Reas S. Khan

1.3k total citations
38 papers, 1.0k citations indexed

About

Reas S. Khan is a scholar working on Molecular Biology, Cellular and Molecular Neuroscience and Neurology. According to data from OpenAlex, Reas S. Khan has authored 38 papers receiving a total of 1.0k indexed citations (citations by other indexed papers that have themselves been cited), including 11 papers in Molecular Biology, 11 papers in Cellular and Molecular Neuroscience and 10 papers in Neurology. Recurrent topics in Reas S. Khan's work include Neuroinflammation and Neurodegeneration Mechanisms (9 papers), Multiple Sclerosis Research Studies (7 papers) and Sirtuins and Resveratrol in Medicine (7 papers). Reas S. Khan is often cited by papers focused on Neuroinflammation and Neurodegeneration Mechanisms (9 papers), Multiple Sclerosis Research Studies (7 papers) and Sirtuins and Resveratrol in Medicine (7 papers). Reas S. Khan collaborates with scholars based in United States, India and Switzerland. Reas S. Khan's co-authors include Kenneth S. Shindler, Kimberly Dine, Zoë Fonseca-Kelly, Hung Hsuchou, Jayasri Das Sarma, Weihong Pan, Abba J. Kastin, Ling Zuo, C.S. Paulose and Mahasweta Dutt and has published in prestigious journals such as Journal of Neuroscience, PLoS ONE and Journal of Virology.

In The Last Decade

Reas S. Khan

35 papers receiving 1.0k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Reas S. Khan United States 20 365 213 174 166 130 38 1.0k
Haijing Yan China 17 547 1.5× 314 1.5× 53 0.3× 253 1.5× 106 0.8× 42 1.4k
Paras S. Minhas United States 12 618 1.7× 480 2.3× 75 0.4× 338 2.0× 124 1.0× 19 1.5k
Won Gil Cho South Korea 13 440 1.2× 117 0.5× 26 0.1× 138 0.8× 116 0.9× 38 1.1k
Matthew S. Yorek United States 12 209 0.6× 40 0.2× 139 0.8× 224 1.3× 85 0.7× 23 729
Michele Madonna Italy 23 569 1.6× 145 0.7× 29 0.2× 210 1.3× 193 1.5× 45 1.2k
Zhiying Hu China 23 583 1.6× 251 1.2× 37 0.2× 170 1.0× 207 1.6× 63 1.5k
Stephanie A. Cordonnier United States 7 364 1.0× 163 0.8× 160 0.9× 294 1.8× 64 0.5× 8 851
Leonardo Cavone Italy 22 580 1.6× 189 0.9× 33 0.2× 163 1.0× 201 1.5× 31 1.3k
Hsiao‐Yun Lin Taiwan 22 473 1.3× 237 1.1× 37 0.2× 184 1.1× 106 0.8× 40 1.2k

Countries citing papers authored by Reas S. Khan

Since Specialization
Citations

This map shows the geographic impact of Reas S. Khan's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Reas S. Khan with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Reas S. Khan more than expected).

Fields of papers citing papers by Reas S. Khan

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Reas S. Khan. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Reas S. Khan. The network helps show where Reas S. Khan may publish in the future.

Co-authorship network of co-authors of Reas S. Khan

This figure shows the co-authorship network connecting the top 25 collaborators of Reas S. Khan. A scholar is included among the top collaborators of Reas S. Khan based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Reas S. Khan. Reas S. Khan is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
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Chaqour, Brahim, Reas S. Khan, Puya Aravand, et al.. (2024). Pharmacological Activation and Transgenic Overexpression of SIRT1 Attenuate Traumatic Optic Neuropathy Induced by Blunt Head Impact. Translational Vision Science & Technology. 13(9). 27–27.
3.
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Khan, Reas S., Benjamin Davis, Kimberly Dine, et al.. (2023). Nanoparticles Enhance Solubility and Neuroprotective Effects of Resveratrol in Demyelinating Disease. Neurotherapeutics. 20(4). 1138–1153. 17 indexed citations
5.
Willett, Keirnan, et al.. (2021). Neuroprotection mediated by ST266 requires full complement of proteins secreted by amnion-derived multipotent progenitor cells. PLoS ONE. 16(1). e0243862–e0243862. 9 indexed citations
6.
Khan, Reas S., Ahmara G. Ross, Keirnan Willett, et al.. (2020). Amnion-Derived Multipotent Progenitor Cells Suppress Experimental Optic Neuritis and Myelitis. Neurotherapeutics. 18(1). 448–459. 9 indexed citations
7.
Khan, Reas S., et al.. (2019). Effects of Varying Intranasal Treatment Regimens in ST266-Mediated Retinal Ganglion Cell Neuroprotection. Journal of Neuro-Ophthalmology. 39(2). 191–199. 17 indexed citations
8.
Khan, Reas S., Bailey Baumann, Kimberly Dine, et al.. (2019). Dexras1 Deletion and Iron Chelation Promote Neuroprotection in Experimental Optic Neuritis. Scientific Reports. 9(1). 11664–11664. 20 indexed citations
9.
Khan, Reas S., et al.. (2018). RGC Neuroprotection Following Optic Nerve Trauma Mediated By Intranasal Delivery of Amnion Cell Secretome. Investigative Ophthalmology & Visual Science. 59(6). 2470–2470. 20 indexed citations
10.
Khan, Reas S., Kimberly Dine, John G. Geisler, & Kenneth S. Shindler. (2017). Mitochondrial Uncoupler Prodrug of 2,4‐Dinitrophenol, MP201, Prevents Neuronal Damage and Preserves Vision in Experimental Optic Neuritis. Oxidative Medicine and Cellular Longevity. 2017(1). 7180632–7180632. 35 indexed citations
11.
Khan, Reas S., Kimberly Dine, Leah R. Hanson, et al.. (2017). Intranasal Delivery of A Novel Amnion Cell Secretome Prevents Neuronal Damage and Preserves Function In A Mouse Multiple Sclerosis Model. Scientific Reports. 7(1). 41768–41768. 36 indexed citations
12.
Addya, Sankar, Reas S. Khan, Lawrence C. Kenyon, et al.. (2014). Mouse Hepatitis Virus Infection Upregulates Genes Involved in Innate Immune Responses. PLoS ONE. 9(10). e111351–e111351. 16 indexed citations
13.
Khan, Reas S., Kimberly Dine, Esteban Luna, Clarence Ahlem, & Kenneth S. Shindler. (2014). HE3286 Reduces Axonal Loss and Preserves Retinal Ganglion Cell Function in Experimental Optic Neuritis. Investigative Ophthalmology & Visual Science. 55(9). 5744–5744. 23 indexed citations
14.
Khan, Reas S., Yong Chen, Ying Song, et al.. (2013). Dexras1 Mediates Retinal Ganglion Cell Loss Induced by NMDA Excitotoxicity. Investigative Ophthalmology & Visual Science. 54(15). 1414–1414. 1 indexed citations
15.
Chen, Yong, Reas S. Khan, Ying Song, et al.. (2013). Dexras1, a Small GTPase, Is Required for Glutamate-NMDA Neurotoxicity. Journal of Neuroscience. 33(8). 3582–3587. 59 indexed citations
16.
Fonseca-Kelly, Zoë, Jorge Uribe, Reas S. Khan, et al.. (2012). Resveratrol Neuroprotection in a Chronic Mouse Model of Multiple Sclerosis. Frontiers in Neurology. 3. 84–84. 132 indexed citations
17.
Shindler, Kenneth S., et al.. (2011). Macrophage-Mediated Optic Neuritis Induced by Retrograde Axonal Transport of Spike Gene Recombinant Mouse Hepatitis Virus. Journal of Neuropathology & Experimental Neurology. 70(6). 470–480. 25 indexed citations
18.
Wu, Xiaojun, Hung Hsuchou, Abba J. Kastin, et al.. (2010). Interleukin-15 affects serotonin system and exerts antidepressive effects through IL15Rα receptor. Psychoneuroendocrinology. 36(2). 266–278. 30 indexed citations
19.
Pan, Weihong, Xiaojun Wu, Abba J. Kastin, et al.. (2010). Potential Protective Role of IL15Rα During Inflammation. Journal of Molecular Neuroscience. 43(3). 412–423. 10 indexed citations
20.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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