Raz Abdulqadir

404 total citations
15 papers, 273 citations indexed

About

Raz Abdulqadir is a scholar working on Molecular Biology, Neurology and Periodontics. According to data from OpenAlex, Raz Abdulqadir has authored 15 papers receiving a total of 273 indexed citations (citations by other indexed papers that have themselves been cited), including 8 papers in Molecular Biology, 5 papers in Neurology and 4 papers in Periodontics. Recurrent topics in Raz Abdulqadir's work include Gut microbiota and health (7 papers), Barrier Structure and Function Studies (5 papers) and Oral microbiology and periodontitis research (4 papers). Raz Abdulqadir is often cited by papers focused on Gut microbiota and health (7 papers), Barrier Structure and Function Studies (5 papers) and Oral microbiology and periodontitis research (4 papers). Raz Abdulqadir collaborates with scholars based in United States, Canada and United Kingdom. Raz Abdulqadir's co-authors include Rana Al–Sadi, Nick E. Le Brun, M.E.P. Murphy, Geoffrey R. Moore, Xiangyu Cai, Donald L. Gill, Robert M. Nwokonko, Youjun Wang, Natalia A. Loktionova and Yandong Zhou and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Biological Chemistry and Gastroenterology.

In The Last Decade

Raz Abdulqadir

14 papers receiving 273 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Raz Abdulqadir United States	8	140	66	61	61	40	15	273
R. Leschiera France	12	103 0.7×	14 0.2×	48 0.8×	4 0.1×	36 0.9×	19	326
Cornelia Fischer Germany	9	171 1.2×	54 0.8×	45 0.7×	46 0.8×	19 0.5×	12	495
Yohann Grondin United States	8	126 0.9×	19 0.3×	38 0.6×	22 0.4×	11 0.3×	19	334
Paolo Iacovelli Italy	13	88 0.6×	108 1.6×	53 0.9×	4 0.1×	10 0.3×	22	551
Hans‐Peter Bode Germany	13	230 1.6×	60 0.9×	30 0.5×	18 0.3×	62 1.6×	19	459
Didier Demaegd Belgium	8	339 2.4×	40 0.6×	12 0.2×	13 0.2×	25 0.6×	10	475
Li-Guo Jia United States	9	270 1.9×	33 0.5×	4 0.1×	6 0.1×	23 0.6×	9	383
Luis M. Bredeston Argentina	9	150 1.1×	54 0.8×	21 0.3×	3 0.0×	11 0.3×	15	328
Linda Kelsey United States	12	154 1.1×	52 0.8×	3 0.0×	8 0.1×	57 1.4×	16	339
Angela Georgiou Australia	7	91 0.7×	7 0.1×	4 0.1×	10 0.2×	81 2.0×	8	244

Countries citing papers authored by Raz Abdulqadir

Since Specialization

Citations

This map shows the geographic impact of Raz Abdulqadir's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Raz Abdulqadir with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Raz Abdulqadir more than expected).

Fields of papers citing papers by Raz Abdulqadir

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Raz Abdulqadir. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Raz Abdulqadir. The network helps show where Raz Abdulqadir may publish in the future.

Co-authorship network of co-authors of Raz Abdulqadir

This figure shows the co-authorship network connecting the top 25 collaborators of Raz Abdulqadir. A scholar is included among the top collaborators of Raz Abdulqadir based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Raz Abdulqadir. Raz Abdulqadir is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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15 of 15 papers shown

Abdulqadir, Raz, Thomas Ma, & Rana Al–Sadi. (2025). Bifidobacterium bifidumup-regulation of intestinal epithelial tight junction barrier is mediated by TLR-2/TLR-6 signal transduction pathway activation of occludin gene. Physiology. 40(S1).

Abdulqadir, Raz, et al.. (2024). Bifidobacterium bifidum Strain BB1 Inhibits Tumor Necrosis Factor-α–Induced Increase in Intestinal Epithelial Tight Junction Permeability via Toll-Like Receptor-2/Toll-Like Receptor-6 Receptor Complex–Dependent Stimulation of Peroxisome Proliferator-Activated Receptor γ and Suppression of NF-κB p65. American Journal Of Pathology. 194(9). 1664–1683. 4 indexed citations

Kaminsky, Lauren W., et al.. (2024). Lactobacillus acidophilus inhibits the TNF-α-induced increase in intestinal epithelial tight junction permeability via a TLR-2 and PI3K-dependent inhibition of NF-κB activation. Frontiers in Immunology. 15. 1348010–1348010. 22 indexed citations

Abdulqadir, Raz, et al.. (2023). Role of Bifidobacterium in Modulating the Intestinal Epithelial Tight Junction Barrier: Current Knowledge and Perspectives. Current Developments in Nutrition. 7(12). 102026–102026. 55 indexed citations

Abdulqadir, Raz, Rana Al–Sadi, & Thomas Ma. (2023). Bifidobacterium bifidum prevents the IL-1B induced increase in intestinal permeability by a novel mechanism: TLR-2 dependent activation of PPAR-gamma and inhibition of NF-kB signaling pathway. Physiology. 38(S1). 3 indexed citations

Abdulqadir, Raz, et al.. (2023). 768 BIFIDOBACTERIUM BIFIDUM PREVENTS CYTOKINE INDUCED INCREASE IN INTESTINAL TIGHT JUNCTION PERMEABILITY BY A NOVEL MECHANISM: CROSSTALK BETWEEN PPAR-g, NF-kB AND MLCK. Gastroenterology. 164(6). S–163. 1 indexed citations

Abdulqadir, Raz, Rana Al–Sadi, & Thomas Ma. (2023). Bifidobacterium bifidum causes an enhancement of the intestinal epithelial tight junction barrier is mediated by TLR-2 dependent increases of Toll-interacting protein (TOLLIP) expression in a MYD88 independent manner. Physiology. 38(S1). 2 indexed citations

Abdulqadir, Raz, Rana Al–Sadi, & Y. Thomas. (2023). Sa1215 BIFIDOBACTERIUM BIFIDUM CAUSES AN ENHANCEMENT OF THE INTESTINAL EPITHELIAL TIGHT JUNCTION BARRIER IS MEDIATED BY TLR-2-DEPENDENT UPREGULATION OF OCCLUDIN EXPRESSION. Gastroenterology. 164(6). S–329. 1 indexed citations

Abdulqadir, Raz, et al.. (2022). Tu1113: BIFIDOBACTERIUM BIFIDUM CAUSES AN ENHANCEMENT OF INTESTINAL EPITHELIAL TIGHT JUNCTION BARRIER BY A NOVEL MECHANISM INVOLVING PEROXISOME PROLIFERATION-ACTIVATED RECEPTOR GAMMA (PPAR-γ). Gastroenterology. 162(7). S–887. 1 indexed citations

10.

Al–Sadi, Rana, et al.. (2020). Talk about micromanaging! Role of microRNAs in intestinal barrier function. American Journal of Physiology-Gastrointestinal and Liver Physiology. 319(2). G170–G174. 22 indexed citations

11.

Zhou, Yandong, Robert M. Nwokonko, Xiangyu Cai, et al.. (2018). Cross-linking of Orai1 channels by STIM proteins. Proceedings of the National Academy of Sciences. 115(15). E3398–E3407. 50 indexed citations

12.

Cai, Xiangyu, Robert M. Nwokonko, Natalia A. Loktionova, et al.. (2018). Pore properties of Orai1 calcium channel dimers and their activation by the STIM1 ER calcium sensor. Journal of Biological Chemistry. 293(33). 12962–12974. 17 indexed citations

13.

Bradley, Justin M., Raz Abdulqadir, Marlo Firme, et al.. (2015). A Diatom Ferritin Optimized for Iron Oxidation but Not Iron Storage. Journal of Biological Chemistry. 290(47). 28416–28427. 26 indexed citations

14.

Abdulqadir, Raz, et al.. (2013). Mechanism of Ferrous Iron Binding and Oxidation by Ferritin from a Pennate Diatom. Journal of Biological Chemistry. 288(21). 14917–14925. 47 indexed citations

15.

Abdulqadir, Raz, et al.. (2012). Fe-haem bound to Escherichia coli bacterioferritin accelerates iron core formation by an electron transfer mechanism. Biochemical Journal. 444(3). 553–560. 22 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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