R.E. See

2.3k citations

32 papers · 1.9k indexed · h-index 24

Impact in

Cellular and Molecular Neuroscience top 1%
- Neurotransmitter Receptor Influence on Behavior
- Neuroscience and Neuropharmacology Research
- Neuropeptides and Animal Physiology
Behavioral Neuroscience top 2%
- Stress Responses and Cortisol

Papers in ⓘ

Cellular and Molecular Neuroscience 30
- Neurotransmitter Receptor Influence on Behavior 27
- Neuroscience and Neuropharmacology Research 20
- Neuropeptides and Animal Physiology 3
Behavioral Neuroscience 3
- Stress Responses and Cortisol 3

Co-authors: Matthew W. Feltenstein (6 shared papers)William M. Meil (3 shared papers)June Rogers (2 shared papers)Shannon M. Ghee (1 shared paper)Jay Elliott (2 shared papers)Jacqueline F. McGinty (4 shared papers)G Ellison (3 shared papers)Rita A. Fuchs (2 shared papers)
Journals: Psychopharmacology (7 papers)Neuroscience (6 papers)Brain Research (2 papers)Pharmacology Biochemistry and Behavior (2 papers)Neuropharmacology (2 papers)
Partner nations: United States

In The Last Decade

R.E. See

32 papers receiving 1.9k citations

Peers

Countries citing papers authored by R.E. See

Since Specialization

Citations

This map shows the geographic impact of R.E. See's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by R.E. See with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites R.E. See more than expected).

Fields of papers citing papers by R.E. See

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by R.E. See. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by R.E. See. The network helps show where R.E. See may publish in the future.

Co-authors

The 25 scholars most cited alongside R.E. See, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with R.E. See Line = papers co-authored together R.E. See links everyone, so they are left out of the graph.

All Works

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20 of 20 papers shown

Showing the 20 most-cited of 32 papers — load more, or switch the sort, to bring in the rest.

#	Work
1	The neurocircuitry of addiction: an overview British Journal of Pharmacology ·Matthew W. Feltenstein, R.E. See	2008	297
2	Conditioned cued recovery of responding following prolonged withdrawal from self-administered cocaine in rats: an animal model of relapse. PubMed ·William M. Meil, R.E. See	1996	184
3	The neural circuitry underlying reinstatement of heroin-seeking behavior in an animal model of relapse Neuroscience ·June Rogers, Shannon M. Ghee, R.E. See	2007	167
4	The role of dorsal vs ventral striatal pathways in cocaine-seeking behavior after prolonged abstinence in rats Psychopharmacology ·R.E. See, Jay Elliott, Matthew W. Feltenstein	2007	130
5	Potentiation of cue-induced reinstatement of cocaine-seeking in rats by the anxiogenic drug yohimbine Behavioural Brain Research ·Matthew W. Feltenstein, R.E. See	2006	128
6	Extended methamphetamine self-administration enhances reinstatement of drug seeking and impairs novel object recognition in rats Psychopharmacology ·June Rogers, S. De Santis, R.E. See	2008	106
7	Dopamine D1 or D2 receptor antagonism within the basolateral amygdala differentially alters the acquisition of cocaine-cue associations necessary for cue-induced reinstatement of cocaine-seeking Neuroscience ·William J. Berglind, Jordan M. Case, Macon P. Parker, Rita A. Fuchs, R.E. See	2005	80
8	NMDA receptor blockade in the basolateral amygdala disrupts consolidation of stimulus-reward memory and extinction learning during reinstatement of cocaine-seeking in an animal model of relapse Neurobiology of Learning and Memory ·Matthew W. Feltenstein, R.E. See	2007	67
9	Muscarinic receptor antagonism in the basolateral amygdala blocks acquisition of cocaine-stimulus association in a model of relapse to cocaine-seeking behavior in rats Neuroscience ·R.E. See, Joselyn McLaughlin, Rita A. Fuchs	2003	65
10	Tremorous mouth movements in rats administered chronic neuroleptics Psychopharmacology ·G Ellison, R.E. See, Edward D. Levin, Jefferson W. Kinney	1987	61
11	Relapse to cocaine seeking increases activity-regulated gene expression differentially in the prefrontal cortex of abstinent rats Psychopharmacology ·Matthew C. Hearing, Scott W. Miller, R.E. See, Jacqueline F. McGinty	2008	60
12	In vivo assessment of release and metabolism of dopamine in the ventrolateral striatum of awake rats following administration of dopamine D1 and D2 receptor agonists and antagonists Neuropharmacology ·R.E. See, Barbara A. Sorg, M Chapman, Peter W. Kalivas	1991	59
13	Autoradiographic analysis of regional alterations in brain receptors following chronic administration and withdrawal of typical and atypical neuroleptics in rats Journal of Neural Transmission ·R.E. See, Arthur W. Toga, G Ellison	1990	50
14	Oxytocin Reduces Cocaine Seeking and Reverses Chronic Cocaine-Induced Changes in Glutamate Receptor Function The International Journal of Neuropsychopharmacology ·Ling Zhou, Wei Sun, A. Byron Young, Kevin Lee, Jacqueline F. McGinty, R.E. See	2014	47
15	Relapse to cocaine-seeking increases activity-regulated gene expression differentially in the striatum and cerebral cortex of rats following short or long periods of abstinence Brain Structure and Function ·Matthew C. Hearing, R.E. See, Jacqueline F. McGinty	2008	46
16	Tolerance-like attenuation to contingent and noncontingent cocaine-induced elevation of extracellular dopamine in the ventral striatum following 7 days of withdrawal from chronic treatment Psychopharmacology ·William M. Meil, John M. Roll, Jeffrey W. Grimm, A.M. Lynch, R.E. See	1995	38
17	Conditioned reinstatement of drug-seeking behavior with a discrete compound stimulus classically conditioned with intravenous cocaine. Behavioral Neuroscience ·Paul J. Kruzich, K M Congleton, R.E. See	2001	37
18	Systems Level Neuroplasticity in Drug Addiction Cold Spring Harbor Perspectives in Medicine ·Matthew W. Feltenstein, R.E. See	2013	33
19	Characteristics of oral movements in rats during and after chronic haloperidol and fluphenazine administration Psychopharmacology ·R.E. See, EdwardD. Levin, G Ellison	1988	30
20	Differential effects of unique profile antipsychotic drugs on extracellular amino acids in the ventral pallidum and globus pallidus of rats. Journal of Pharmacology and Experimental Therapeutics ·M Chapman, R.E. See	1996	28

About R.E. See

R.E. See is a scholar working on Cellular and Molecular Neuroscience, Behavioral Neuroscience, Cognitive Neuroscience, Biological Psychiatry and Molecular Biology, having authored 32 papers that have together received 1.9k indexed citations. Recurring topics across this work include Neurotransmitter Receptor Influence on Behavior (27 papers), Neuroscience and Neuropharmacology Research (20 papers), Receptor Mechanisms and Signaling (13 papers), Memory and Neural Mechanisms (12 papers), Neuropeptides and Animal Physiology (3 papers), Stress Responses and Cortisol (3 papers), Neuroendocrine regulation and behavior (3 papers) and Parkinson's Disease Mechanisms and Treatments (2 papers). The work is most often cited by research in Cellular and Molecular Neuroscience (1.6k citations), Behavioral Neuroscience (202 citations), Cognitive Neuroscience (632 citations), Biological Psychiatry (69 citations) and Endocrine and Autonomic Systems (104 citations). R.E. See has collaborated with scholars based in United States. Frequent co-authors include Matthew W. Feltenstein, William M. Meil, June Rogers, Shannon M. Ghee, Jay Elliott, Jacqueline F. McGinty, G Ellison, Rita A. Fuchs, Matthew C. Hearing and Jeffrey W. Grimm. Their work appears in journals such as Psychopharmacology, Neuroscience, Brain Research, Pharmacology Biochemistry and Behavior and Neuropharmacology.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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