Richard Holtmeier

741 total citations
7 papers, 205 citations indexed

About

Richard Holtmeier is a scholar working on Cardiology and Cardiovascular Medicine, Oncology and Epidemiology. According to data from OpenAlex, Richard Holtmeier has authored 7 papers receiving a total of 205 indexed citations (citations by other indexed papers that have themselves been cited), including 3 papers in Cardiology and Cardiovascular Medicine, 2 papers in Oncology and 2 papers in Epidemiology. Recurrent topics in Richard Holtmeier's work include Hormonal and reproductive studies (2 papers), Cell Adhesion Molecules Research (2 papers) and Cardiovascular Function and Risk Factors (2 papers). Richard Holtmeier is often cited by papers focused on Hormonal and reproductive studies (2 papers), Cell Adhesion Molecules Research (2 papers) and Cardiovascular Function and Risk Factors (2 papers). Richard Holtmeier collaborates with scholars based in Germany, Netherlands and Finland. Richard Holtmeier's co-authors include Jörg Stypmann, Dieter Häussinger, Jonghee Hwang, Linda F. Lorenz, Kari Alitalo, Karsten Müssig, Jennifer Axnick, Daniel Eberhard, Michael Roden and Harri Nurmi and has published in prestigious journals such as Nature, Scientific Reports and Journal of Medicinal Chemistry.

In The Last Decade

Richard Holtmeier

7 papers receiving 203 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Richard Holtmeier Germany	6	71	53	47	45	32	7	205
Pál Kaposi Novák Hungary	7	94 1.3×	52 1.0×	39 0.8×	66 1.5×	67 2.1×	9	347
Zan Lv China	10	169 2.4×	48 0.9×	124 2.6×	65 1.4×	26 0.8×	11	350
Alexey Gurevich United States	7	40 0.6×	31 0.6×	54 1.1×	44 1.0×	14 0.4×	12	160
Viktoria Kalna United Kingdom	6	126 1.8×	45 0.8×	13 0.3×	29 0.6×	32 1.0×	7	224
Taek Chung South Korea	11	86 1.2×	54 1.0×	178 3.8×	106 2.4×	49 1.5×	28	381
NORIYUKI MASAKI Japan	13	95 1.3×	18 0.3×	22 0.5×	176 3.9×	31 1.0×	37	425
Dongying Chen United States	8	155 2.2×	11 0.2×	63 1.3×	62 1.4×	26 0.8×	12	275
Kathrin Rupertus Germany	9	84 1.2×	36 0.7×	24 0.5×	192 4.3×	12 0.4×	10	287
Wendong Chen China	5	115 1.6×	157 3.0×	29 0.6×	123 2.7×	208 6.5×	15	374
Hsin-Chieh Tsay Germany	4	213 3.0×	88 1.7×	67 1.4×	24 0.5×	54 1.7×	5	339

Countries citing papers authored by Richard Holtmeier

Since Specialization

Citations

This map shows the geographic impact of Richard Holtmeier's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Richard Holtmeier with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Richard Holtmeier more than expected).

Fields of papers citing papers by Richard Holtmeier

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Richard Holtmeier. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Richard Holtmeier. The network helps show where Richard Holtmeier may publish in the future.

Co-authorship network of co-authors of Richard Holtmeier

This figure shows the co-authorship network connecting the top 25 collaborators of Richard Holtmeier. A scholar is included among the top collaborators of Richard Holtmeier based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Richard Holtmeier. Richard Holtmeier is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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7 of 7 papers shown

1.

Otto, Mandy, Melanie Müller, Richard Holtmeier, et al.. (2021). Development of heart failure with preserved ejection fraction in type 2 diabetic mice is ameliorated by preserving vascular function. Life Sciences. 284. 119925–119925. 10 indexed citations

2.

Wistuba, Joachim, Jörg Stypmann, Michael Kuhlmann, et al.. (2020). Testicular blood supply is altered in the 41,XXY* Klinefelter syndrome mouse model. Scientific Reports. 10(1). 14369–14369. 6 indexed citations

3.

Lorenz, Linda F., Jennifer Axnick, Shentong Fang, et al.. (2018). Mechanosensing by β1 integrin induces angiocrine signals for liver growth and survival. Nature. 562(7725). 128–132. 120 indexed citations

4.

Damm, Oliver, Joerg Stypmann, Michael Kuhlmann, et al.. (2016). Altered testicular vascularization and impaired blood supply in the 41,XXY* mouse model for Klinefelter syndrome. Endocrine Abstracts. 1 indexed citations

5.

Brix, Tobias, Richard Holtmeier, Desiree Abdurrachim, et al.. (2014). Velocity mapping of the aortic flow at 9.4 T in healthy mice and mice with induced heart failure using time-resolved three-dimensional phase-contrast MRI (4D PC MRI). Magnetic Resonance Materials in Physics Biology and Medicine. 28(4). 315–327. 13 indexed citations

6.

Persigehl, Thors ten, Janine Ring, Christoph Bremer, et al.. (2013). Non-invasive monitoring of tumor-vessel infarction by retargeted truncated tissue factor tTF–NGR using multi-modal imaging. Angiogenesis. 17(1). 235–246. 30 indexed citations

7.

Schwöppe, Christian, Christoph Schliemann, Torsten Keßler, et al.. (2013). Anticancer Therapy by Tumor Vessel Infarction with Polyethylene Glycol Conjugated Retargeted Tissue Factor. Journal of Medicinal Chemistry. 56(6). 2337–2347. 25 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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