Raphael Pavani

1.1k total citations
23 papers, 553 citations indexed

About

Raphael Pavani is a scholar working on Molecular Biology, Epidemiology and Oncology. According to data from OpenAlex, Raphael Pavani has authored 23 papers receiving a total of 553 indexed citations (citations by other indexed papers that have themselves been cited), including 21 papers in Molecular Biology, 9 papers in Epidemiology and 3 papers in Oncology. Recurrent topics in Raphael Pavani's work include DNA Repair Mechanisms (15 papers), CRISPR and Genetic Engineering (11 papers) and Trypanosoma species research and implications (9 papers). Raphael Pavani is often cited by papers focused on DNA Repair Mechanisms (15 papers), CRISPR and Genetic Engineering (11 papers) and Trypanosoma species research and implications (9 papers). Raphael Pavani collaborates with scholars based in United States, Brazil and United Kingdom. Raphael Pavani's co-authors include Maria Carolina Elias, André Nussenzweig, Wei Wu, Elsa Callén, Marcelo S. da Silva, Maria Isabel Nogueira Cano, Dali Zong, Michael J. Kruhlak, Gabriel Matos‐Rodrigues and Nancy Wong and has published in prestigious journals such as Science, Nature Communications and Genes & Development.

In The Last Decade

Raphael Pavani

22 papers receiving 549 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Raphael Pavani United States 15 451 145 86 83 55 23 553
Marie‐Michelle Genois Canada 11 397 0.9× 91 0.6× 81 0.9× 134 1.6× 39 0.7× 12 486
Shao-bing Hua United States 14 408 0.9× 149 1.0× 94 1.1× 99 1.2× 44 0.8× 22 583
Phil Y. Yao United States 9 427 0.9× 116 0.8× 86 1.0× 38 0.5× 23 0.4× 15 546
Jane E. Itzhaki United Kingdom 7 409 0.9× 167 1.2× 76 0.9× 111 1.3× 69 1.3× 7 502
Karthik Maddi Germany 5 313 0.7× 335 2.3× 32 0.4× 59 0.7× 15 0.3× 5 620
Idit Hazan United States 9 409 0.9× 132 0.9× 20 0.2× 52 0.6× 51 0.9× 12 548
Jer-Yuan Hsu United States 10 894 2.0× 98 0.7× 15 0.2× 59 0.7× 111 2.0× 11 1.1k
Sanja Blašković Switzerland 7 301 0.7× 56 0.4× 17 0.2× 39 0.5× 25 0.5× 11 462
Timurs Maculins United Kingdom 8 450 1.0× 88 0.6× 9 0.1× 72 0.9× 63 1.1× 8 527
George K. Arhin United States 10 473 1.0× 213 1.5× 64 0.7× 11 0.1× 25 0.5× 12 586

Countries citing papers authored by Raphael Pavani

Since Specialization
Citations

This map shows the geographic impact of Raphael Pavani's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Raphael Pavani with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Raphael Pavani more than expected).

Fields of papers citing papers by Raphael Pavani

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Raphael Pavani. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Raphael Pavani. The network helps show where Raphael Pavani may publish in the future.

Co-authorship network of co-authors of Raphael Pavani

This figure shows the co-authorship network connecting the top 25 collaborators of Raphael Pavani. A scholar is included among the top collaborators of Raphael Pavani based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Raphael Pavani. Raphael Pavani is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
2.
Zong, Dali, Raphael Pavani, & André Nussenzweig. (2025). New twist on BRCA1-mediated DNA recombination repair and tumor suppression. Trends in Cell Biology. 36(1). 4–12. 1 indexed citations
3.
Mendes, I.C., Raphael Pavani, Andréa Mara Macedo, et al.. (2024). DNA lesions that block transcription induce the death of Trypanosoma cruzi via ATR activation, which is dependent on the presence of R-loops. DNA repair. 141. 103726–103726. 1 indexed citations
4.
Reginato, Giordano, Yanbo Wang, Jingzhou Hao, et al.. (2024). HLTF disrupts Cas9-DNA post-cleavage complexes to allow DNA break processing. Nature Communications. 15(1). 5789–5789. 17 indexed citations
5.
Kooij, Bert van de, Raphael Pavani, Arne van Hoeck, et al.. (2024). EXO1 protects BRCA1-deficient cells against toxic DNA lesions. Molecular Cell. 84(4). 659–674.e7. 20 indexed citations
6.
Pavani, Raphael, Veenu Tripathi, Dali Zong, et al.. (2024). Structure and repair of replication-coupled DNA breaks. Science. 385(6710). eado3867–eado3867. 45 indexed citations
7.
Wang, Dongpeng, Wei Wu, Elsa Callén, et al.. (2022). Active DNA demethylation promotes cell fate specification and the DNA damage response. Science. 378(6623). 983–989. 69 indexed citations
8.
Matos‐Rodrigues, Gabriel, Niek van Wietmarschen, Wei Wu, et al.. (2022). S1-END-seq reveals DNA secondary structures in human cells. Molecular Cell. 82(19). 3538–3552.e5. 34 indexed citations
9.
Wu, Wei, Raphael Pavani, Jacob Paiano, et al.. (2022). DNA-PK promotes DNA end resection at DNA double strand breaks in G0 cells. eLife. 11. 17 indexed citations
10.
Pavani, Raphael & Maria Carolina Elias. (2021). Following Trypanosoma cruzi RPA-DNA Interaction Using Fluorescent In Situ Hybridization Coupled with Immunofluorescence (FISH/IF). Methods in molecular biology. 2281. 209–215. 3 indexed citations
11.
Paiano, Jacob, Wei Wu, Raphael Pavani, et al.. (2021). Role of 53BP1 in end protection and DNA synthesis at DNA breaks. Genes & Development. 35(19-20). 1356–1367. 33 indexed citations
12.
Olbrich, Teresa, María Vega-Sendino, Desiree Tillo, et al.. (2021). CTCF is a barrier for 2C-like reprogramming. Nature Communications. 12(1). 4856–4856. 44 indexed citations
13.
Pavani, Raphael, et al.. (2020). Nuclear export of replication protein A in the nonreplicative infective forms of Trypanosoma cruzi. FEBS Letters. 594(10). 1596–1607. 7 indexed citations
14.
Callén, Elsa, Dali Zong, Wei Wu, et al.. (2019). 53BP1 Enforces Distinct Pre- and Post-resection Blocks on Homologous Recombination. Molecular Cell. 77(1). 26–38.e7. 83 indexed citations
15.
Damasceno, Flávia Silva, Agustín Hernández, María Julia Barisón, et al.. (2018). The glutamine synthetase of Trypanosoma cruzi is required for its resistance to ammonium accumulation and evasion of the parasitophorous vacuole during host-cell infection. PLoS neglected tropical diseases. 12(1). e0006170–e0006170. 23 indexed citations
16.
Silva, Marcelo S. da, Raphael Pavani, Jeziel D. Damasceno, et al.. (2017). Nuclear DNA Replication in Trypanosomatids: There Are No Easy Methods for Solving Difficult Problems. Trends in Parasitology. 33(11). 858–874. 28 indexed citations
17.
Silva, Marcelo S. da, Marcela Segatto, Raphael Pavani, et al.. (2016). Consequences of acute oxidative stress in Leishmania amazonensis : From telomere shortening to the selection of the fittest parasites. Biochimica et Biophysica Acta (BBA) - Molecular Cell Research. 1864(1). 138–150. 28 indexed citations
18.
Pavani, Raphael, Marcelo S. da Silva, Carlos A. H. Fernandes, et al.. (2016). Replication Protein A Presents Canonical Functions and Is Also Involved in the Differentiation Capacity of Trypanosoma cruzi. PLoS neglected tropical diseases. 10(12). e0005181–e0005181. 26 indexed citations
19.
Pavani, Raphael, Marcelo S. Reis, Vincent Noël, et al.. (2015). Glyceraldehyde 3-Phosphate Dehydrogenase-Telomere Association Correlates with Redox Status in Trypanosoma cruzi. PLoS ONE. 10(3). e0120896–e0120896. 28 indexed citations
20.
Pavani, Raphael, Carlos A. H. Fernandes, Elton J. R. Vasconcelos, et al.. (2014). RPA‐1 from Leishmania amazonensis (LaRPA‐1) structurally differs from other eukaryote RPA‐1 and interacts with telomeric DNA via its N‐terminal OB‐fold domain. FEBS Letters. 588(24). 4740–4748. 13 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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