. Epilepsia. 43(11). 1296–1305. 52 indexed citations
17.
18.
Morris, Dale L., et al.. (1992). Enhanced suppression of humoral immunity in DBA2 mice following subchronic exposure to 2,3,7,8-tetrachlorodibenzo-p-dioxin (TCDD). Toxicology and Applied Pharmacology. 112(1). 128–132. 18 indexed citations
19.
Holsapple, Michael P., et al.. (1991). Role of ah receptor in suppression of in vivo antibody response by 2 3 7 8 tetrachlorodibenzo p dioxin tcdd is dependent on exposure conditions. The FASEB Journal. 5(4). 508. 1 indexed citations
20.
Taft, William C., et al.. (1988). Cerebral ischemia decreases endogenous calcium-dependent protein phosphorylation in gerbil brain. Brain Research. 447(1). 159–163. 59 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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