Ralph Witzgall

6.9k total citations · 1 hit paper
95 papers, 5.2k citations indexed

About

Ralph Witzgall is a scholar working on Molecular Biology, Genetics and Pathology and Forensic Medicine. According to data from OpenAlex, Ralph Witzgall has authored 95 papers receiving a total of 5.2k indexed citations (citations by other indexed papers that have themselves been cited), including 66 papers in Molecular Biology, 48 papers in Genetics and 19 papers in Pathology and Forensic Medicine. Recurrent topics in Ralph Witzgall's work include Genetic and Kidney Cyst Diseases (41 papers), Renal and related cancers (31 papers) and Biomedical Research and Pathophysiology (19 papers). Ralph Witzgall is often cited by papers focused on Genetic and Kidney Cyst Diseases (41 papers), Renal and related cancers (31 papers) and Biomedical Research and Pathophysiology (19 papers). Ralph Witzgall collaborates with scholars based in Germany, United States and France. Ralph Witzgall's co-authors include Joseph V. Bonventre, Stefan Somlo, Dennis Brown, Wilhelm Kriz, Christoph Schwarz, J. V. Bonventre, Yiqiang Cai, Yoshiko Maeda, Norbert Gretz and Elise O’Leary and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Biological Chemistry and Journal of Clinical Investigation.

In The Last Decade

Ralph Witzgall

95 papers receiving 5.1k citations

Hit Papers

Polycystin-2 is an intracellular calcium release channel 2002 2026 2010 2018 2002 100 200 300 400 500

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Ralph Witzgall Germany 35 3.4k 1.8k 857 811 434 95 5.2k
James P. Calvet United States 43 4.0k 1.2× 3.2k 1.8× 492 0.6× 1.1k 1.3× 159 0.4× 123 5.5k
Vincent H. Gattone United States 46 3.6k 1.0× 3.2k 1.8× 1.4k 1.7× 1.1k 1.4× 207 0.5× 139 6.7k
Bernhard Schermer Germany 42 4.0k 1.2× 2.2k 1.2× 1.5k 1.7× 501 0.6× 237 0.5× 126 6.0k
Marco Seri Italy 38 2.3k 0.7× 1.4k 0.8× 394 0.5× 279 0.3× 463 1.1× 193 5.3k
Anna Spada Italy 46 2.6k 0.8× 1.4k 0.8× 390 0.5× 259 0.3× 445 1.0× 172 7.0k
Robert Kleta United Kingdom 42 2.8k 0.8× 518 0.3× 1.1k 1.3× 1.2k 1.5× 475 1.1× 147 5.7k
Brian Harding United Kingdom 46 2.8k 0.8× 1.1k 0.6× 597 0.7× 300 0.4× 909 2.1× 162 6.9k
Terry Watnick United States 41 3.5k 1.0× 4.2k 2.3× 459 0.5× 1.1k 1.4× 220 0.5× 78 5.3k
Dorien J.M. Peters Netherlands 47 6.4k 1.9× 6.3k 3.5× 965 1.1× 1.6k 1.9× 415 1.0× 171 9.8k
Lee S. Weinstein United States 55 5.4k 1.6× 3.7k 2.0× 948 1.1× 382 0.5× 543 1.3× 184 10.4k

Countries citing papers authored by Ralph Witzgall

Since Specialization
Citations

This map shows the geographic impact of Ralph Witzgall's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Ralph Witzgall with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Ralph Witzgall more than expected).

Fields of papers citing papers by Ralph Witzgall

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Ralph Witzgall. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Ralph Witzgall. The network helps show where Ralph Witzgall may publish in the future.

Co-authorship network of co-authors of Ralph Witzgall

This figure shows the co-authorship network connecting the top 25 collaborators of Ralph Witzgall. A scholar is included among the top collaborators of Ralph Witzgall based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Ralph Witzgall. Ralph Witzgall is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Beck, Sebastian, et al.. (2025). The quantity of ligand–receptor interactions between nanoparticles and target cells. Nanoscale Horizons. 10(4). 803–823. 6 indexed citations
2.
Przybilla, Frédéric, Reinhard Rachel, Thomas Hirsch, et al.. (2024). Impact of surface chemistry of upconversion nanoparticles on time-dependent cytotoxicity in non-cancerous epithelial cells. Scientific Reports. 14(1). 30610–30610. 4 indexed citations
3.
Jiménez-Moreno, Natalia, Madhu Kollareddy, Zuriñe Antón, et al.. (2023). ATG8-dependent LMX1B-autophagy crosstalk shapes human midbrain dopaminergic neuronal resilience. The Journal of Cell Biology. 222(5). 9 indexed citations
4.
Witzgall, Ralph. (2017). Nail-patella syndrome. Pflügers Archiv - European Journal of Physiology. 469(7-8). 927–936. 24 indexed citations
5.
Wagenknecht, Hans‐Achim, et al.. (2013). Photochemically Active Fluorophore–DNA/RNA Conjugates for Cellular Imaging of Nucleic Acids by Readout in Electron Microscopy. ChemistryOpen. 2(4). 136–140. 2 indexed citations
6.
Kruspe, Dagmar, et al.. (2009). 09-P005 The Wilms tumour protein is required for kidney function in adult mice. Mechanisms of Development. 126. S152–S152. 1 indexed citations
7.
Rascle, Anne, et al.. (2008). The LIM-homeodomain transcription factor LMX1B regulates expression of NF-kappa B target genes. Experimental Cell Research. 315(1). 76–96. 21 indexed citations
8.
Witzgall, Ralph. (2007). TRPP2 Channel Regulation. Handbook of experimental pharmacology. 363–375. 11 indexed citations
9.
Suleiman, Hani, Daniel Heudobler, Yangu Zhao, et al.. (2007). The podocyte-specific inactivation of Lmx1b, Ldb1 and E2a yields new insight into a transcriptional network in podocytes. Developmental Biology. 304(2). 701–712. 42 indexed citations
10.
Aguiari, Gianluca, Alessandra Mangolini, Paolo Pinton, et al.. (2006). Polycystin-1 promotes PKCα-mediated NF-κB activation in kidney cells. Biochemical and Biophysical Research Communications. 350(2). 257–262. 14 indexed citations
11.
Hoffmann, Sigrid, Pamela J. Kaisaki, Patrick Danoy, et al.. (2005). Missense Mutation in Sterile α Motif of Novel Protein SamCystin is Associated with Polycystic Kidney Disease in (cy/+) Rat. Journal of the American Society of Nephrology. 16(12). 3517–3526. 73 indexed citations
12.
Rohr, Claudia M., Jürgen Prestel, Laurence Heidet, et al.. (2002). The LIM-homeodomain transcription factor Lmx1b plays a crucial role in podocytes. Journal of Clinical Investigation. 109(8). 1073–1082. 8 indexed citations
13.
Rohr, Claudia M., Jürgen Prestel, Laurence Heidet, et al.. (2002). The LIM-homeodomain transcription factor Lmx1b plays a crucial role in podocytes. Journal of Clinical Investigation. 109(8). 1073–1082. 73 indexed citations
14.
Witzgall, Ralph, Bettina Kränzlin, Norbert Gretz, & Nicholas Obermüller. (2002). Impaired endocytosis may represent an obstacle to gene therapy in polycystic kidney disease. Kidney International. 61(1). S132–S137. 5 indexed citations
15.
Endlich, Karlhans, Wilhelm Kriz, & Ralph Witzgall. (2001). Update in podocyte biology. Current Opinion in Nephrology & Hypertension. 10(3). 331–340. 79 indexed citations
16.
Gallagher, Anna‐Rachel, Nicholas Obermüller, Anna Cedzich, Norbert Gretz, & Ralph Witzgall. (2000). An ever-expanding story of cyst formation. Cell and Tissue Research. 300(3). 361–371. 9 indexed citations
17.
Huang, Zhiqing, Bärbel Philippin, Eileen O’Leary, et al.. (1999). Expression of the Transcriptional Repressor Protein Kid-1 Leads to the Disintegration of the Nucleolus. Journal of Biological Chemistry. 274(12). 7640–7648. 18 indexed citations
18.
Witzgall, Ralph. (1999). The Proximal Tubule Phenotype and Its Disruption in Acute Renal Failure and Polycystic Kidney Disease. Nephron Experimental Nephrology. 7(1). 15–19. 15 indexed citations
19.
Obermüller, Nicholas, N. Gretz, Wilhelm Kriz, Robert F. Reilly, & Ralph Witzgall. (1998). The swelling-activated chloride channel ClC-2, the chloride channel ClC-3, and ClC-5, a chloride channel mutated in kidney stone disease, are expressed in distinct subpopulations of renal epithelial cells.. Journal of Clinical Investigation. 101(3). 635–642. 67 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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