Robert Düster

934 total citations
11 papers, 497 citations indexed

About

Robert Düster is a scholar working on Molecular Biology, Oncology and Cellular and Molecular Neuroscience. According to data from OpenAlex, Robert Düster has authored 11 papers receiving a total of 497 indexed citations (citations by other indexed papers that have themselves been cited), including 9 papers in Molecular Biology, 6 papers in Oncology and 1 paper in Cellular and Molecular Neuroscience. Recurrent topics in Robert Düster's work include Cancer-related Molecular Pathways (6 papers), RNA modifications and cancer (5 papers) and Genomics and Chromatin Dynamics (4 papers). Robert Düster is often cited by papers focused on Cancer-related Molecular Pathways (6 papers), RNA modifications and cancer (5 papers) and Genomics and Chromatin Dynamics (4 papers). Robert Düster collaborates with scholars based in Germany, United States and Denmark. Robert Düster's co-authors include Matthias Geyer, Ines H. Kaltheuner, Maximilian Schmitz, K. Anand, Ann Katrin Greifenberg, Koen Bartholomeeusen, Dalibor Blažek, Christian A. Bösken, Nathanael S. Gray and Jos Prickaerts and has published in prestigious journals such as Journal of Biological Chemistry, Nature Communications and Molecular Cell.

In The Last Decade

Robert Düster

11 papers receiving 493 citations

Peers

Robert Düster
Anil Narasimha United States
Chen‐Chun Pai United Kingdom
Ruth Thompson United States
Rachel A. Toroni United States
Cathleen Brdlik United States
Kristen Bisanz United States
Craig MacKay United Kingdom
Wesley J. Errington United States
Anil Narasimha United States
Robert Düster
Citations per year, relative to Robert Düster Robert Düster (= 1×) peers Anil Narasimha

Countries citing papers authored by Robert Düster

Since Specialization
Citations

This map shows the geographic impact of Robert Düster's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Robert Düster with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Robert Düster more than expected).

Fields of papers citing papers by Robert Düster

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Robert Düster. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Robert Düster. The network helps show where Robert Düster may publish in the future.

Co-authorship network of co-authors of Robert Düster

This figure shows the co-authorship network connecting the top 25 collaborators of Robert Düster. A scholar is included among the top collaborators of Robert Düster based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Robert Düster. Robert Düster is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

11 of 11 papers shown
1.
Düster, Robert, K. Anand, Maximilian Schmitz, et al.. (2024). Structural basis of Cdk7 activation by dual T-loop phosphorylation. Nature Communications. 15(1). 10 indexed citations
2.
Schmitz, Maximilian, Ines H. Kaltheuner, K. Anand, et al.. (2023). The reversible inhibitor SR-4835 binds Cdk12/cyclin K in a noncanonical G-loop conformation. Journal of Biological Chemistry. 300(1). 105501–105501. 6 indexed citations
3.
Düster, Robert, Yanlong Ji, Kuan‐Ting Pan, Henning Urlaub, & Matthias Geyer. (2022). Functional characterization of the human Cdk10/Cyclin Q complex. Open Biology. 12(3). 210381–210381. 11 indexed citations
4.
Düster, Robert, et al.. (2022). Transcriptional CDK Inhibitors as Potential Treatment Option for Testicular Germ Cell Tumors. Cancers. 14(7). 1690–1690. 7 indexed citations
5.
Kaltheuner, Ines H., K. Anand, Jonas Moecking, et al.. (2021). Abemaciclib is a potent inhibitor of DYRK1A and HIP kinases involved in transcriptional regulation. Nature Communications. 12(1). 6607–6607. 24 indexed citations
6.
Düster, Robert, Ines H. Kaltheuner, Maximilian Schmitz, & Matthias Geyer. (2021). 1,6-Hexanediol, commonly used to dissolve liquid–liquid phase separated condensates, directly impairs kinase and phosphatase activities. Journal of Biological Chemistry. 296. 100260–100260. 125 indexed citations
7.
Бугай, А. Н., Alexandre J.C. Quaresma, Caroline C. Friedel, et al.. (2019). P-TEFb Activation by RBM7 Shapes a Pro-survival Transcriptional Response to Genotoxic Stress. Molecular Cell. 74(2). 254–267.e10. 63 indexed citations
8.
Liang, Yanke, Alan L. Leggett, Lianbo Li, et al.. (2019). Development of a Selective CDK7 Covalent Inhibitor Reveals Predominant Cell-Cycle Phenotype. Cell chemical biology. 26(6). 792–803.e10. 115 indexed citations
9.
Greifenberg, Ann Katrin, Robert Düster, Koen Bartholomeeusen, et al.. (2015). Structural and Functional Analysis of the Cdk13/Cyclin K Complex. Cell Reports. 14(2). 320–331. 103 indexed citations
10.
Düster, Robert, Jos Prickaerts, & Arjan Blokland. (2013). Purinergic Signaling and Hippocampal Long-Term Potentiation. Current Neuropharmacology. 12(1). 37–43. 22 indexed citations
11.
Schmidt, Doris, et al.. (2011). Glutathione-S-transferase pi 1(GSTP1) gene silencing in prostate cancer cells is reversed by the histone deacetylase inhibitor depsipeptide. Biochemical and Biophysical Research Communications. 412(4). 606–611. 11 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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